日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Involvement of nuclear receptor corepressor 2 (NCOR2) in estrogen-induced repression of arcuate Kiss1 expression in female rats

核受体共抑制因子2 (NCOR2)参与雌激素诱导的雌性大鼠弓状核Kiss1表达抑制

Takizawa, Marina; Miyazaki, Sae; Tsuchida, Hitomi; Nagae, Mayuko; Seki, Shunsuke; Hirabayashi, Masumi; Osakada, Fumitaka; Inoue, Naoko; Tsukamura, Hiroko; Uenoyama, Yoshihisa

GPR75 signaling is dispensable for reproduction but contributes to feeding and body growth in rats on normal chow and is involved in high-fat diet-induced hyperphagia, obesity, and hyperglycemia development

GPR75信号通路对于繁殖并非必需,但有助于正常饲料喂养的大鼠的摄食和身体生长,并参与高脂饮食诱导的过度摄食、肥胖和高血糖症的发生发展。

Otsuka, Yuki; Inoue, Naoko; Hazim, Safiullah; Seki, Shunsuke; Tsuchida, Hitomi; Nagae, Mayuko; Sugimoto, Yusuke; Kasugai, Daiki; Yogo, Kyoko; Nakanishi, Marina; Ieda, Nahoko; Goto, Teppei; Hirabayashi, Masumi; Tsukamura, Hiroko; Uenoyama, Yoshihisa

AVPV Kiss1 neuron-specific knockdown of purinergic P2X2 receptor suppresses LH surge and ovulation in Kiss1-Cre rats.

AVPV Kiss1 神经元特异性敲低嘌呤能 P2X2 受体可抑制 Kiss1-Cre 大鼠的 LH 峰值和排卵

Hazim Safiullah, Seki Shunsuke, Yabushita Ryoya, Nagae Mayuko, Tsuchida Hitomi, Hirabayashi Masumi, Uenoyama Yoshihisa, Tsukamura Hiroko, Inoue Naoko

Conditional Oprk1-dependent Kiss1 deletion in kisspeptin neurons caused estrogen-dependent LH pulse disruption and LH surge attenuation in female rats

在kisspeptin神经元中条件性Oprk1依赖性Kiss1缺失导致雌性大鼠LH脉冲紊乱和LH峰值减弱

Nagae, Mayuko; Yamada, Koki; Enomoto, Yuki; Kometani, Mari; Tsuchida, Hitomi; Panthee, Arvinda; Nonogaki, Miku; Matsunaga, Nao; Takizawa, Marina; Matsuzaki, Sena; Hirabayashi, Masumi; Inoue, Naoko; Tsukamura, Hiroko; Uenoyama, Yoshihisa

Sex difference in developmental changes in visualized Kiss1 neurons in newly generated Kiss1-Cre rats

新生成的Kiss1-Cre大鼠中可视化Kiss1神经元发育变化的性别差异

Yamada, Koki; Nagae, Mayuko; Mano, Tetsuya; Tsuchida, Hitomi; Hazim, Safiullah; Goto, Teppei; Sanbo, Makoto; Hirabayashi, Masumi; Inoue, Naoko; Uenoyama, Yoshihisa; Tsukamura, Hiroko

PSAT217 Estrogen Deficiency as a Cause of Decreased 1- alpha - hydroxylase Activity

PSAT217 雌激素缺乏是 1-α-羟化酶活性降低的原因

Uenoyama, Yoshihisa; Tsuchida, Hitomi; Nagae, Mayuko; Inoue, Naoko; Tsukamura, Hiroko; AlAyedh, N; alhadab, a; Cellucci, T; Batthish, M; Heede, S; Zachos, M; Winograd, Deborah; Appelbaum, Heather; Sviderskaya, Tatiana

Direct evidence that KNDy neurons maintain gonadotropin pulses and folliculogenesis as the GnRH pulse generator

直接证据表明,KNDy神经元作为GnRH脉冲发生器,维持促性腺激素脉冲和卵泡发生。

Nagae, Mayuko; Uenoyama, Yoshihisa; Okamoto, Saki; Tsuchida, Hitomi; Ikegami, Kana; Goto, Teppei; Majarune, Sutisa; Nakamura, Sho; Sanbo, Makoto; Hirabayashi, Masumi; Kobayashi, Kenta; Inoue, Naoko; Tsukamura, Hiroko

Role of KNDy Neurons Expressing Kisspeptin, Neurokinin B, and Dynorphin A as a GnRH Pulse Generator Controlling Mammalian Reproduction

表达Kisspeptin、神经激肽B和强啡肽A的KNDy神经元作为促性腺激素释放激素脉冲发生器在哺乳动物生殖中的作用

Uenoyama, Yoshihisa; Nagae, Mayuko; Tsuchida, Hitomi; Inoue, Naoko; Tsukamura, Hiroko

Ad libitum feeding triggers puberty onset associated with increases in arcuate Kiss1 and Pdyn expression in growth-retarded rats

自由采食可诱导生长迟缓大鼠进入青春期,并伴有弓状核Kiss1和Pdyn表达的增加。

Majarune, Sutisa; Nima, Pelden; Sugimoto, Arisa; Nagae, Mayuko; Inoue, Naoko; Tsukamura, Hiroko; Uenoyama, Yoshihisa