uPAR promotes tumor-like biologic behaviors of fibroblast-like synoviocytes through PI3K/Akt signaling pathway in patients with rheumatoid arthritis.
在类风湿性关节炎患者中,uPAR 通过 PI3K/Akt 信号通路促进成纤维细胞样滑膜细胞的肿瘤样生物学行为
期刊:Cellular & Molecular Immunology
影响因子:19.8
doi:10.1038/cmi.2016.60
Liu Yan, Pan Yun Feng, Xue You-Qiu, Fang Lin-Kai, Guo Xing-Hua, Guo Xin, Liu Meng, Mo Bi-Yao, Yang Meng-Ru, Liu Fang, Wu Yun-Ting, Olsen Nancy, Zheng Song Guo
信号转导
细胞生物学
肿瘤
关节炎
PI3K/Akt
肿瘤免疫