日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Therapeutic effect of myogenic cells modified to express neurotrophic factors in a rat model of sciatic nerve injury.

肌源性细胞经修饰表达神经营养因子后对大鼠坐骨神经损伤模型的治疗效果

Dadon-Nachum M, Ben-Zur T, Srugo I, Shamir H M, Melamed E, Yaffe D, Offen D

Intracerebral adult stem cells transplantation increases brain-derived neurotrophic factor levels and protects against phencyclidine-induced social deficit in mice

脑内成体干细胞移植可提高小鼠脑源性神经营养因子水平,并能保护小鼠免受苯环利定诱导的社交障碍。

Barzilay, R; Ben-Zur, T; Sadan, O; Bren, Z; Taler, M; Lev, N; Tarasenko, I; Uzan, R; Gil-Ad, I; Melamed, E; Weizman, A; Offen, D

Bax ablation prevents dopaminergic neurodegeneration in the 1-methyl- 4-phenyl-1,2,3,6-tetrahydropyridine mouse model of Parkinson's disease

在1-甲基-4-苯基-1,2,3,6-四氢吡啶诱导的帕金森病小鼠模型中,Bax基因消融可预防多巴胺能神经退行性变。

Vila, M; Jackson-Lewis, V; Vukosavic, S; Djaldetti, R; Liberatore, G; Offen, D; Korsmeyer, S J; Przedborski, S

6-Hydroxydopamine increases ubiquitin-conjugates and protein degradation: implications for the pathogenesis of Parkinson's disease

6-羟基多巴胺增加泛素结合物和蛋白质降解:对帕金森病发病机制的启示

Elkon, H; Melamed, E; Offen, D

The involvement of p53 in dopamine-induced apoptosis of cerebellar granule neurons and leukemic cells overexpressing p53

p53参与多巴胺诱导的小脑颗粒神经元和过表达p53的白血病细胞的凋亡

Daily, D; Barzilai, A; Offen, D; Kamsler, A; Melamed, E; Ziv, I

Transgenic mice expressing human Bcl-2 in their neurons are resistant to 6-hydroxydopamine and 1-methyl-4-phenyl-1,2,3,6- tetrahydropyridine neurotoxicity

神经元中表达人Bcl-2的转基因小鼠对6-羟基多巴胺和1-甲基-4-苯基-1,2,3,6-四氢吡啶的神经毒性具有抵抗力。

Offen, D; Beart, P M; Cheung, N S; Pascoe, C J; Hochman, A; Gorodin, S; Melamed, E; Bernard, R; Bernard, O

Monoamine-induced apoptotic neuronal cell death

单胺诱导的神经元细胞凋亡

Zilkha-Falb, R; Ziv, I; Nardi, N; Offen, D; Melamed, E; Barzilai, A

Dopamine-induced apoptosis is inhibited in PC12 cells expressing Bcl-2

在表达 Bcl-2 的 PC12 细胞中,多巴胺诱导的细胞凋亡受到抑制

Offen, D; Ziv, I; Panet, H; Wasserman, L; Stein, R; Melamed, E; Barzilai, A

A novel splicing factor is an integral component of 200S large nuclear ribonucleoprotein (InRNP) particles

一种新型剪接因子是200S大核糖核蛋白(InRNP)颗粒的组成部分

Ast, G; Goldblatt, D; Offen, D; Sperling, J; Sperling, R

U1, U2, and U6 small nuclear ribonucleoproteins (snRNPs) are associated with large nuclear RNP particles containing transcripts of an amplified gene in vivo

U1、U2 和 U6 小核核糖核蛋白 (snRNP) 与含有扩增基因转录本的大型核 RNP 颗粒在体内相关联。

Sperling, R; Spann, P; Offen, D; Sperling, J