日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Critical role of the β isoform of protein kinase C (PKCβ) in angiotensin II-induced oxidative stress in vascular smooth muscle cells.

蛋白激酶 C (PKCβ) β 亚型在血管平滑肌细胞中血管紧张素 II 诱导的氧化应激中起关键作用。

Tajima Hirotaka, Naganishi Sayaka, Mukohda Masashi, Ishida Mahiro, Hamada Naoki, Shigemi Naoto, Yamasaki Takuma, Nakamura Sho, Matsui Toshiyasu, Mizuno Risuke, Ozaki Hiroshi

Renal Endocytic Regulation of Vitamin D Metabolism during Maturation and Aging in Laying Hens

产蛋母鸡成熟和衰老过程中肾脏内吞对维生素 D 代谢的调节

Nami Kuwata, Hatsune Mukohda, Hiroto Uchida, Ryo Takamatsu, Muhammet Mustafa Binici, Takahisa Yamada, Toshie Sugiyama

Treatment with Ligilactobacillus murinus lowers blood pressure and intestinal permeability in spontaneously hypertensive rats

使用 Ligilactobacillus murinus 治疗可降低自发性高血压大鼠的血压和肠道通透性

Masashi Mukohda, Takanori Yano, Toshiyasu Matsui, Sho Nakamura, Jiro Miyamae, Kensuke Toyama, Ryoji Mitsui, Risuke Mizuno, Hiroshi Ozaki

Failure to vasodilate in response to salt loading blunts renal blood flow and causes salt-sensitive hypertension

盐负荷后血管舒张反应不足会降低肾血流量,导致盐敏感性高血压。

Wu, Jing; Agbor, Larry N; Fang, Shi; Mukohda, Masashi; Nair, Anand R; Nakagawa, Pablo; Sharma, Avika; Morgan, Donald A; Grobe, Justin L; Rahmouni, Kamal; Weiss, Robert M; McCormick, James A; Sigmund, Curt D

Increased Blood Pressure Causes Lymphatic Endothelial Dysfunction via Oxidative Stress in Spontaneously Hypertensive Rats

自发性高血压大鼠血压升高通过氧化应激导致淋巴管内皮功能障碍

Masashi Mukohda, Risuke Mizuno, Hiroshi Ozaki

RhoBTB1 protects against hypertension and arterial stiffness by restraining phosphodiesterase 5 activity

RhoBTB1 通过抑制磷酸二酯酶 5 活性来预防高血压和动脉僵硬

Masashi Mukohda, Shi Fang, Jing Wu, Larry N Agbor, Anand R Nair, Stella-Rita C Ibeawuchi, Chunyan Hu, Xuebo Liu, Ko-Ting Lu, Deng-Fu Guo, Deborah R Davis, Henry L Keen, Frederick W Quelle, Curt D Sigmund

Interference With Endothelial PPAR (Peroxisome Proliferator-Activated Receptor)-γ Causes Accelerated Cerebral Vascular Dysfunction in Response to Endogenous Renin-Angiotensin System Activation

干扰内皮细胞过氧化物酶体增殖物激活受体γ (PPARγ) 会导致内源性肾素-血管紧张素系统激活后脑血管功能障碍加速发生

Nair, Anand R; Agbor, Larry N; Mukohda, Masashi; Liu, Xuebo; Hu, Chunyan; Wu, Jing; Sigmund, Curt D

Hypertension-Causing Mutation in Peroxisome Proliferator-Activated Receptor γ Impairs Nuclear Export of Nuclear Factor-κB p65 in Vascular Smooth Muscle

过氧化物酶体增殖物激活受体γ中的高血压致病突变会损害血管平滑肌中核因子-κB p65的核输出

Mukohda, Masashi; Lu, Ko-Ting; Guo, Deng-Fu; Wu, Jing; Keen, Henry L; Liu, Xuebo; Ketsawatsomkron, Pimonrat; Stump, Madeliene; Rahmouni, Kamal; Quelle, Frederick W; Sigmund, Curt D

Endothelial PPAR-γ provides vascular protection from IL-1β-induced oxidative stress

内皮 PPAR-γ 保护血管免受 IL-1β 诱导的氧化应激

Masashi Mukohda, Madeliene Stump, Pimonrat Ketsawatsomkron, Chunyan Hu, Frederick W Quelle, Curt D Sigmund

Nervous System Expression of PPARγ and Mutant PPARγ Has Profound Effects on Metabolic Regulation and Brain Development

PPARγ 和突变型 PPARγ 在神经系统中的表达对代谢调节和大脑发育有深远的影响

Madeliene Stump, Deng-Fu Guo, Ko-Ting Lu, Masashi Mukohda, Martin D Cassell, Andrew W Norris, Kamal Rahmouni, Curt D Sigmund