日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Therapeutic potential of garlic, aged garlic extract and garlic‑derived compounds on pancreatic cancer (Review)

大蒜、陈年大蒜提取物及大蒜衍生化合物对胰腺癌的治疗潜力(综述)

Kawasaki, Hiromichi; Nussbaum, Gabriel

CD47 and thrombospondin-1 contribute to immune evasion by Porphyromonas gingivalis

CD47 和血小板反应蛋白-1 有助于牙龈卟啉单胞菌逃避免疫反应

Angabo, Sarah; Pandi, Karthikeyan; David, Keren; Steinmetz, Orit; Makkawi, Hasnaa; Farhat, Maria; Eli-Berchoer, Luba; Darawshi, Nadeem; Kawasaki, Hiromichi; Nussbaum, Gabriel

Porphyromonas gingivalis induction of TLR2 association with Vinculin enables PI3K activation and immune evasion

牙龈卟啉单胞菌诱导TLR2与黏着斑蛋白结合,从而激活PI3K并逃避免疫反应。

Pandi, Karthikeyan; Angabo, Sarah; Gnanasekaran, Jeba; Makkawi, Hasnaa; Eli-Berchoer, Luba; Glaser, Fabian; Nussbaum, Gabriel

The cationic liposome CCS/C adjuvant induces immunity to influenza independently of the adaptor protein MyD88

阳离子脂质体CCS/C佐剂可诱导对流感的免疫力,且该作用不依赖于衔接蛋白MyD88。

Even-Or, Orli; Avniel-Polak, Shani; Barenholz, Yechezkel; Nussbaum, Gabriel

Development of a Novel Backbone Cyclic Peptide Inhibitor of the Innate Immune TLR/IL1R Signaling Protein MyD88.

开发一种新型的先天免疫 TLR/IL1R 信号蛋白 MyD88 骨架环肽抑制剂

Dishon Shira, Schumacher Adi, Fanous Joseph, Talhami Alaa, Kassis Ibrahim, Karussis Dimitrios, Gilon Chaim, Hoffman Amnon, Nussbaum Gabriel

Porphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis

牙龈卟啉单胞菌通过操纵补体和TLR信号通路,使细菌清除与炎症脱钩,从而促进菌群失调。

Maekawa, Tomoki; Krauss, Jennifer L; Abe, Toshiharu; Jotwani, Ravi; Triantafilou, Martha; Triantafilou, Kathy; Hashim, Ahmed; Hoch, Shifra; Curtis, Michael A; Nussbaum, Gabriel; Lambris, John D; Hajishengallis, George

Involvement of Toll-like receptors 2 and 4 in the innate immune response to Treponema denticola and its outer sheath components

Toll样受体2和4参与了针对牙龈密螺旋体及其外鞘成分的先天免疫反应。

Nussbaum, Gabriel; Ben-Adi, Shimon; Genzler, Tamar; Sela, Michael; Rosen, Graciela

Induction of IgG3 to LPS via Toll-like receptor 4 co-stimulation.

通过 Toll 样受体 4 共刺激诱导 LPS 产生 IgG3

Quintana Francisco J, Solomon Aderet, Cohen Irun R, Nussbaum Gabriel