Mountz John D相关文献与解析，生知库 | 链接生命科学的每一步

Increased development of T-bet(+)CD11c(+) B cells predisposes to lupus in females: Analysis in BXD2 mouse and genetic crosses

T-bet(+)CD11c(+) B 细胞发育增加易导致女性患狼疮：BXD2 小鼠和遗传杂交分析

doi:10.1016/j.clim.2023.109842

Sullivan, Kathryn A; Chapman, Casey; Lu, Lu; Ashbrook, David G; Wang, Yong; Alduraibi, Fatima K; Lu, Changming; Sun, Chao-Wang; Liu, Shanrun; Williams, Robert W; Mountz, John D; Hsu, Hui-Chen

Host Genetics But Not Commensal Microbiota Determines the Initial Development of Systemic Autoimmune Disease in BXD2 Mice

宿主遗传而非共生微生物群决定了BXD2小鼠系统性自身免疫性疾病的初始发展

期刊:Arthritis & Rheumatology

影响因子:10.9

doi:10.1002/art.42008

Hong, Huixian; Alduraibi, Fatima; Ponder, David; Duck, Wayne L; Morrow, Casey D; Foote, Jeremy B; Schoeb, Trenton R; Fatima, Huma; Elson, Charles O 3rd; Hsu, Hui-Chen; Mountz, John D

Autoreactive B cells in SLE, villains or innocent bystanders?

系统性红斑狼疮中的自身反应性B细胞，是罪魁祸首还是无辜的旁观者？

期刊:Immunological Reviews

影响因子:8.3

doi:10.1111/imr.12815

Hamilton, Jennie A; Hsu, Hui-Chen; Mountz, John D

Dysregulation of T Follicular Helper Cells in Lupus

狼疮中滤泡辅助性T细胞功能失调

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1801150

Mountz, John D; Hsu, Hui-Chen; Ballesteros-Tato, Andre

Editorial: STATus of STAT3 in Psoriatic Arthritis

社论：STAT3在银屑病关节炎中的地位

期刊:Arthritis & Rheumatology

影响因子:10.9

doi:10.1002/art.40445

Interleukin-21 promotes germinal center reaction by skewing the follicular regulatory T cell to follicular helper T cell balance in autoimmune BXD2 mice

白细胞介素-21通过调节自身免疫性BXD2小鼠体内滤泡调节性T细胞与滤泡辅助性T细胞的平衡来促进生发中心反应。

期刊:Arthritis & Rheumatology

影响因子:10.9

doi:10.1002/art.38735

Ding, Yanna; Li, Jun; Yang, PingAr; Luo, Bao; Wu, Qi; Zajac, Allan J; Wildner, Oliver; Hsu, Hui-Chen; Mountz, John D

Inhibition of fucosylation reshapes inflammatory macrophages and suppresses type II collagen-induced arthritis

抑制岩藻糖基化可重塑炎症巨噬细胞并抑制II型胶原诱导的关节炎

期刊:Arthritis & Rheumatology

影响因子:10.9

doi:10.1002/art.38711

Li, Jun; Hsu, Hui-Chen; Ding, Yana; Li, Hao; Wu, Qi; Yang, PingAr; Luo, Bao; Rowse, Amber L; Spalding, David M; Bridges, S Louis Jr; Mountz, John D

Dysregulated cytokine production by dendritic cells modulates B cell responses in the NZM2410 mouse model of lupus

树突状细胞细胞因子生成失调会调节NZM2410小鼠狼疮模型中的B细胞反应。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0102151

Sang, Allison; Zheng, Ying-Yi; Yin, Yiming; Dozmorov, Igor; Li, Hao; Hsu, Hui-Chen; Mountz, John D; Morel, Laurence

Death receptor 5-targeted depletion of interleukin-23-producing macrophages, Th17, and Th1/17 associated with defective tyrosine phosphatase in mice and patients with rheumatoid arthritis.

死亡受体 5 靶向耗竭产生白细胞介素-23 的巨噬细胞、Th17 和 Th1/17，与小鼠和类风湿性关节炎患者中酪氨酸磷酸酶缺陷有关

期刊:Arthritis and Rheumatism

影响因子:

doi:10.1002/art.38057

Li Jun, Yang PingAr, Wu Qi, Li Hao, Ding Yanna, Hsu Hui-Chen, Spalding David M, Mountz John D

IL-4 alters TLR7-induced B cell developmental program in lupus

IL-4 改变狼疮中 TLR7 诱导的 B 细胞发育程序

Increased development of T-bet(+)CD11c(+) B cells predisposes to lupus in females: Analysis in BXD2 mouse and genetic crosses

T-bet(+)CD11c(+) B 细胞发育增加易导致女性患狼疮：BXD2 小鼠和遗传杂交分析

Host Genetics But Not Commensal Microbiota Determines the Initial Development of Systemic Autoimmune Disease in BXD2 Mice

宿主遗传而非共生微生物群决定了BXD2小鼠系统性自身免疫性疾病的初始发展

Autoreactive B cells in SLE, villains or innocent bystanders?

系统性红斑狼疮中的自身反应性B细胞，是罪魁祸首还是无辜的旁观者？

Dysregulation of T Follicular Helper Cells in Lupus

狼疮中滤泡辅助性T细胞功能失调

Editorial: STATus of STAT3 in Psoriatic Arthritis

社论：STAT3在银屑病关节炎中的地位

Interleukin-21 promotes germinal center reaction by skewing the follicular regulatory T cell to follicular helper T cell balance in autoimmune BXD2 mice

白细胞介素-21通过调节自身免疫性BXD2小鼠体内滤泡调节性T细胞与滤泡辅助性T细胞的平衡来促进生发中心反应。

Inhibition of fucosylation reshapes inflammatory macrophages and suppresses type II collagen-induced arthritis

抑制岩藻糖基化可重塑炎症巨噬细胞并抑制II型胶原诱导的关节炎

Dysregulated cytokine production by dendritic cells modulates B cell responses in the NZM2410 mouse model of lupus

树突状细胞细胞因子生成失调会调节NZM2410小鼠狼疮模型中的B细胞反应。

Death receptor 5-targeted depletion of interleukin-23-producing macrophages, Th17, and Th1/17 associated with defective tyrosine phosphatase in mice and patients with rheumatoid arthritis.

死亡受体 5 靶向耗竭产生白细胞介素-23 的巨噬细胞、Th17 和 Th1/17，与小鼠和类风湿性关节炎患者中酪氨酸磷酸酶缺陷有关