日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Early life adversity increases risk for chronic post-traumatic pain, data from humans and rodents

早期生活逆境会增加患慢性创伤后疼痛的风险,来自人类和啮齿动物的数据

McKibben, Lauren A; Woolard, Alice; McLean, Samuel A; Zhao, Ying; Verma, Taanvii; Mickelson, Jacqueline; Lu, Hongxia; Lobo, Jarred; House, Stacey L; Beaudoin, Francesca L; An, Xinming; Stevens, Jennifer S; Neylan, Thomas C; Jovanovic, Tanja; Germine, Laura T; Rauch, Scott L; Haran, John P; Storrow, Alan B; Lewandowski, Christopher; Hendry, Phyllis L; Sheikh, Sophia; Jones, Christopher W; Punches, Brittany E; Hudak, Lauren A; Pascual, Jose L; Seamon, Mark J; Pearson, Claire; Peak, David A; Merchant, Roland C; Domeier, Robert M; Rathlev, Niels K; O'Neil, Brian J; Sanchez, Leon D; Bruce, Steven E; Sheridan, John F; Kessler, Ronald C; Koenen, Karestan C; Ressler, Kerry J; Linnstaedt, Sarah D

Further evidence that peritraumatic 17β-estradiol levels influence chronic posttraumatic pain outcomes in women, data from both humans and animals.

进一步的证据表明,创伤后 17β-雌二醇水平会影响女性慢性创伤后疼痛的结果,数据来自人类和动物

Son Esther, Gaither Rachel, Lobo Jarred, Zhao Ying, McKibben Lauren A, Arora Rhea, Albertorio-Sáez Liz, Mickelson Jacqueline, Wanstrath Britannia J, Bhatia Simran, Stevens Jennifer S, Jovanovic Tanja, Koenen Karestan, Kessler Ronald, Ressler Kerry, Beaudoin Francesca L, McLean Samuel A, Linnstaedt Sarah D

Duration of Reduction in Enduring Stress-Induced Hyperalgesia Via FKBP51 Inhibition Depends on Timing of Administration Relative to Traumatic Stress Exposure

通过抑制FKBP51来减轻持久性应激诱发痛觉过敏的持续时间取决于给药时间与创伤性应激暴露的时间关系。

Wanstrath, Britannia J; McLean, Samuel A; Zhao, Ying; Mickelson, Jacqueline; Bauder, Michael; Hausch, Felix; Linnstaedt, Sarah D

Anticoagulation increases alveolar hemorrhage in mice infected with influenza A

抗凝治疗会增加感染甲型流感病毒小鼠的肺泡出血。

Tatsumi, Kohei; Antoniak, Silvio; Subramaniam, Saravanan; Gondouin, Bertrand; Neidich, Scott D; Beck, Melinda A; Mickelson, Jacqueline; Monroe, Dougald M 3rd; Bastarache, Julie A; Mackman, Nigel

Differential contribution of FXa and thrombin to vascular inflammation in a mouse model of sickle cell disease.

FXa 和凝血酶对镰状细胞病小鼠模型血管炎症的不同贡献

Sparkenbaugh Erica M, Chantrathammachart Pichika, Mickelson Jacqueline, van Ryn Joanne, Hebbel Robert P, Monroe Dougald M, Mackman Nigel, Key Nigel S, Pawlinski Rafal