日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Multiomics Profiling of T-cell Leukemia and Lymphoma Enables Targeted Therapeutic Discovery.

T细胞白血病和淋巴瘤的多组学分析可实现靶向治疗的发现。

Ianevski Aleksandr, Nader Kristen, Nguyen Julia, Sorger Helena, Timonen Sanna, Julia Edith, Pölöske Daniel, Spirk Katrin, Wagner Christina, Jungherz Dennis, Nakano Minoru, Kadambat Nair Sisira, Ianevski Philipp, Kankainen Matti, Dias Diogo, Cichońska Anna, Pemovska Tea, Pirker Christine, Berger Walter, Braun Till, Moriggl Richard, Bachy Emmanuel, Mustjoki Satu, Herling Marco, Neubauer Heidi A, Haibe-Kains Benjamin, Aittokallio Tero

Preclinical models of hepatosplenic γδ T-cell lymphoma with an activating STAT5B mutation display sensitivity to JAK inhibitor upadacitinib

具有激活性 STAT5B 突变的肝脾 γδ T 细胞淋巴瘤临床前模型对 JAK 抑制剂 upadacitinib 敏感

Aung, Myint Myat Khine; Schönefeldt, Susann; Pfalz-Kraupp, Sophie; Wais, Tamara; Suske, Tobias; Zahma, Safia; Franz, Stefan; Müllebner, Andrea; Feurstein, Gabriela; Wagner, Christina; Eder, Thomas; Pemovska, Tea; Agreiter, Christiane; Pichler, Alexander; Staber, Philipp B; Hofer, Martin; Steinborn, Ralf; Strohmer, Svenja-Verena; Simonitsch-Klupp, Ingrid; Bauer, Marcus; Wilfer, Andreas; Weber, Thomas; Potdar, Swapnil; Aittokallio, Tero; Jungherz, Dennis; Müller, Tony A; List, Julia; Gotthardt, Dagmar; Grebien, Florian; Bekiaris, Vasileios; Herling, Marco; Moriggl, Richard; Neubauer, Heidi A

Tyrosine kinase targeting uncovers oncogenic pathway plasticity in Tasmanian devil transmissible cancers.

酪氨酸激酶靶向研究揭示了塔斯马尼亚恶魔传染性癌症的致癌通路可塑性。

Schönbichler Anna, Orlova Anna, Kreindl Carmen, Endler Lukas, Wilson Richard, Kosack Lindsay, Hofmann Anna, Viczenczova Csilla, Darby Jocelyn, Erdogan Fettah, Patchett Amanda L, Koren Anna, Kubicek Stefan, Müller Mathias, Flies Andrew S, Bergthaler Andreas, Moriggl Richard

IQDMA disrupts STAT5 nuclear transport through CDC42-PAK2 axis collapse in cutaneous T-cell lymphoma.

IQDMA 通过 CDC42-PAK2 轴的崩溃破坏 STAT5 核转运,从而导致皮肤 T 细胞淋巴瘤。

Dey Saptaswa, Sorger Helena, Schlederer Michaela, Perchthaler Isabella, Metzelder Martin, Kenner Lukas, Moriggl Richard, Wolf Peter

STAT1/3/5 Functions Driving Lipid and Energy Metabolism in Cancer and Immunity

STAT1/3/5 在癌症和免疫中驱动脂质和能量代谢的功能

Rinnerthaler, Mark; Sykora, Martina; Christoforakos, Anastasios; Aberger, Fritz; Ferbeyre, Gerardo; Moriggl, Richard

A direct effect of the hematocrit on blood glucose: Evidence from hypoxia- and erythropoietin-treated mice.

红细胞比容对血糖的直接影响:来自缺氧和促红细胞生成素治疗的小鼠的证据

Scherer Thomas, Metz Matthäus, Beghini Marianna, Bilban Martin, Gensthaler Lisa, Luca Andreea C, Kaplanian Mairam, Abu Eid Sameer, Koldyka Oliver, Hackl Martina T, Dürr Sabine, Rivelles Elisa, Schönecker Stefanie S, Pöltl Lisa, Kaya Ayperi, Chami Rime, Nusko Laura, Tschare Claudia, Ablaza Kathleen, Höbler Anna-Lena, Klimek Peter, Leutner Michael, Yamamoto Masayuki, Suzuki Norio, Stemmer Kerstin, Zeyda Maximilian, Steinacher Daniel, Nics Lukas, Müller Antonia M S, Helbich Thomas H, Moriggl Richard, Kautzky-Willer Alexandra, Windberger Ursula, Prager Gerhard, Fürnsinn Clemens

Anti-Cancer Potential of a new Derivative of Caffeic Acid Phenethyl Ester targeting the Centrosome

咖啡酸苯乙酯新衍生物靶向中心体的抗癌潜力

Giordano, Catello; Kendler, Jonatan; Sexl, Maximilian; Kollman, Sebastian; Varenicja, Maxim; Szabó, Boglárka; Timelthaler, Gerald; Kirchhofer, Dominik; Hollóczki, Oldamur; Turner, Suzanne D; Moriggl, Richard; Kenner, Lukas; Touaibia, Mohamed; Merkel, Olaf

Cooperative Hedgehog/GLI and JAK/STAT signaling drives immunosuppressive tryptophan/kynurenine metabolism via synergistic induction of IDO1 in skin cancer

在皮肤癌中,Hedgehog/GLI 和 JAK/STAT 信号通路协同作用,通过 IDO1 的协同诱导驱动免疫抑制性色氨酸/犬尿氨酸代谢。

Elmer, Dominik P; Stockmaier, Georg; Grund-Gröschke, Sandra; Strobl, Victoria; Dang, Hieu-Hoa; Wiederstein, Markus; Licha, David; Strobl, Anna; Eglseer, Anna; Sternberg, Christina; Tesanovic, Suzana; Gruber, Wolfgang; Wolff, Florian; Moriggl, Richard; Risch, Angela; Reischl, Roland; Huber, Christian G; Krenn, Peter W; Fortelny, Nikolaus; Horejs-Hoeck, Jutta; Aberger, Fritz

Correction: Weber et al. Stat5 Exerts Distinct, Vital Functions in the Cytoplasm and Nucleus of Bcr-Abl(+) K562 and Jak2(V617F)(+) HEL Leukemia Cells. Cancers 2015, 7, 503-537

更正:Weber 等人,《Stat5 在 Bcr-Abl(+) K562 和 Jak2(V617F)(+) HEL 白血病细胞的细胞质和细胞核中发挥不同的重要功能》,Cancers 2015, 7, 503-537。

Weber, Axel; Borghouts, Corina; Brendel, Christian; Moriggl, Richard; Delis, Natalia; Brill, Boris; Vafaizadeh, Vida; Groner, Bernd

Loss of STAT3 in acute myeloid leukemia favors tissue infiltration linked to CXCR4 signaling

急性髓系白血病中STAT3的缺失促进了与CXCR4信号通路相关的组织浸润。

Zdársky, Bernhard; Edtmayer, Sophie; Witalisz-Siepracka, Agnieszka; Weiss, Stefanie; Boigenzahn, Stefanie; Heindl, Kerstin; Zahma, Safia; Győrffy, Balázs; Moriggl, Richard; Stoiber, Dagmar