日期 影响因子
日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:
–

RNA/DNA-binding protein TDP43 regulates DNA mismatch repair genes with implications for genome stability.

RNA/DNA结合蛋白TDP43调控DNA错配修复基因，对基因组稳定性有重要影响。

期刊:Nucleic Acids Research
影响因子:13.1
doi:10.1093/nar/gkaf920
Provasek Vincent E, Bacolla Albino, Rangaswamy Suganya, Kodavati Manohar, Mitra Joy, Yusuf Issa O, Malojirao Vikas H, Vasquez Velmarini, Britz Gavin W, Li Guo-Min, Xu Zuoshang, Mitra Sankar, Garruto Ralph M, Tainer John A, Hegde Muralidhar L
信号转导
TDP43
发表日期：2025
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Characterizing the Repair of DNA Double-Strand Breaks: A Review of Surrogate Plasmid-Based Reporter Methods

DNA双链断裂修复的特征分析：基于替代质粒的报告基因方法综述

期刊:Methods in Molecular Biology
影响因子:
doi:10.1007/978-1-0716-3373-1_11
Dutta, Arijit; Mitra, Joy; Hegde, Pavana M; Mitra, Sankar; Hegde, Muralidhar L
发表日期：2023
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SARS-CoV-2 and the central nervous system: Emerging insights into hemorrhage-associated neurological consequences and therapeutic considerations

SARS-CoV-2 与中枢神经系统：出血相关神经系统后果及治疗考量的新见解

期刊:Ageing Research Reviews
影响因子:12.4
doi:10.1016/j.arr.2022.101687
Mitra, Joy; Kodavati, Manohar; Provasek, Vincent E; Rao, K S; Mitra, Sankar; Hamilton, Dale J; Horner, Philip J; Vahidy, Farhaan S; Britz, Gavin W; Kent, Thomas A; Hegde, Muralidhar L
SARS-CoV-2
神经科学
发表日期：2022
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Fine-tuning of DNA base excision/strand break repair via acetylation

通过乙酰化作用精细调控DNA碱基切除/链断裂修复

期刊:DNA Repair
影响因子:2.7
doi:10.1016/j.dnarep.2020.102931
Bhakat, Kishor K; Sengupta, Shiladitya; Mitra, Sankar
信号转导
乙酰化
表观遗传
发表日期：2020
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Motor neuron disease-associated loss of nuclear TDP-43 is linked to DNA double-strand break repair defects

运动神经元疾病相关的核内TDP-43蛋白丢失与DNA双链断裂修复缺陷有关。

期刊:Proceedings of the National Academy of Sciences of the United States of America
影响因子:9.1
doi:10.1073/pnas.1818415116
Mitra, Joy; Guerrero, Erika N; Hegde, Pavana M; Liachko, Nicole F; Wang, Haibo; Vasquez, Velmarini; Gao, Junling; Pandey, Arvind; Taylor, J Paul; Kraemer, Brian C; Wu, Ping; Boldogh, Istvan; Garruto, Ralph M; Mitra, Sankar; Rao, K S; Hegde, Muralidhar L
神经科学
发表日期：2019
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Microhomology-mediated end joining is activated in irradiated human cells due to phosphorylation-dependent formation of the XRCC1 repair complex

在受辐射的人类细胞中，由于磷酸化依赖性的XRCC1修复复合物的形成，微同源介导的末端连接被激活。

期刊:Nucleic Acids Research
影响因子:13.1
doi:10.1093/nar/gkw1262
Dutta, Arijit; Eckelmann, Bradley; Adhikari, Sanjay; Ahmed, Kazi Mokim; Sengupta, Shiladitya; Pandey, Arvind; Hegde, Pavana M; Tsai, Miaw-Sheue; Tainer, John A; Weinfeld, Michael; Hegde, Muralidhar L; Mitra, Sankar
RCC1
信号转导
磷酸化
细胞生物学
XRCC1
发表日期：2017
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DNA damage responses in central nervous system and age-associated neurodegeneration

中枢神经系统DNA损伤反应与年龄相关的神经退行性变

期刊:Mechanisms of Ageing and Development
影响因子:5.1
doi:10.1016/j.mad.2017.01.010
Hegde, Muralidhar L; Bohr, Vilhelm A; Mitra, Sankar
毒理研究
神经科学
发表日期：2017
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8-Oxoguanine DNA glycosylase1-driven DNA repair-A paradoxical role in lung aging

8-氧鸟嘌呤DNA糖基化酶1驱动的DNA修复——在肺衰老中发挥矛盾作用

期刊:Mechanisms of Ageing and Development
影响因子:5.1
doi:10.1016/j.mad.2016.06.009
German, Peter; Saenz, David; Szaniszlo, Peter; Aguilera-Aguirre, Leopoldo; Pan, Lang; Hegde, Muralidhar L; Bacsi, Attila; Hajas, Gyorgy; Radak, Zsolt; Ba, Xueqing; Mitra, Sankar; Papaconstantinou, John; Boldogh, Istvan
衰老
发表日期：2017
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Chronic oxidative damage together with genome repair deficiency in the neurons is a double whammy for neurodegeneration: Is damage response signaling a potential therapeutic target?

神经元中慢性氧化损伤和基因组修复缺陷是导致神经退行性疾病的双重打击：损伤反应信号是否是潜在的治疗靶点？

期刊:Mechanisms of Ageing and Development
影响因子:5.1
doi:10.1016/j.mad.2016.09.005
Wang, Haibo; Dharmalingam, Prakash; Vasquez, Velmarini; Mitra, Joy; Boldogh, Istvan; Rao, K S; Kent, Thomas A; Mitra, Sankar; Hegde, Muralidhar L
信号转导
毒理研究
神经科学
发表日期：2017
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Pre-Replicative Repair of Oxidized Bases Maintains Fidelity in Mammalian Genomes: The Cowcatcher Role of NEIL1 DNA Glycosylase

氧化碱基的复制前修复维持哺乳动物基因组的保真度：NEIL1 DNA糖基化酶的“牛栏”作用

期刊:Genes
影响因子:2.8
doi:10.3390/genes8070175
Rangaswamy, Suganya; Pandey, Arvind; Mitra, Sankar; Hegde, Muralidhar L
发表日期：2017
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