日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Instantaneous visual genotyping and facile site-specific transgenesis via CRISPR-Cas9 and phiC31 integrase

利用 CRISPR-Cas9 和 phiC31 整合酶实现即时可视化基因分型和简便的位点特异性转基因

Ma, Junyan; Zhang, Weiting; Rahimialiabadi, Simin; Ganesh, Nikkitha Umesh; Sun, Zhengwang; Parvez, Saba; Peterson, Randall T; Yeh, Jing-Ruey Joanna

BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment

BAFF 减弱黑色素瘤肿瘤微环境中的免疫抑制单核细胞

Wei Liu, Paweł Stachura, Haifeng C Xu, Renáta Váraljai, Prashant Shinde, Nikkitha Umesh Ganesh, Matthias Mack, Anke Van Lierop, Anfei Huang, Balamurugan Sundaram, Karl S Lang, Daniel Picard, Ute Fischer, Marc Remke, Bernhard Homey, Alexander Roesch, Dieter Häussinger, Philipp A Lang, Arndt Borkhardt

Ref-1 redox activity alters cancer cell metabolism in pancreatic cancer: exploiting this novel finding as a potential target

Ref-1 氧化还原活性改变胰腺癌中的癌细胞代谢:利用这一新发现作为潜在目标

Silpa Gampala, Fenil Shah, Xiaoyu Lu, Hye-Ran Moon, Olivia Babb, Nikkitha Umesh Ganesh, George Sandusky, Emily Hulsey, Lee Armstrong, Amber L Mosely, Bumsoo Han, Mircea Ivan, Jing-Ruey Joanna Yeh, Mark R Kelley, Chi Zhang, Melissa L Fishel

NDUFS3 depletion permits complex I maturation and reveals TMEM126A/OPA7 as an assembly factor binding the ND4-module intermediate

NDUFS3 的缺失使得复合物 I 得以成熟,并揭示了 TMEM126A/OPA7 作为一种组装因子,能够结合 ND4 模块中间体。

Luigi D'Angelo,Elisa Astro,Monica De Luise,Ivana Kurelac,Nikkitha Umesh-Ganesh,Shujing Ding,Ian M Fearnley,Giuseppe Gasparre,Massimo Zeviani,Anna Maria Porcelli,Erika Fernandez-Vizarra,Luisa Iommarini

The Neglected Liaison: Targeting Cancer Cell Metabolic Reprogramming Modifies the Composition of Non-Malignant Populations of the Tumor Microenvironment

被忽视的纽带:靶向癌细胞代谢重编程可改变肿瘤微环境中非恶性细胞群的组成

Iorio, Maria; Umesh Ganesh, Nikkitha; De Luise, Monica; Porcelli, Anna Maria; Gasparre, Giuseppe; Kurelac, Ivana

Repurposing the serotonin agonist Tegaserod as an anticancer agent in melanoma: molecular mechanisms and clinical implications

将血清素激动剂替加色罗重新用于黑色素瘤抗癌治疗:分子机制和临床意义

Liu, Wei; Stachura, Paweł; Xu, Haifeng C; Umesh Ganesh, Nikkitha; Cox, Fiona; Wang, Ruifeng; Lang, Karl S; Gopalakrishnan, Jay; Häussinger, Dieter; Homey, Bernhard; Lang, Philipp A; Pandyra, Aleksandra A

Inducing cancer indolence by targeting mitochondrial Complex I is potentiated by blocking macrophage-mediated adaptive responses

通过靶向线粒体复合物 I 诱导癌症惰性生长,可通过阻断巨噬细胞介导的适应性反应而增强。

Ivana Kurelac ,Luisa Iommarini ,Renaud Vatrinet ,Laura Benedetta Amato ,Monica De Luise ,Giulia Leone ,Giulia Girolimetti ,Nikkitha Umesh Ganesh ,Victoria Louise Bridgeman ,Luigi Ombrato ,Marta Columbaro ,Moira Ragazzi ,Lara Gibellini ,Manuela Sollazzo ,Rene Gunther Feichtinger ,Silvia Vidali ,Maurizio Baldassarre ,Sarah Foriel ,Michele Vidone ,Andrea Cossarizza ,Daniela Grifoni ,Barbara Kofler ,Ilaria Malanchi ,Anna Maria Porcelli ,Giuseppe Gasparre

A Humanized Bone Niche Model Reveals Bone Tissue Preservation Upon Targeting Mitochondrial Complex I in Pseudo-Orthotopic Osteosarcoma

人源化骨微环境模型揭示靶向线粒体复合物 I 可保护假性原位骨肉瘤中的骨组织

Ivana Kurelac ,Ander Abarrategi ,Moira Ragazzi ,Luisa Iommarini ,Nikkitha Umesh Ganesh ,Thomas Snoeks ,Dominique Bonnet ,Anna Maria Porcelli ,Ilaria Malanchi ,Giuseppe Gasparre