Murakami Shunichi相关文献与解析，生知库

FGF and ERK signaling coordinately regulate mineralization-related genes and play essential roles in osteocyte differentiation

FGF和ERK信号通路协同调控矿化相关基因，并在骨细胞分化中发挥关键作用。

期刊:Journal of Bone and Mineral Metabolism

影响因子:2.2

doi:10.1007/s00774-011-0288-2

Kyono, Ai; Avishai, Nanthawan; Ouyang, Zhufeng; Landreth, Gary E; Murakami, Shunichi

NF449 is a novel inhibitor of fibroblast growth factor receptor 3 (FGFR3) signaling active in chondrocytes and multiple myeloma cells.

NF449 是一种新型的成纤维细胞生长因子受体 3 (FGFR3) 信号抑制剂，该信号在软骨细胞和多发性骨髓瘤细胞中具有活性

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M109.083626

Krejci Pavel, Murakami Shunichi, Prochazkova Jirina, Trantirek Lukas, Chlebova Katarina, Ouyang Zhufeng, Aklian Anie, Smutny Jiri, Bryja Vitezslav, Kozubik Alois, Wilcox William R

FGFR3 promotes synchondrosis closure and fusion of ossification centers through the MAPK pathway

FGFR3通过MAPK通路促进软骨结合闭合和骨化中心融合。

期刊:Human Molecular Genetics

影响因子:3.2

doi:10.1093/hmg/ddn339

Matsushita, Takehiko; Wilcox, William R; Chan, Yuk Yu; Kawanami, Aya; Bükülmez, Hülya; Balmes, Gener; Krejci, Pavel; Mekikian, Pertchoui B; Otani, Kazuyuki; Yamaura, Isakichi; Warman, Matthew L; Givol, David; Murakami, Shunichi

Extracellular signal-regulated kinase 1 (ERK1) and ERK2 play essential roles in osteoblast differentiation and in supporting osteoclastogenesis

细胞外信号调节激酶1 (ERK1) 和 ERK2 在成骨细胞分化和支持破骨细胞生成中发挥着至关重要的作用。

期刊:Molecular and Cellular Biology

影响因子:2.7

doi:10.1128/MCB.01549-08

Matsushita, Takehiko; Chan, Yuk Yu; Kawanami, Aya; Balmes, Gener; Landreth, Gary E; Murakami, Shunichi

Constitutive activation of MEK1 in chondrocytes causes Stat1-independent achondroplasia-like dwarfism and rescues the Fgfr3-deficient mouse phenotype

软骨细胞中MEK1的组成型激活会导致Stat1非依赖性的软骨发育不全样侏儒症，并能挽救Fgfr3缺陷小鼠的表型

期刊:Genes & Development

影响因子:7.7

doi:10.1101/gad.1179104

Murakami, Shunichi; Balmes, Gener; McKinney, Sandra; Zhang, Zhaoping; Givol, David; de Crombrugghe, Benoit

Excessive dynamic airway collapse during general anesthesia: a case report

全身麻醉期间过度动态气道塌陷：病例报告

Loss of jab1 in osteochondral progenitor cells severely impairs embryonic limb development in mice

小鼠骨软骨祖细胞中jab1基因的缺失会严重损害胚胎肢体发育。

FGF and ERK signaling coordinately regulate mineralization-related genes and play essential roles in osteocyte differentiation

FGF和ERK信号通路协同调控矿化相关基因，并在骨细胞分化中发挥关键作用。

NF449 is a novel inhibitor of fibroblast growth factor receptor 3 (FGFR3) signaling active in chondrocytes and multiple myeloma cells.

NF449 是一种新型的成纤维细胞生长因子受体 3 (FGFR3) 信号抑制剂，该信号在软骨细胞和多发性骨髓瘤细胞中具有活性

FGFR3 promotes synchondrosis closure and fusion of ossification centers through the MAPK pathway

FGFR3通过MAPK通路促进软骨结合闭合和骨化中心融合。

Extracellular signal-regulated kinase 1 (ERK1) and ERK2 play essential roles in osteoblast differentiation and in supporting osteoclastogenesis

细胞外信号调节激酶1 (ERK1) 和 ERK2 在成骨细胞分化和支持破骨细胞生成中发挥着至关重要的作用。

Constitutive activation of MEK1 in chondrocytes causes Stat1-independent achondroplasia-like dwarfism and rescues the Fgfr3-deficient mouse phenotype

软骨细胞中MEK1的组成型激活会导致Stat1非依赖性的软骨发育不全样侏儒症，并能挽救Fgfr3缺陷小鼠的表型