Nuclear receptor 4A1 (NR4A1) upregulated by n-butylidenephthalide via the mitogen-activated protein kinase (MAPK) pathway ameliorates drug-induced gingival enlargement.

核受体 4A1 (NR4A1) 通过丝裂原活化蛋白激酶 (MAPK) 通路被 n-丁烯基酞上调，从而改善药物引起的牙龈增生