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Corrigendum to "Loss-of-function W4645R mutation in the RyR2-caffeine binding site: implications for synchrony and arrhythmogenesis" [Cell Calcium 123 (2024) 102925]

对“RyR2-咖啡因结合位点功能丧失W4645R突变：对同步性和心律失常的影响”的更正[Cell Calcium 123 (2024) 102925]

期刊:Cell Calcium

影响因子:4

doi:10.1016/j.ceca.2024.102960

Fernández-Morales, José-Carlos; Toth, Noemi; Bayram, Pinar; Rienzo, Taylor; Morad, Martin

GPCR/Calcium/cAMP

发表日期：2024

文献求助收藏

Loss-of-function W4645R mutation in the RyR2-caffeine binding site: implications for synchrony and arrhythmogenesis.

RyR2-咖啡因结合位点的W4645R功能丧失突变：对同步性和心律失常的影响

期刊:Cell Calcium

影响因子:4

doi:10.1016/j.ceca.2024.102925

FernÃ¡ndez-Morales JosÃ©-Carlos, Toth Noemi, Bayram Pinar, Rienzo Taylor, Morad Martin

其它

发表日期：2024

文献求助收藏

Calcium Signaling Consequences of RyR2-S4938F Mutation Expressed in Human iPSC-Derived Cardiomyocytes

人诱导多能干细胞来源的心肌细胞中表达的RyR2-S4938F突变对钙信号传导的影响

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms242015307

Toth, Noemi; Zhang, Xiao-Hua; Zamaro, Alexandra; Morad, Martin

Mutation in RyR2-FKBP Binding site alters Ca(2+) signaling modestly but increases "arrhythmogenesis" in human stem cells derived cardiomyocytes.

RyR2-FKBP 结合位点的突变会轻微改变 Ca(2+) 信号，但会增加人类干细胞衍生的心肌细胞的“心律失常发生”

期刊:Cell Calcium

影响因子:4

doi:10.1016/j.ceca.2021.102500

FernÃ¡ndez-Morales JosÃ©-Carlos, Xia Yanli, Renzo Taylor J, Zhang Xiao-Hua, Morad Martin

Calcium signaling consequences of RyR2 mutations associated with CPVT1 introduced via CRISPR/Cas9 gene editing in human-induced pluripotent stem cell-derived cardiomyocytes: Comparison of RyR2-R420Q, F2483I, and Q4201R

利用 CRISPR/Cas9 基因编辑技术在人诱导多能干细胞来源的心肌细胞中引入与儿茶酚胺敏感性室性心动过速1型 (CPVT1) 相关的 RyR2 突变，并比较 RyR2-R420Q、F2483I 和 Q4201R 突变对钙信号传导的影响。

期刊:Heart Rhythm

影响因子:5.7

doi:10.1016/j.hrthm.2020.09.007

Zhang, Xiao-Hua; Wei, Hua; Xia, Yanli; Morad, Martin

Ca(2+) signaling of human pluripotent stem cells-derived cardiomyocytes as compared to adult mammalian cardiomyocytes

人多能干细胞来源的心肌细胞与成年哺乳动物心肌细胞的Ca(2+)信号传导比较

期刊:Cell Calcium

影响因子:4

doi:10.1016/j.ceca.2020.102244

Zhang, Xiao-Hua; Morad, Martin

Regulation of Ca(2+) signaling by acute hypoxia and acidosis in cardiomyocytes derived from human induced pluripotent stem cells.

急性缺氧和酸中毒对人诱导多能干细胞来源的心肌细胞中 Ca(2+) 信号的调节

期刊:Cell Calcium

影响因子:4

doi:10.1016/j.ceca.2018.12.006

Regulation of Ca(2+) signaling by acute hypoxia and acidosis in rat neonatal cardiomyocytes

急性缺氧和酸中毒对新生大鼠心肌细胞Ca(2+)信号的调节

期刊:Journal of Molecular and Cellular Cardiology

影响因子:4.7

doi:10.1016/j.yjmcc.2017.10.004

Fernández-Morales, José-Carlos; Morad, Martin

CRISPR/Cas9 Gene editing of RyR2 in human stem cell-derived cardiomyocytes provides a novel approach in investigating dysfunctional Ca(2+) signaling.

利用 CRISPR/Cas9 对人类干细胞衍生的心肌细胞中的 RyR2 进行基因编辑，为研究功能失调的 Ca(2+) 信号提供了一种新方法

期刊:Cell Calcium

影响因子:4

doi:10.1016/j.ceca.2018.04.009

Wei Hua, Zhang Xiao-Hua, Clift Cassandra, Yamaguchi Naohiro, Morad Martin

Attempts to Create Transgenic Mice Carrying the Q3924E Mutation in RyR2 Ca(2+) Binding Site

尝试构建携带 RyR2 Ca(2+) 结合位点 Q3924E 突变的转基因小鼠

Corrigendum to "Loss-of-function W4645R mutation in the RyR2-caffeine binding site: implications for synchrony and arrhythmogenesis" [Cell Calcium 123 (2024) 102925]

对“RyR2-咖啡因结合位点功能丧失W4645R突变：对同步性和心律失常的影响”的更正[Cell Calcium 123 (2024) 102925]

Loss-of-function W4645R mutation in the RyR2-caffeine binding site: implications for synchrony and arrhythmogenesis.

RyR2-咖啡因结合位点的W4645R功能丧失突变：对同步性和心律失常的影响

Calcium Signaling Consequences of RyR2-S4938F Mutation Expressed in Human iPSC-Derived Cardiomyocytes

人诱导多能干细胞来源的心肌细胞中表达的RyR2-S4938F突变对钙信号传导的影响

Mutation in RyR2-FKBP Binding site alters Ca(2+) signaling modestly but increases "arrhythmogenesis" in human stem cells derived cardiomyocytes.

RyR2-FKBP 结合位点的突变会轻微改变 Ca(2+) 信号，但会增加人类干细胞衍生的心肌细胞的“心律失常发生”

Calcium signaling consequences of RyR2 mutations associated with CPVT1 introduced via CRISPR/Cas9 gene editing in human-induced pluripotent stem cell-derived cardiomyocytes: Comparison of RyR2-R420Q, F2483I, and Q4201R

利用 CRISPR/Cas9 基因编辑技术在人诱导多能干细胞来源的心肌细胞中引入与儿茶酚胺敏感性室性心动过速1型 (CPVT1) 相关的 RyR2 突变，并比较 RyR2-R420Q、F2483I 和 Q4201R 突变对钙信号传导的影响。

Ca(2+) signaling of human pluripotent stem cells-derived cardiomyocytes as compared to adult mammalian cardiomyocytes

人多能干细胞来源的心肌细胞与成年哺乳动物心肌细胞的Ca(2+)信号传导比较

Regulation of Ca(2+) signaling by acute hypoxia and acidosis in cardiomyocytes derived from human induced pluripotent stem cells.

急性缺氧和酸中毒对人诱导多能干细胞来源的心肌细胞中 Ca(2+) 信号的调节

Regulation of Ca(2+) signaling by acute hypoxia and acidosis in rat neonatal cardiomyocytes

急性缺氧和酸中毒对新生大鼠心肌细胞Ca(2+)信号的调节

CRISPR/Cas9 Gene editing of RyR2 in human stem cell-derived cardiomyocytes provides a novel approach in investigating dysfunctional Ca(2+) signaling.

利用 CRISPR/Cas9 对人类干细胞衍生的心肌细胞中的 RyR2 进行基因编辑，为研究功能失调的 Ca(2+) 信号提供了一种新方法