Mivechi Nahid F相关文献与解析，生知库 | 链接生命科学的每一步

Autophagy and senescence facilitate the development of antiestrogen resistance in ER positive breast cancer

自噬和衰老促进ER阳性乳腺癌抗雌激素耐药性的发展

doi:10.3389/fendo.2024.1298423

McGrath, Michael K; Abolhassani, Ali; Guy, Luke; Elshazly, Ahmed M; Barrett, John T; Mivechi, Nahid F; Gewirtz, David A; Schoenlein, Patricia V

Targeted Deletion of Hsf1, 2, and 4 Genes in Mice

小鼠中 Hsf1、2 和 4 基因的靶向删除

期刊:Methods in Molecular Biology

影响因子:

doi:10.1007/978-1-4939-7477-1_1

Jin, Xiongjie; Eroglu, Binnur; Moskophidis, Demetrius; Mivechi, Nahid F

发表日期：2018

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Epitope-optimized alpha-fetoprotein genetic vaccines prevent carcinogen-induced murine autochthonous hepatocellular carcinoma

表位优化的甲胎蛋白基因疫苗可预防致癌物诱导的小鼠原发性肝细胞癌

期刊:Hepatology

影响因子:15.8

doi:10.1002/hep.26893

Hong, Yuan; Peng, Yibing; Guo, Z Sheng; Guevara-Patino, Jose; Pang, Junfeng; Butterfield, Lisa H; Mivechi, Nahid F; Munn, David H; Bartlett, David L; He, Yukai

Inactivating Mutations in GT198 in Familial and Early-Onset Breast and Ovarian Cancers

家族性和早发性乳腺癌和卵巢癌中GT198的失活突变

期刊:Genes and Cancer

影响因子:

doi:10.1177/1947601913486344

Peng, Min; Bakker, Janine L; Dicioccio, Richard A; Gille, Johan J P; Zhao, Hua; Odunsi, Kunle; Sucheston, Lara; Jaafar, Lahcen; Mivechi, Nahid F; Waisfisz, Quinten; Ko, Lan

GT198 Splice Variants Display Dominant-Negative Activities and Are Induced by Inactivating Mutations

GT198剪接变体表现出显性负性活性，并由失活突变诱导产生。

期刊:Genes and Cancer

影响因子:

doi:10.1177/1947601913486345

Peng, Min; Yang, Zheqiong; Zhang, Hao; Jaafar, Lahcen; Wang, Guanghu; Liu, Min; Flores-Rozas, Hernan; Xu, Jianming; Mivechi, Nahid F; Ko, Lan

发表日期：2013

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Inactivation of heat shock factor Hsf4 induces cellular senescence and suppresses tumorigenesis in vivo

热休克因子Hsf4的失活可诱导细胞衰老并抑制体内肿瘤发生。

期刊:Molecular Cancer Research

影响因子:4.7

doi:10.1158/1541-7786.MCR-11-0530

Jin, Xiongjie; Eroglu, Binnur; Cho, Wonkyoung; Yamaguchi, Yukihiro; Moskophidis, Demetrius; Mivechi, Nahid F

Heat shock factor Hsf1 cooperates with ErbB2 (Her2/Neu) protein to promote mammary tumorigenesis and metastasis

热休克因子Hsf1与ErbB2（Her2/Neu）蛋白协同作用，促进乳腺肿瘤的发生和转移。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M112.377481

Xi, Caixia; Hu, Yanzhong; Buckhaults, Phillip; Moskophidis, Demetrius; Mivechi, Nahid F

Promotion of heat shock factor Hsf1 degradation via adaptor protein filamin A-interacting protein 1-like (FILIP-1L)

通过衔接蛋白丝状蛋白A相互作用蛋白1样蛋白（FILIP-1L）促进热休克因子Hsf1降解

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M111.255851

Hu, Yanzhong; Mivechi, Nahid F

发表日期：2011

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Targeted deletion of Hsf1, 2, and 4 genes in mice

小鼠中 Hsf1、2 和 4 基因的靶向敲除

期刊:Methods in Molecular Biology

影响因子:

doi:10.1007/978-1-61779-295-3_1

Jin, Xiongjie; Eroglu, Binnur; Moskophidis, Demetrius; Mivechi, Nahid F

发表日期：2011

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Development of PolyHis-Targeting PROTAC Degraders

开发针对多聚组氨酸的PROTAC降解剂

Autophagy and senescence facilitate the development of antiestrogen resistance in ER positive breast cancer

自噬和衰老促进ER阳性乳腺癌抗雌激素耐药性的发展

Targeted Deletion of Hsf1, 2, and 4 Genes in Mice

小鼠中 Hsf1、2 和 4 基因的靶向删除

Epitope-optimized alpha-fetoprotein genetic vaccines prevent carcinogen-induced murine autochthonous hepatocellular carcinoma

表位优化的甲胎蛋白基因疫苗可预防致癌物诱导的小鼠原发性肝细胞癌

Inactivating Mutations in GT198 in Familial and Early-Onset Breast and Ovarian Cancers

家族性和早发性乳腺癌和卵巢癌中GT198的失活突变

GT198 Splice Variants Display Dominant-Negative Activities and Are Induced by Inactivating Mutations

GT198剪接变体表现出显性负性活性，并由失活突变诱导产生。

Inactivation of heat shock factor Hsf4 induces cellular senescence and suppresses tumorigenesis in vivo

热休克因子Hsf4的失活可诱导细胞衰老并抑制体内肿瘤发生。

Heat shock factor Hsf1 cooperates with ErbB2 (Her2/Neu) protein to promote mammary tumorigenesis and metastasis

热休克因子Hsf1与ErbB2（Her2/Neu）蛋白协同作用，促进乳腺肿瘤的发生和转移。

Promotion of heat shock factor Hsf1 degradation via adaptor protein filamin A-interacting protein 1-like (FILIP-1L)

通过衔接蛋白丝状蛋白A相互作用蛋白1样蛋白（FILIP-1L）促进热休克因子Hsf1降解

Targeted deletion of Hsf1, 2, and 4 genes in mice

小鼠中 Hsf1、2 和 4 基因的靶向敲除