日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

HMG Box-containing Protein 1 (HBP1) Protects Against Pancreatic Injury in Acute Pancreatitis but Promotes Neoplastic Progression.

含有 HMG 盒的蛋白 1 (HBP1) 可保护急性胰腺炎中的胰腺免受损伤,但会促进肿瘤进展

Lee Seung-Won, Ekstrom Taelor, Manalo Elise C, Ye Shangyuan, Berry Mark, Grygoryev Dmytro, Szczepaniak Malwina, Lee Jinho, Marmolejo Carlos Origel, Haverlack Syber, Lee Juyoung, Shah Vidhi M, Keith Dove, Muschler John L, Chin Koei, Sears Rosalie C, Weisberg Stuart P, Morgan Terry, Kim Jungsun

A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK pathway

一项针对转移性胰腺导管腺癌的窗口期试验揭示了靶向KRAS-MEK通路治疗的耐药机制。

Tsuda, Motoyuki; Keith, Dove; Daniel, Colin J; Pelz, Carl; Blise, Katie E; Ozmen, Tugba Y; Ozmen, Furkan; Hawthone, Kevin; Eng, Jennifer R; Li, Xi; Wang, Xiaoyan; Zimny, Hayley; Sivagnanam, Shamilene; Betre, Konjit; Kirchberger, Nell; Chong, Benson; Lee, Jinho; Matter, Aubry; Smith, Alexander; Agritelley, Ethan S; Waugh, Trent; Sannecy, Ashwin; Alarcon, Kimberly; Youm, Isaac; Protzek, Sara; Egger, Julian; English, Isabel A; Yang, Min; Shah, Vidhi M; Link, Jason M; Creason, Allison L; Worth, Patrick J; Goodyear, Shaun M; Chin, Koei; Muschler, John L; Suciu, Christopher G; Corless, Christopher L; Cao, Sha; Soucek, Laura; Kardosh, Adel; Coussens, Lisa M; Brody, Jonathan R; Lopez, Charles D; Mills, Gordon B; Sears, Rosalie C

Suppression of dystroglycan function accompanies pancreatic acinar-to-ductal metaplasia and favours dysplasia development

肌营养不良蛋白聚糖功能抑制伴随胰腺腺泡-导管化生,并促进发育不良的发生。

Huang, Ge; Ternes, Luke; Lanciault, Christian; MacPherson-Hawthorne, Kevin; Chang, Young Hwan; Sears, Rosalie C; Muschler, John L

Longitudinal Analysis of Human Pancreatic Adenocarcinoma Development Reveals Transient Gene Expression Signatures

人类胰腺腺癌发展纵向分析揭示瞬时基因表达特征

Kim, Jungsun; Ekstrom, Taelor; Yang, Wenli; Donahue, Greg; Grygoryev, Dmytro; Ngo, Thuy T M; Muschler, John L; Morgan, Terry; Zaret, Kenneth S

Endocytic trafficking of laminin is controlled by dystroglycan and is disrupted in cancers

层粘连蛋白的内吞运输受肌营养不良蛋白聚糖控制,并在癌症中受到干扰。

Leonoudakis, Dmitri; Huang, Ge; Akhavan, Armin; Fata, Jimmie E; Singh, Manisha; Gray, Joe W; Muschler, John L

Loss of cell-surface laminin anchoring promotes tumor growth and is associated with poor clinical outcomes

细胞表面层粘连蛋白锚定的丧失会促进肿瘤生长,并与不良临床预后相关。

Akhavan, Armin; Griffith, Obi L; Soroceanu, Liliana; Leonoudakis, Dmitri; Luciani-Torres, Maria Gloria; Daemen, Anneleen; Gray, Joe W; Muschler, John L

Dystroglycan controls signaling of multiple hormones through modulation of STAT5 activity

肌营养不良蛋白聚糖通过调节STAT5活性来控制多种激素的信号传导。

Leonoudakis, Dmitri; Singh, Manisha; Mohajer, Roozbeh; Mohajer, Pouya; Fata, Jimmie E; Campbell, Kevin P; Muschler, John L

Nuclear translocation of beta-dystroglycan reveals a distinctive trafficking pattern of autoproteolyzed mucins

β-肌营养不良蛋白聚糖的核转位揭示了自蛋白水解粘蛋白独特的运输模式

Oppizzi, Maria Luisa; Akhavan, Armin; Singh, Manisha; Fata, Jimmie E; Muschler, John L

Dystroglycan loss disrupts polarity and beta-casein induction in mammary epithelial cells by perturbing laminin anchoring

肌营养不良蛋白聚糖的缺失会扰乱乳腺上皮细胞的极性和β-酪蛋白的诱导,其机制是通过干扰层粘连蛋白的锚定。

Weir, M Lynn; Oppizzi, Maria Luisa; Henry, Michael D; Onishi, Akiko; Campbell, Kevin P; Bissell, Mina J; Muschler, John L