Mingxia Bi相关文献与解析，生知库 | 链接生命科学的每一步

Deubiquitylase OTUD3 Mediates Endoplasmic Reticulum Stress through Regulating Fortilin Stability to Restrain Dopaminergic Neurons Apoptosis

去泛素化酶OTUD3通过调节Fortilin稳定性介导内质网应激抑制多巴胺能神经元凋亡

doi:10.3390/antiox12040809

Ling Chen, Xuejie Huan, Fengju Jia, Zhen Zhang, Mingxia Bi, Lin Fu, Xixun Du, Xi Chen, Chunling Yan, Qian Jiao, Hong Jiang

"Sibling" battle or harmony: crosstalk between nesfatin-1 and ghrelin

“兄弟姐妹”之争或和谐：nesfatin-1 与 ghrelin 之间的串扰

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-022-04193-6

Xi Chen, Jing Dong, Qian Jiao, Xixun Du, Mingxia Bi, Hong Jiang

发表日期：2022

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Deficient immunoproteasome assembly drives gain of α-synuclein pathology in Parkinson's disease

免疫蛋白酶体组装缺陷导致帕金森病中 α-突触核蛋白病理增加

期刊:Redox Biology

影响因子:10.7

doi:10.1016/j.redox.2021.102167

Mingxia Bi, Xixun Du, Xue Xiao, Yingying Dai, Qian Jiao, Xi Chen, Lingqiang Zhang, Hong Jiang

Ferroptosis was more initial in cell death caused by iron overload and its underlying mechanism in Parkinson's disease

铁死亡是铁过载导致的细胞死亡的初始阶段，其在帕金森病中的潜在机制

期刊:Free Radical Biology and Medicine

影响因子:7.1

doi:10.1016/j.freeradbiomed.2020.03.015

Pei Zhang, Ling Chen, Qiqi Zhao, Xixun Du, Mingxia Bi, Yong Li, Qian Jiao, Hong Jiang

The S-nitrosylation of parkin attenuated the ubiquitination of divalent metal transporter 1 in MPP+-treated SH-SY5Y cells

parkin 的 S-亚硝化减弱了 MPP+ 处理的 SH-SY5Y 细胞中二价金属转运蛋白 1 的泛素化

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-020-72630-2

Yanmin Zhong #, Xin Li #, Xixun Du, Mingxia Bi, Fengtong Ma, Junxia Xie, Hong Jiang

Investigation of Behavioral Dysfunctions Induced by Monoamine Depletions in a Mouse Model of Parkinson's Disease

帕金森病小鼠模型中单胺类物质耗竭诱发的行为障碍调查

期刊:Frontiers in Cellular Neuroscience

影响因子:4.2

doi:10.3389/fncel.2018.00241

Yong Li, Qian Jiao, Xixun Du, Mingxia Bi, Shuaishuai Han, Lingling Jiao, Hong Jiang

GHSR deficiency exacerbates Parkinson's disease pathology by impairing autophagy

GHSR 缺乏会通过损害自噬加剧帕金森病病理

Astrocyte-derived apolipoprotein D is required for neuronal survival in Parkinson's disease

星形胶质细胞衍生的载脂蛋白 D 是帕金森病中神经元存活所必需的

Deubiquitylase OTUD3 Mediates Endoplasmic Reticulum Stress through Regulating Fortilin Stability to Restrain Dopaminergic Neurons Apoptosis

去泛素化酶OTUD3通过调节Fortilin稳定性介导内质网应激抑制多巴胺能神经元凋亡

"Sibling" battle or harmony: crosstalk between nesfatin-1 and ghrelin

“兄弟姐妹”之争或和谐：nesfatin-1 与 ghrelin 之间的串扰

Deficient immunoproteasome assembly drives gain of α-synuclein pathology in Parkinson's disease

免疫蛋白酶体组装缺陷导致帕金森病中 α-突触核蛋白病理增加

Ferroptosis was more initial in cell death caused by iron overload and its underlying mechanism in Parkinson's disease

铁死亡是铁过载导致的细胞死亡的初始阶段，其在帕金森病中的潜在机制

The S-nitrosylation of parkin attenuated the ubiquitination of divalent metal transporter 1 in MPP+-treated SH-SY5Y cells

parkin 的 S-亚硝化减弱了 MPP+ 处理的 SH-SY5Y 细胞中二价金属转运蛋白 1 的泛素化

Investigation of Behavioral Dysfunctions Induced by Monoamine Depletions in a Mouse Model of Parkinson's Disease

帕金森病小鼠模型中单胺类物质耗竭诱发的行为障碍调查