Miyata Shingo相关文献与解析，生知库 | 链接生命科学的每一步

Involvement of inflammatory responses in the brain to the onset of major depressive disorder due to stress exposure

压力暴露引发的重度抑郁症中，大脑炎症反应参与其中

期刊:Frontiers in Aging Neuroscience

影响因子:4.5

doi:10.3389/fnagi.2022.934346

Miyata, Shingo; Ishino, Yugo; Shimizu, Shoko; Tohyama, Masaya

Effects of 3 Weeks of Water Immersion and Restraint Stress on Sleep in Mice

小鼠经3周水浸和束缚应激后睡眠的影响

期刊:Frontiers in Neuroscience

影响因子:3.2

doi:10.3389/fnins.2019.01072

Yasugaki, Shinnosuke; Liu, Chih-Yao; Kashiwagi, Mitsuaki; Kanuka, Mika; Honda, Takato; Miyata, Shingo; Yanagisawa, Masashi; Hayashi, Yu

发表日期：2019

文献求助收藏

Effects of task-specific paretic ankle plantar flexor training on walking in a stroke patient: a single-case study

针对特定任务的偏瘫侧踝关节跖屈肌训练对中风患者步行能力的影响：单例研究

期刊:Journal of Physical Therapy Science

影响因子:

doi:10.1589/jpts.30.443

Miyata, Shingo; Terada, Shigeru; Matsui, Nobumasa; Uchiyama, Keita

Association between chronic stress-induced structural abnormalities in Ranvier nodes and reduced oligodendrocyte activity in major depression

慢性应激诱导的郎飞氏结结构异常与重度抑郁症中少突胶质细胞活性降低之间的关联

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/srep23084

Miyata, Shingo; Taniguchi, Manabu; Koyama, Yoshihisa; Shimizu, Shoko; Tanaka, Takashi; Yasuno, Fumihiko; Yamamoto, Akihide; Iida, Hidehiro; Kudo, Takashi; Katayama, Taiichi; Tohyama, Masaya

Disturbance of oligodendrocyte function plays a key role in the pathogenesis of schizophrenia and major depressive disorder

少突胶质细胞功能紊乱在精神分裂症和重度抑郁症的发病机制中起着关键作用。

期刊:Biomed Research International

影响因子:2.3

doi:10.1155/2015/492367

Miyata, Shingo; Hattori, Tsuyoshi; Shimizu, Shoko; Ito, Akira; Tohyama, Masaya

(+)-Catechin protects dermal fibroblasts against oxidative stress-induced apoptosis

(+)-儿茶素保护真皮成纤维细胞免受氧化应激诱导的细胞凋亡

期刊:BMC Complementary and Alternative Medicine

影响因子:3.4

doi:10.1186/1472-6882-14-133

Tanigawa, Tomoko; Kanazawa, Shigeyuki; Ichibori, Ryoko; Fujiwara, Takashi; Magome, Takuya; Shingaki, Kenta; Miyata, Shingo; Hata, Yuki; Tomita, Koichi; Matsuda, Ken; Kubo, Tateki; Tohyama, Masaya; Yano, Kenji; Hosokawa, Ko

L-arginine stimulates fibroblast proliferation through the GPRC6A-ERK1/2 and PI3K/Akt pathway

L-精氨酸通过GPRC6A-ERK1/2和PI3K/Akt通路刺激成纤维细胞增殖。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0092168

Fujiwara, Takashi; Kanazawa, Shigeyuki; Ichibori, Ryoko; Tanigawa, Tomoko; Magome, Takuya; Shingaki, Kenta; Miyata, Shingo; Tohyama, Masaya; Hosokawa, Ko

The endoplasmic reticulum-resident chaperone heat shock protein 47 protects the Golgi apparatus from the effects of O-glycosylation inhibition

内质网驻留分子伴侣热休克蛋白47保护高尔基体免受O-糖基化抑制的影响。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0069732

Miyata, Shingo; Mizuno, Tatsunori; Koyama, Yoshihisa; Katayama, Taiichi; Tohyama, Masaya

发表日期：2013

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An Efficient Electroporation Protocol Supporting In Vitro Studies of Oligodendrocyte Biology.

一种高效的电穿孔方案，支持少突胶质细胞生物学的体外研究

Editorial: Oligodendrocytes: from their development to function and dysfunction

社论：少突胶质细胞：从发育到功能及功能障碍

Involvement of inflammatory responses in the brain to the onset of major depressive disorder due to stress exposure

压力暴露引发的重度抑郁症中，大脑炎症反应参与其中

Effects of 3 Weeks of Water Immersion and Restraint Stress on Sleep in Mice

小鼠经3周水浸和束缚应激后睡眠的影响

Effects of task-specific paretic ankle plantar flexor training on walking in a stroke patient: a single-case study

针对特定任务的偏瘫侧踝关节跖屈肌训练对中风患者步行能力的影响：单例研究

Association between chronic stress-induced structural abnormalities in Ranvier nodes and reduced oligodendrocyte activity in major depression

慢性应激诱导的郎飞氏结结构异常与重度抑郁症中少突胶质细胞活性降低之间的关联

Disturbance of oligodendrocyte function plays a key role in the pathogenesis of schizophrenia and major depressive disorder

少突胶质细胞功能紊乱在精神分裂症和重度抑郁症的发病机制中起着关键作用。

(+)-Catechin protects dermal fibroblasts against oxidative stress-induced apoptosis

(+)-儿茶素保护真皮成纤维细胞免受氧化应激诱导的细胞凋亡

L-arginine stimulates fibroblast proliferation through the GPRC6A-ERK1/2 and PI3K/Akt pathway

L-精氨酸通过GPRC6A-ERK1/2和PI3K/Akt通路刺激成纤维细胞增殖。

The endoplasmic reticulum-resident chaperone heat shock protein 47 protects the Golgi apparatus from the effects of O-glycosylation inhibition

内质网驻留分子伴侣热休克蛋白47保护高尔基体免受O-糖基化抑制的影响。