日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Proteoglycan aggrecan conducting T cell activation and apoptosis in a murine model of rheumatoid arthritis

蛋白聚糖聚集蛋白聚糖在类风湿性关节炎小鼠模型中介导T细胞活化和凋亡

Hanyecz, A; Olasz, K; Tarjanyi, O; Nemeth, P; Mikecz, K; Glant, T T; Boldizsar, F

Disease-promoting and -protective genomic loci on mouse chromosomes 3 and 19 control the incidence and severity of autoimmune arthritis.

小鼠 3 号和 19 号染色体上的致病和保护性基因组位点控制着自身免疫性关节炎的发生率和严重程度

Glant T T, Adarichev V A, Boldizsar F, Besenyei T, Laszlo A, Mikecz K, Rauch T A

T cell receptor (TCR) signal strength controls arthritis severity in proteoglycan-specific TCR transgenic mice

T细胞受体(TCR)信号强度控制蛋白聚糖特异性TCR转基因小鼠的关节炎严重程度

Olasz, K; Boldizsar, F; Kis-Toth, K; Tarjanyi, O; Hegyi, A; van Eden, W; Rauch, T A; Mikecz, K; Glant, T T

Osteoarthritis-like damage of cartilage in the temporomandibular joints in mice with autoimmune inflammatory arthritis

患有自身免疫性炎症性关节炎的小鼠颞下颌关节软骨出现类似骨关节炎的损伤

Ghassemi-Nejad, S; Kobezda, T; Rauch, T A; Matesz, C; Glant, T T; Mikecz, K

Congenic strains displaying similar clinical phenotype of arthritis represent different immunologic models of inflammation

表现出相似关节炎临床表型的同源品系代表了不同的炎症免疫学模型。

Adarichev, V A; Vegvari, A; Szabo, Z; Kis-Toth, K; Mikecz, K; Glant, T T

A longitudinal study on an autoimmune murine model of ankylosing spondylitis

一项关于强直性脊柱炎自身免疫小鼠模型的纵向研究

Bárdos, T; Szabó, Z; Czipri, M; Vermes, C; Tunyogi-Csapó, M; Urban, R M; Mikecz, K; Glant, T T

Continuous nasal administration of antigen is critical to maintain tolerance in adoptively transferred autoimmune arthritis in SCID mice

在SCID小鼠中,持续鼻腔给药抗原对于维持过继转移的自身免疫性关节炎的耐受性至关重要。

Bárdos, T; Czipri, M; Vermes, C; Zhang, J; Mikecz, K; Glant, T T

Anti-inflammatory and chondroprotective effect of TSG-6 (tumor necrosis factor-alpha-stimulated gene-6) in murine models of experimental arthritis

TSG-6(肿瘤坏死因子-α刺激基因-6)在小鼠实验性关节炎模型中的抗炎和软骨保护作用

Bárdos, T; Kamath, R V; Mikecz, K; Glant, T T

Complex pattern of Th1 and Th2 activation with a preferential increase of autoreactive Th1 cells in BALB/c mice with proteoglycan (aggrecan)-induced arthritis

BALB/c小鼠中,蛋白聚糖(聚集蛋白聚糖)诱导的关节炎表现出复杂的Th1和Th2激活模式,其中自身反应性Th1细胞优先增加。

Holló, K; Glant, T T; Garzó, M; Finnegan, A; Mikecz, K; Buzás, E

Antigen-specific B cells present cartilage proteoglycan (aggrecan) to an autoreactive T cell hybridoma derived from a mouse with proteoglycan-induced arthritis

抗原特异性B细胞将软骨蛋白聚糖(聚集蛋白聚糖)呈递给源自患有蛋白聚糖诱导性关节炎小鼠的自身反应性T细胞杂交瘤。

Brennan, F R; Mikecz, K; Buzás, E I; Ragasa, D; Cs-Szabó, G; Negroiu, G; Glant, T T