日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

The E3 ubiquitin ligase ITCH negatively regulates intercellular communication via gap junctions by targeting connexin43 for lysosomal degradation

E3 泛素连接酶 ITCH 通过靶向连接蛋白 43 进行溶酶体降解,从而负向调节通过间隙连接的细胞间通讯

Max Zachrisson Totland #, Lars Mørland Knudsen #, Nikoline Lander Rasmussen, Yasufumi Omori, Vigdis Sørensen, Vilde C Wivestad Elster, Jakob Mørkved Stenersen, Mathias Larsen, Caroline Lunder Jensen, Anna A Zickfeldt Lade, Emilie Bruusgaard, Sebastian Basing, Kushtrim Kryeziu, Andreas Brech, Trond A

TBK1 phosphorylation activates LIR-dependent degradation of the inflammation repressor TNIP1

TBK1 磷酸化激活 LIR 依赖性炎症抑制因子 TNIP1 的降解

Jianwen Zhou #, Nikoline Lander Rasmussen #, Hallvard Lauritz Olsvik, Vyacheslav Akimov, Zehan Hu, Gry Evjen, Stéphanie Kaeser-Pebernard, Devanarayanan Siva Sankar, Carole Roubaty, Pauline Verlhac, Nicole van de Beek, Fulvio Reggiori, Yakubu Princely Abudu, Blagoy Blagoev, Trond Lamark, Terje Johans

NBR1: The archetypal selective autophagy receptor

NBR1:典型的选择性自噬受体

Rasmussen, Nikoline Lander; Kournoutis, Athanasios; Lamark, Trond; Johansen, Terje

Regulation of gap junction intercellular communication by connexin ubiquitination: physiological and pathophysiological implications

连接蛋白泛素化调控间隙连接细胞间通讯:生理和病理生理意义

Totland, Max Zachrisson; Rasmussen, Nikoline Lander; Knudsen, Lars Mørland; Leithe, Edward