Molinari Maurizio相关文献与解析，生知库

Sakai, Yasuyoshi; Behrends, Christian; Cuervo, Ana Maria; Debnath, Jayanta; Izumi, Masanori; Jenny, Andreas; Molinari, Maurizio; Nakamura, Shuhei; Oku, Masahide; Otegui, Marisa S; Santambrogio, Laura; Shen, Han-Ming; Taguchi, Tomohiko; Thumm, Michael; Ushimaru, Takashi; Xie, Zhiping; Reggiori, Fulvio

自噬

发表日期：2026

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Portioning organelles for autophagic clearance

将细胞器进行分拣以进行自噬清除

期刊:Autophagy

影响因子:14.3

doi:10.1080/15548627.2025.2597458

Rudinskiy, Mikhail; Molinari, Maurizio

Microautophagy: current understanding of its molecular mechanisms and functions

微自噬：目前对其分子机制和功能的理解

期刊:Autophagy Reports

影响因子:

doi:10.1080/27694127.2026.2626661

Sakai, Yasuyoshi; Behrends, Christian; Debnath, Jayanta; Izumi, Masanori; Jenny, Andreas; Molinari, Maurizio; Nakamura, Shuhei; Oku, Masahide; Otegui, Marisa S; Santambrogio, Laura; Shen, Han-Ming; Taguchi, Tomohiko; Thumm, Michael; Ushimaru, Takashi; Xie, Zhiping; Cuervo, Ana Maria; Reggiori, Fulvio

自噬

发表日期：2026

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The intrinsically disordered regions of organellophagy receptors are interchangeable and control organelle fragmentation, ER-phagy and mitophagy flux.

细胞器自噬受体的固有无序区是可互换的，并控制细胞器碎片化、内质网自噬和线粒体自噬通量

期刊:Nature Cell Biology

影响因子:19.1

doi:10.1038/s41556-025-01728-4

Rudinskiy Mikhail, Galli Carmela, Raimondi Andrea, Molinari Maurizio

Allosteric Modulation of GCase Enhances Lysosomal Activity and Reduces ER Stress in GCase-Related Disorders.

GCase 的变构调节增强溶酶体活性并降低 GCase 相关疾病中的内质网应激

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms26094392

Fregno Ilaria, PÃ©rez-Carmona Natalia, Rudinskiy Mikhail, SoldÃ Tatiana, Bergmann Timothy J, Ruano Ana, Delgado Aida, Cubero Elena, Bellotto Manolo, GarcÃa-Collazo Ana MarÃa, Molinari Maurizio

免疫/内分泌

发表日期：2025

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A novel allosteric GCase modulator prevents Tau accumulation in GBA1(WT) and GBA1(L444P/L444P) cellular models.

一种新型变构 GCase 调节剂可防止 Tau 在 GBA1(WT) 和 GBA1(L444P/L444P) 细胞模型中积累

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/s41598-025-02346-8

Ciccaldo Matteo, PÃ©rez-Carmona NatÃ lia, Piovesana Ester, Cano-Crespo Sara, Ruano Ana, Delgado Aida, Fregno Ilaria, Calvo-Flores GuzmÃ¡n Beatriz, Bellotto Manolo, Molinari Maurizio, Taylor Joanne, Papin StÃ©phanie, GarcÃa-Collazo Ana MarÃa, Paganetti Paolo

细胞生物学

发表日期：2025

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CNX:FAM134B-driven ERLAD of ATZ polymers proceeds via enhanced formation of VAPA:ORP1L:RAB7 contact sites between ER and endolysosomes

CNX:FAM134B驱动的ATZ聚合物ERLAD是通过增强内质网和内溶酶体之间VAPA:ORP1L:RAB7接触位点的形成而进行的。

期刊:Autophagy Reports

影响因子:

doi:10.1080/27694127.2025.2574355

Fasana, Elisa; Fregno, Ilaria; Molinari, Maurizio

VAPA

发表日期：2025

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Control of nuclear envelope dynamics during acute ER stress by LINC complexes disassembly and selective, asymmetric autophagy of the outer nuclear membrane

急性内质网应激期间，LINC复合物解体和外核膜选择性不对称自噬调控核膜动态变化。

期刊:Autophagy

影响因子:14.3

doi:10.1080/15548627.2023.2299123

Kucińska, Marika K; Molinari, Maurizio

TMX5/TXNDC15, a natural trapping mutant of the PDI family is a client of the proteostatic factor ERp44

TMX5/TXNDC15 是 PDI 家族的一种天然捕获突变体，也是蛋白稳态因子 ERp44 的底物。

期刊:Life Science Alliance

影响因子:2.9

doi:10.26508/lsa.202403047

Soldà, Tatiana; Galli, Carmela; Guerra, Concetta; Hoefner, Carolin; Molinari, Maurizio

发表日期：2024

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ER-phagy: mechanisms, regulation, and diseases connected to the lysosomal clearance of the endoplasmic reticulum

内质网自噬：与溶酶体清除内质网相关的机制、调控和疾病

期刊:Physiological Reviews

影响因子:28.7

doi:10.1152/physrev.00038.2021

Reggiori, Fulvio; Molinari, Maurizio

Microautophagy: definition, classification, and the complexity of the underlying mechanisms

微自噬：定义、分类及其潜在机制的复杂性

Portioning organelles for autophagic clearance

将细胞器进行分拣以进行自噬清除

Microautophagy: current understanding of its molecular mechanisms and functions

微自噬：目前对其分子机制和功能的理解

The intrinsically disordered regions of organellophagy receptors are interchangeable and control organelle fragmentation, ER-phagy and mitophagy flux.

细胞器自噬受体的固有无序区是可互换的，并控制细胞器碎片化、内质网自噬和线粒体自噬通量

Allosteric Modulation of GCase Enhances Lysosomal Activity and Reduces ER Stress in GCase-Related Disorders.

GCase 的变构调节增强溶酶体活性并降低 GCase 相关疾病中的内质网应激

A novel allosteric GCase modulator prevents Tau accumulation in GBA1(WT) and GBA1(L444P/L444P) cellular models.

一种新型变构 GCase 调节剂可防止 Tau 在 GBA1(WT) 和 GBA1(L444P/L444P) 细胞模型中积累

CNX:FAM134B-driven ERLAD of ATZ polymers proceeds via enhanced formation of VAPA:ORP1L:RAB7 contact sites between ER and endolysosomes

CNX:FAM134B驱动的ATZ聚合物ERLAD是通过增强内质网和内溶酶体之间VAPA:ORP1L:RAB7接触位点的形成而进行的。

Control of nuclear envelope dynamics during acute ER stress by LINC complexes disassembly and selective, asymmetric autophagy of the outer nuclear membrane

急性内质网应激期间，LINC复合物解体和外核膜选择性不对称自噬调控核膜动态变化。

TMX5/TXNDC15, a natural trapping mutant of the PDI family is a client of the proteostatic factor ERp44

TMX5/TXNDC15 是 PDI 家族的一种天然捕获突变体，也是蛋白稳态因子 ERp44 的底物。

ER-phagy: mechanisms, regulation, and diseases connected to the lysosomal clearance of the endoplasmic reticulum

内质网自噬：与溶酶体清除内质网相关的机制、调控和疾病