日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Dimerization of GAS2 mediates crosslinking of microtubules and F-actin.

GAS2 的二聚化介导微管和 F-肌动蛋白的交联

An Jiancheng, Imasaki Tsuyoshi, Narita Akihiro, Niwa Shinsuke, Sasaki Ryohei, Makino Tsukasa, Nitta Ryo, Kikkawa Masahide

Kinesins in Caenorhabditis elegans neuronal morphogenesis

秀丽隐杆线虫神经元形态发生中的驱动蛋白

Niwa, Shinsuke; Chiba, Kyoko

SLC-25A46 regulates mitochondrial fusion through the mitofusin protein FZO-1 and is essential for maintaining neuronal morphology.

SLC-25A46 通过线粒体融合蛋白 FZO-1 调节线粒体融合,对维持神经元形态至关重要

Obinata Hiroyuki, Watanabe Taisei, Takahashi Hironori, Shimo Satoshi, Oda Toshiyuki, Sugimoto Asako, Niwa Shinsuke

Biased movement of monomeric kinesin-3 KLP-6 explained by a symmetric Brownian ratchet model

用对称布朗棘轮模型解释单体驱动蛋白-3 KLP-6 的偏向运动

Kita, Tomoki; Sasaki, Kazuo; Niwa, Shinsuke

The KASH protein UNC-83 differentially regulates kinesin-1 activity to control developmental stage-specific nuclear migration.

KASH 蛋白 UNC-83 差异性地调节驱动蛋白-1 的活性,从而控制发育阶段特异性的核迁移

Gümüşderelioğlu Selin, Sahabandu Natalie, Elnatan Daniel, Gregory Ellen F, Chiba Kyoko, Niwa Shinsuke, Luxton G W Gant, McKenney Richard J, Starr Daniel A

Total Internal Reflection Fluorescence (TIRF) Single-Molecule Assay to Analyze the Motility of Kinesin.

全内反射荧光 (TIRF) 单分子检测分析驱动蛋白的运动性

Kita Tomoki, Niwa Shinsuke

Modeling the motion of disease-associated KIF1A heterodimers

模拟疾病相关KIF1A异二聚体的运动

Kita, Tomoki; Sasaki, Kazuo; Niwa, Shinsuke

De novo mutations in KIF1A-associated neuronal disorder (KAND) dominant-negatively inhibit motor activity and axonal transport of synaptic vesicle precursors

KIF1A相关神经元疾病(KAND)中的新生突变以显性负性方式抑制运动活性和突触小泡前体的轴突运输

Anazawa, Yuzu; Kita, Tomoki; Iguchi, Rei; Hayashi, Kumiko; Niwa, Shinsuke

Synergistic autoinhibition and activation mechanisms control kinesin-1 motor activity

协同的自身抑制和激活机制控制着驱动蛋白-1的运动活性

Chiba, Kyoko; Ori-McKenney, Kassandra M; Niwa, Shinsuke; McKenney, Richard J

An ALS-associated KIF5A mutant forms oligomers and aggregates and induces neuronal toxicity

与肌萎缩侧索硬化症相关的KIF5A突变体形成寡聚体和聚集体,并诱导神经元毒性。

Nakano, Juri; Chiba, Kyoko; Niwa, Shinsuke