Mussil Bianka相关文献与解析，生知库 | 链接生命科学的每一步

GÃ¼ttler Thomas, Aksu Metin, Dickmanns Antje, Stegmann Kim M, Gregor Kathrin, Rees Renate, Taxer Waltraud, Rymarenko Oleh, SchÃ¼nemann JÃ¼rgen, Dienemann Christian, Gunkel Philip, Mussil Bianka, Krull Jens, Teichmann Ulrike, GroÃ Uwe, Cordes Volker C, Dobbelstein Matthias, GÃ¶rlich Dirk

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发表日期：2021

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Genotoxic stress increases cytoplasmic mitochondrial DNA editing by human APOBEC3 mutator enzymes at a single cell level

基因毒性应激在单细胞水平上增加人APOBEC3突变酶对细胞质线粒体DNA的编辑。

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/s41598-019-39245-8

Mussil, Bianka; Suspène, Rodolphe; Caval, Vincent; Durandy, Anne; Wain-Hobson, Simon; Vartanian, Jean-Pierre

Increased BST2 expression during simian immunodeficiency virus infection is not a determinant of disease progression in rhesus monkeys.

在猴免疫缺陷病毒感染期间，BST2 表达增加并不是恒河猴疾病进展的决定因素

期刊:Retrovirology

影响因子:3.9

doi:10.1186/s12977-015-0219-8

Mussil Bianka, Javed Aneela, TÃ¶pfer Katharina, Sauermann Ulrike, Sopper Sieghart

Human APOBEC3A isoforms translocate to the nucleus and induce DNA double strand breaks leading to cell stress and death

人类APOBEC3A亚型易位至细胞核，诱导DNA双链断裂，导致细胞应激和死亡。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0073641

Mussil, Bianka; Suspène, Rodolphe; Aynaud, Marie-Ming; Gauvrit, Anne; Vartanian, Jean-Pierre; Wain-Hobson, Simon

Increased APOBEC3G and APOBEC3F expression is associated with low viral load and prolonged survival in simian immunodeficiency virus infected rhesus monkeys.

APOBEC3G 和 APOBEC3F 表达增加与感染猴免疫缺陷病毒的恒河猴的病毒载量低和生存期延长有关

期刊:Retrovirology

影响因子:3.9

doi:10.1186/1742-4690-8-77

Mussil Bianka, Sauermann Ulrike, Motzkus Dirk, Stahl-Hennig Christiane, Sopper Sieghart

Neutralization of SARS-CoV-2 by highly potent, hyperthermostable, and mutation-tolerant nanobodies.

利用高效、超耐热、耐突变的纳米抗体中和 SARS-CoV-2

Genotoxic stress increases cytoplasmic mitochondrial DNA editing by human APOBEC3 mutator enzymes at a single cell level

基因毒性应激在单细胞水平上增加人APOBEC3突变酶对细胞质线粒体DNA的编辑。

Increased BST2 expression during simian immunodeficiency virus infection is not a determinant of disease progression in rhesus monkeys.

在猴免疫缺陷病毒感染期间，BST2 表达增加并不是恒河猴疾病进展的决定因素

Human APOBEC3A isoforms translocate to the nucleus and induce DNA double strand breaks leading to cell stress and death

人类APOBEC3A亚型易位至细胞核，诱导DNA双链断裂，导致细胞应激和死亡。

Increased APOBEC3G and APOBEC3F expression is associated with low viral load and prolonged survival in simian immunodeficiency virus infected rhesus monkeys.

APOBEC3G 和 APOBEC3F 表达增加与感染猴免疫缺陷病毒的恒河猴的病毒载量低和生存期延长有关