Murray Lyndsay M相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

Alterations in cardiac function correlate with a disruption in fatty acid metabolism in a mouse model of SMA.

SMA小鼠模型中心脏功能的改变与脂肪酸代谢紊乱相关

期刊:Human Molecular Genetics

影响因子:3.2

doi:10.1093/hmg/ddaf006

Nair Nithya N, Kline Rachel A, Boyd Imogen, Anikumar Meenakshi, Thomson Adrian, Lamont Douglas J, Gray Gillian A, Wishart Thomas M, Murray Lyndsay M

发表日期：2025

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Motor unit recovery following Smn restoration in mouse models of spinal muscular atrophy

脊髓性肌萎缩症小鼠模型中Smn恢复后运动单元的恢复

期刊:Human Molecular Genetics

doi:10.1093/hmg/ddac097

Comley, Laura H; Kline, Rachel A; Thomson, Alison K; Woschitz, Victoria; Landeros, Eric Villalón; Osman, Erkan Y; Lorson, Christian L; Murray, Lyndsay M

发表日期：2022

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Synaptic withdrawal following nerve injury is influenced by postnatal maturity, muscle-specific properties, and the presence of underlying pathology in mice

小鼠神经损伤后的突触撤退受出生后成熟度、肌肉特异性以及潜在病理状况的影响。

期刊:Journal of Anatomy

影响因子:1.9

doi:10.1111/joa.13187

Mole, Alannah J; Bell, Sarah; Thomson, Alison K; Dissanayake, Kosala N; Ribchester, Richard R; Murray, Lyndsay M

发表日期：2020

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Abnormal fatty acid metabolism is a core component of spinal muscular atrophy

脂肪酸代谢异常是脊髓性肌萎缩症的核心组成部分。

期刊:Annals of Clinical and Translational Neurology

影响因子:3.9

doi:10.1002/acn3.50855

Deguise, Marc-Olivier; Baranello, Giovanni; Mastella, Chiara; Beauvais, Ariane; Michaud, Jean; Leone, Alessandro; De Amicis, Ramona; Battezzati, Alberto; Dunham, Christopher; Selby, Kathryn; Warman Chardon, Jodi; McMillan, Hugh J; Huang, Yu-Ting; Courtney, Natalie L; Mole, Alannah J; Kubinski, Sabrina; Claus, Peter; Murray, Lyndsay M; Bowerman, Melissa; Gillingwater, Thomas H; Bertoli, Simona; Parson, Simon H; Kothary, Rashmi

发表日期：2019

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Reduced P53 levels ameliorate neuromuscular junction loss without affecting motor neuron pathology in a mouse model of spinal muscular atrophy

在脊髓性肌萎缩症小鼠模型中，降低 P53 水平可改善神经肌肉接头丢失，而不影响运动神经元病理。

期刊:Cell Death & Disease

doi:10.1038/s41419-019-1727-6

Courtney, Natalie L; Mole, Alannah J; Thomson, Alison K; Murray, Lyndsay M

发表日期：2019

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Comparison of independent screens on differentially vulnerable motor neurons reveals alpha-synuclein as a common modifier in motor neuron diseases

对不同易损性运动神经元的独立筛选结果进行比较，发现α-突触核蛋白是运动神经元疾病中的常见修饰因子。

期刊:PLoS Genetics

影响因子:3.7

doi:10.1371/journal.pgen.1006680

Kline, Rachel A; Kaifer, Kevin A; Osman, Erkan Y; Carella, Francesco; Tiberi, Ariana; Ross, Jolill; Pennetta, Giuseppa; Lorson, Christian L; Murray, Lyndsay M

发表日期：2017

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Myogenic program dysregulation is contributory to disease pathogenesis in spinal muscular atrophy

肌源性程序失调是脊髓性肌萎缩症发病机制的促成因素。

期刊:Human Molecular Genetics

影响因子:3.2

doi:10.1093/hmg/ddu142

Boyer, Justin G; Deguise, Marc-Olivier; Murray, Lyndsay M; Yazdani, Armin; De Repentigny, Yves; Boudreau-Larivière, Céline; Kothary, Rashmi

发表日期：2014

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The Smn-independent beneficial effects of trichostatin A on an intermediate mouse model of spinal muscular atrophy

曲古抑菌素A对脊髓性肌萎缩症中间小鼠模型的Smn非依赖性有益作用

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0101225

Liu, Hong; Yazdani, Armin; Murray, Lyndsay M; Beauvais, Ariane; Kothary, Rashmi

发表日期：2014

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Defects in pancreatic development and glucose metabolism in SMN-depleted mice independent of canonical spinal muscular atrophy neuromuscular pathology

SMN缺失小鼠的胰腺发育和葡萄糖代谢缺陷与典型的脊髓性肌萎缩症神经肌肉病理无关

期刊:Human Molecular Genetics

doi:10.1093/hmg/ddu052

Bowerman, Melissa; Michalski, John-Paul; Beauvais, Ariane; Murray, Lyndsay M; DeRepentigny, Yves; Kothary, Rashmi

发育与干细胞

发表日期：2014

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Fasudil improves survival and promotes skeletal muscle development in a mouse model of spinal muscular atrophy

法舒地尔可提高脊髓性肌萎缩症小鼠模型的存活率并促进骨骼肌发育。

期刊:BMC Medicine

影响因子:8.3

doi:10.1186/1741-7015-10-24

Bowerman, Melissa; Murray, Lyndsay M; Boyer, Justin G; Anderson, Carrie L; Kothary, Rashmi

发育与干细胞

发表日期：2012

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