Modulation of the microhomology-mediated end joining pathway suppresses large deletions and enhances homology-directed repair following CRISPR-Cas9-induced DNA breaks
微同源介导的末端连接途径的调控可抑制大片段缺失,并增强 CRISPR-Cas9 诱导的 DNA 断裂后的同源定向修复。
期刊:BMC Biology
影响因子:4.4
doi:10.1186/s12915-024-01896-z
Baolei Yuan #,Chongwei Bi #,Yeteng Tian #,Jincheng Wang,Yiqing Jin,Khaled Alsayegh,Muhammad Tehseen,Gang Yi,Xuan Zhou,Yanjiao Shao,Fernanda Vargas Romero,Wolfgang Fischle,Juan Carlos Izpisua Belmonte,Samir Hamdan,Yanyi Huang,Mo Li