日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas

胰腺癌全基因组关联研究信号的特征分析表明,PDX1能够缓冲外分泌胰腺的压力。

Hoskins, Jason W; Christensen, Trevor A; Eiser, Daina; Char, Erin; Mobaraki, Michael; O'Brien, Aidan; Collins, Irene; Zhong, Jun; Patel, Minal B; Prasad, Gauri; Arda, H Efsun; Connelly, Katelyn E; Amundadottir, Laufey T

Integrative screening identifies functional variants and VNTRs underlying GWAS signals at the 5p15.33 multi-cancer susceptibility locus.

整合筛选可识别 5p15.33 多癌易感位点上 GWAS 信号的功能变异和 VNTR。

O'Brien Aidan, Kong Hyunkyung, Patel Harsh, Ho Michelle, Patel Minal B, Zhong Jun, Xu Mai, Papenberg Brenen W, Connelly Katelyn E, Collins Irene, Hennessey Rebecca, Thakur Rohit, Sowards Hayley, Funderburk Karen, Luong Thong, Florez-Vargas Oscar, Myers Timothy, Jermusyk Ashley, Gorman Bryan, Luo Wen, Jones Kristine, Das Sudipto, Lan Qing, Rothman Nathaniel, McKay James D, Hung Rayjean J, Amos Christopher I, Iles Mark M, Koutros Stella, Landi Maria Teresa, Law Matthew H, Stolzenberg-Solomon Rachael Z, Wolpin Brian M, Hassan Manal, Klein Alison P, Antwi Samuel O, Orr Nick, Chanock Stephen J, Lindström Sara, Hoskins Jason W, Stern Marc-Henri, Andresson Thorkell, Shi Jianxin, Prokunina-Olsson Ludmila, Choi Jiyeon, Brown Kevin M, Amundadottir Laufey T

Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene for pancreatic ductal adenocarcinoma

大规模多组学分析鉴定出非编码体细胞驱动突变,并将ZFP36L2确定为胰腺导管腺癌的驱动基因。

Zhong, Jun; O'Brien, Aidan; Patel, Minal B; Eiser, Daina; Mobaraki, Michael; Collins, Irene; Wang, Li; Guo, Konnie; TruongVo, ThucNhi; Jermusyk, Ashley; Das, Sudipto; O'Neill, Maura; Dill, Courtney D; Wells, Andrew D; Leonard, Michelle E; Pippin, James A; Grant, Struan F A; Zhang, Tongwu; Andresson, Thorkell; Connelly, Katelyn E; Shi, Jianxin; Arda, H Efsun; Hoskins, Jason W; Amundadottir, Laufey T

Ppp1r3b is a metabolic switch that shifts hepatic energy storage from lipid to glycogen.

Ppp1r3b 是一种代谢开关,可将肝脏能量储存从脂质转变为糖原

Creasy Kate Townsend, Mehta Minal B, Schneider Carolin V, Park Joseph, Zhang David, Shewale Swapnil V, Millar John S, Vujkovic Marijana, Hand Nicholas J, Titchenell Paul M, Baur Joseph A, Rader Daniel J

Correction: Signatures of inflammation and impending multiple organ dysfunction in the hyperacute phase of trauma: A prospective cohort study

更正:创伤超急性期炎症和即将发生的多器官功能障碍的特征:一项前瞻性队列研究

Cabrera, Claudia P; Manson, Joanna; Shepherd, Joanna M; Torrance, Hew D; Watson, David; Longhi, M Paula; Hoti, Mimoza; Patel, Minal B; O'Dwyer, Michael; Nourshargh, Sussan; Pennington, Daniel J; Barnes, Michael R; Brohi, Karim

Signatures of inflammation and impending multiple organ dysfunction in the hyperacute phase of trauma: A prospective cohort study

创伤超急性期炎症和即将发生的多器官功能障碍的特征:一项前瞻性队列研究

Claudia P Cabrera, Joanna Manson, Joanna M Shepherd, Hew D Torrance, David Watson, M Paula Longhi, Mimoza Hoti, Minal B Patel, Michael O'Dwyer, Sussan Nourshargh, Daniel J Pennington, Michael R Barnes, Karim Brohi

Hepatic protein phosphatase 1 regulatory subunit 3B (Ppp1r3b) promotes hepatic glycogen synthesis and thereby regulates fasting energy homeostasis

肝蛋白磷酸酶1调节亚基3B(Ppp1r3b)促进肝糖原合成,从而调节禁食能量稳态。

Mehta, Minal B; Shewale, Swapnil V; Sequeira, Raymond N; Millar, John S; Hand, Nicholas J; Rader, Daniel J