Neve Kim A相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

Mice carrying the human dopamine D2 receptor pathogenic mutation p.Met374Arg exhibit hyperactivity and aberrant D2 receptor function

携带人类多巴胺D2受体致病突变p.Met374Arg的小鼠表现出活动过度和D2受体功能异常。

期刊:Molecular Pharmacology

doi:10.1016/j.molpha.2025.100080

Rodriguez-Contreras, Dayana; Lebowitz, Joseph J; Reed, Cheryl; Walker-Ziegler, Elizabeth; Buck, David C; Li, Guochuan; Xie, Shu; Li, Yulong; Fedorov, Lev M; Phillips, Tamara J; Williams, John T; Neve, Kim A

发表日期：2025

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Comparison of the function of two novel human dopamine D2 receptor variants identifies a likely mechanism for their pathogenicity.

对两种新型人类多巴胺 D2 受体变体的功能进行比较，揭示了它们致病性的可能机制

期刊:Biochemical Pharmacology

影响因子:5.6

doi:10.1016/j.bcp.2024.116228

Rodriguez-Contreras Dayana, GarcÃa-NafrÃa Javier, Chan Amy E, Shinde Ujwal, Neve Kim A

发表日期：2024

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A Gain-of-Function Variant in Dopamine D2 Receptor and Progressive Chorea and Dystonia Phenotype

多巴胺D2受体功能获得性变异与进行性舞蹈症和肌张力障碍表型

期刊:Movement Disorders

影响因子:7.6

doi:10.1002/mds.28385

van der Weijden, Marlous C M; Rodriguez-Contreras, Dayana; Delnooz, Cathérine C S; Robinson, Brooks G; Condon, Alec F; Kielhold, Michelle L; Stormezand, Gilles N; Ma, Kai Yu; Dufke, Claudia; Williams, John T; Neve, Kim A; Tijssen, Marina A J; Verbeek, Dineke S

发表日期：2021

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Correlated Gene Expression and Anatomical Communication Support Synchronized Brain Activity in the Mouse Functional Connectome

基因表达和解剖结构通讯的相关性支持小鼠功能连接组中同步的脑活动

期刊:Journal of Neuroscience

doi:10.1523/JNEUROSCI.2910-17.2018

Mills, Brian D; Grayson, David S; Shunmugavel, Anandakumar; Miranda-Dominguez, Oscar; Feczko, Eric; Earl, Eric; Neve, Kim A; Fair, Damien A

发表日期：2018

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Cocaine-induced adaptation of dopamine D2S, but not D2L autoreceptors

可卡因诱导多巴胺D2S自身受体（而非D2L自身受体）的适应性变化

影响因子:6.4

doi:10.7554/eLife.31924

Robinson, Brooks G; Condon, Alec F; Radl, Daniela; Borrelli, Emiliana; Williams, John T; Neve, Kim A

发表日期：2017

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DNA synthesis and neuronal apoptosis caused by familial Alzheimer disease mutants of the amyloid precursor protein are mediated by the p21 activated kinase PAK3

家族性阿尔茨海默病中淀粉样前体蛋白突变体引起的DNA合成和神经元凋亡是由p21激活激酶PAK3介导的。

期刊:Journal of Neuroscience

doi:10.1523/JNEUROSCI.23-17-06914.2003

McPhie, Donna L; Coopersmith, Robert; Hines-Peralta, Andrew; Chen, Yuzhi; Ivins, Kathryn J; Manly, Susan P; Kozlowski, Michael R; Neve, Kim A; Neve, Rachael L

阿尔茨海默症

发表日期：2003

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