Muhammad S Alam相关文献与解析，生知库 | 链接生命科学的每一步

Muhammad S Alam ，Matthias M Gaida ，Hagen R Witzel ，Shizuka Otsuka ，Aamna Abbasi ，Theresa Guerin ，Abdalla Abdelmaksoud ，Nathan Wong ，Margaret C Cam ，Serguei Kozlov ，Jonathan D Ashwell

免疫/内分泌

信号转导

树突状细胞

Calcineurin is an adaptor required for assembly of the TCR signaling complex

钙调磷酸酶是TCR信号复合物组装所必需的接头蛋白。

期刊:Cell Reports

影响因子:7.5

doi:10.1016/j.celrep.2024.114568

Shizuka Otsuka ,Debjani Dutta ,Chuan-Jin Wu ,Muhammad S Alam ,Jonathan D Ashwell

信号转导

发表日期：2024

文献求助收藏

Optineurin Deficiency and Insufficiency Lead to Higher Microglial TDP-43 Protein Levels

视神经素缺乏和不足导致小胶质细胞 TDP-43 蛋白水平升高

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms23126829

Nikolina Prtenjaca, Matea Rob, Muhammad S Alam, Andrea Markovinovic, Cristiana Stuani, Emanuele Buratti, Ivana Munitic

Cocirculation of COVID-19 and dengue: A perspective from Pakistan

新冠病毒与登革热的共同传播：来自巴基斯坦的视角

期刊:Journal of Medical Virology

影响因子:4.6

doi:10.1002/jmv.26567

Rana, Muhammad S; Alam, Muhammad M; Ikram, Aamer; Zaidi, Syed S Z; Salman, Muhammad; Khurshid, Adnan

Unique properties of TCR-activated p38 are necessary for NFAT-dependent T-cell activation

TCR激活的p38的独特特性对于NFAT依赖性T细胞活化是必要的

期刊:PLoS Biology

影响因子:7.8

doi:10.1371/journal.pbio.2004111

Muhammad S Alam, Matthias M Gaida, Subrata Debnath, Harichandra D Tagad, Lisa M Miller Jenkins, Ettore Appella, M Jubayer Rahman, Jonathan D Ashwell

Proximity Ligation Assay (PLA)

邻位连接试验 (PLA)

期刊:Current Protocols in Immunology

Discovery and Characterization of a Biologically Active Non-ATP-Competitive p38 MAP Kinase Inhibitor

发现并表征具有生物活性的非 ATP 竞争性 p38 MAP 激酶抑制剂

期刊:Journal of Biomolecular Screening

影响因子:

doi:10.1177/1087057115615518

Brice A P Wilson, Muhammad S Alam, Tad Guszczynski, Michal Jakob, Shilpa R Shenoy, Carter A Mitchell, Ekaterina I Goncharova, Jason R Evans, Peter Wipf, Gang Liu, Jonathan D Ashwell, Barry R O'Keefe

Selective inhibition of the p38 alternative activation pathway in infiltrating T cells inhibits pancreatic cancer progression

选择性抑制浸润性 T 细胞中的 p38 替代活化途径可抑制胰腺癌进展

期刊:Nature Medicine

影响因子:58.7

doi:10.1038/nm.3957

Muhammad S Alam, Matthias M Gaida, Frank Bergmann, Felix Lasitschka, Thomas Giese, Nathalia A Giese, Thilo Hackert, Ulf Hinz, S Perwez Hussain, Serguei V Kozlov, Jonathan D Ashwell

TNFR1 signaling promotes pancreatic tumor growth by limiting dendritic cell number and function

TNFR1信号通路通过限制树突状细胞的数量和功能来促进胰腺肿瘤的生长。

Calcineurin is an adaptor required for assembly of the TCR signaling complex

钙调磷酸酶是TCR信号复合物组装所必需的接头蛋白。

Optineurin Deficiency and Insufficiency Lead to Higher Microglial TDP-43 Protein Levels

视神经素缺乏和不足导致小胶质细胞 TDP-43 蛋白水平升高

Cocirculation of COVID-19 and dengue: A perspective from Pakistan

新冠病毒与登革热的共同传播：来自巴基斯坦的视角

Unique properties of TCR-activated p38 are necessary for NFAT-dependent T-cell activation

TCR激活的p38的独特特性对于NFAT依赖性T细胞活化是必要的

Proximity Ligation Assay (PLA)

邻位连接试验 (PLA)

Discovery and Characterization of a Biologically Active Non-ATP-Competitive p38 MAP Kinase Inhibitor

发现并表征具有生物活性的非 ATP 竞争性 p38 MAP 激酶抑制剂

Selective inhibition of the p38 alternative activation pathway in infiltrating T cells inhibits pancreatic cancer progression

选择性抑制浸润性 T 细胞中的 p38 替代活化途径可抑制胰腺癌进展