Mo Charlie Y相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

Deep mutational scanning identifies Cas1 and Cas2 variants that enhance type II-A CRISPR-Cas spacer acquisition

深度突变扫描可识别增强 II-A 型 CRISPR-Cas 间隔序列获取的 Cas1 和 Cas2 变体。

期刊:Nature Communications

影响因子:15.7

doi:10.1038/s41467-025-60925-9

Hofmann, Raphael; Herman, Calvin; Mo, Charlie Y; Mathai, Jacob; Marraffini, Luciano A

发表日期：2025

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Bacteriophage infection drives loss of β-lactam resistance in methicillin-resistant Staphylococcus aureus

噬菌体感染导致耐甲氧西林金黄色葡萄球菌丧失β-内酰胺类抗生素耐药性

影响因子:6.4

doi:10.7554/eLife.102743

Tran, My; Hernandez Viera, Angel J; Tran, Patricia Q; Nilsen, Erick D; Tran, Lily; Mo, Charlie Y

发表日期：2025

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Systematic, high-throughput characterization of bacteriophage gene essentiality on diverse hosts.

对不同宿主上噬菌体基因必需性的系统性、高通量表征

doi:10.1101/2024.10.10.617714

Chen Jackie, Nilsen Erick D, Chitboonthavisuk Chutikarn, Mo Charlie Y, Raman Srivatsan

发表日期：2024

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Type III-A CRISPR-Cas Csm Complexes: Assembly, Periodic RNA Cleavage, DNase Activity Regulation, and Autoimmunity

III-A型CRISPR-Cas Csm复合物：组装、周期性RNA切割、DNase活性调控和自身免疫

期刊:Molecular Cell

影响因子:16.6

doi:10.1016/j.molcel.2018.11.007

Jia, Ning; Mo, Charlie Y; Wang, Chongyuan; Eng, Edward T; Marraffini, Luciano A; Patel, Dinshaw J

免疫/内分泌

发表日期：2019

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Advancement of the 5-Amino-1-(Carbamoylmethyl)-1H-1,2,3-Triazole-4-Carboxamide Scaffold to Disarm the Bacterial SOS Response

利用5-氨基-1-(氨甲酰甲基)-1H-1,2,3-三唑-4-甲酰胺骨架抑制细菌SOS反应

期刊:Frontiers in Microbiology

影响因子:4.5

doi:10.3389/fmicb.2018.02961

Selwood, Trevor; Larsen, Brian J; Mo, Charlie Y; Culyba, Matthew J; Hostetler, Zachary M; Kohli, Rahul M; Reitz, Allen B; Baugh, Simon D P

发表日期：2018

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Inhibitors of LexA Autoproteolysis and the Bacterial SOS Response Discovered by an Academic-Industry Partnership

产学研合作发现 LexA 自蛋白水解抑制剂和细菌 SOS 反应抑制剂

期刊:ACS Infectious Diseases

影响因子:3.8

doi:10.1021/acsinfecdis.7b00122

Mo, Charlie Y; Culyba, Matthew J; Selwood, Trevor; Kubiak, Jeffrey M; Hostetler, Zachary M; Jurewicz, Anthony J; Keller, Paul M; Pope, Andrew J; Quinn, Amy; Schneck, Jessica; Widdowson, Katherine L; Kohli, Rahul M

发表日期：2018

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Non-equilibrium repressor binding kinetics link DNA damage dose to transcriptional timing within the SOS gene network.

非平衡阻遏物结合动力学将 DNA 损伤剂量与 SOS 基因网络中的转录时间联系起来

期刊:PLoS Genetics

影响因子:3.7

doi:10.1371/journal.pgen.1007405

Culyba Matthew J, Kubiak Jeffrey M, Mo Charlie Y, Goulian Mark, Kohli Rahul M

发表日期：2018

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Targets for Combating the Evolution of Acquired Antibiotic Resistance

应对获得性抗生素耐药性演变的目标

期刊:Biochemistry

doi:10.1021/acs.biochem.5b00109

Culyba, Matthew J; Mo, Charlie Y; Kohli, Rahul M

发表日期：2015

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High-throughput mutagenesis reveals functional determinants for DNA targeting by activation-induced deaminase

高通量诱变揭示了活化诱导脱氨酶靶向DNA的功能决定因素

期刊:Nucleic Acids Research

影响因子:13.1

doi:10.1093/nar/gku689

Gajula, Kiran S; Huwe, Peter J; Mo, Charlie Y; Crawford, Daniel J; Stivers, James T; Radhakrishnan, Ravi; Kohli, Rahul M

发表日期：2014

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Specificity determinants for autoproteolysis of LexA, a key regulator of bacterial SOS mutagenesis

LexA自蛋白水解的特异性决定因素，LexA是细菌SOS诱变的关键调节因子

期刊:Biochemistry

doi:10.1021/bi500026e

Mo, Charlie Y; Birdwell, L Dillon; Kohli, Rahul M

发表日期：2014

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