Ohno Masuo相关文献与解析，生知库 | 链接生命科学的每一步

Accelerated long-term forgetting: A sensitive paradigm for detecting subtle cognitive impairment and evaluating BACE1 inhibitor efficacy in preclinical Alzheimer's disease

加速长期遗忘：一种用于检测轻微认知障碍和评估BACE1抑制剂在阿尔茨海默病临床前期疗效的敏感范式

期刊:Frontiers in Dementia

影响因子:

doi:10.3389/frdem.2023.1161875

Glutamatergic transmission aberration: a major cause of behavioral deficits in a murine model of Down's syndrome

谷氨酸能传递异常：唐氏综合征小鼠模型行为缺陷的主要原因

期刊:Journal of Neuroscience

影响因子:4

doi:10.1523/JNEUROSCI.5338-13.2014

Kaur, Gurjinder; Sharma, Ajay; Xu, Wenjin; Gerum, Scott; Alldred, Melissa J; Subbanna, Shivakumar; Basavarajappa, Balapal S; Pawlik, Monika; Ohno, Masuo; Ginsberg, Stephen D; Wilson, Donald A; Guilfoyle, David N; Levy, Efrat

发表日期：2014

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Reversal of autophagy dysfunction in the TgCRND8 mouse model of Alzheimer's disease ameliorates amyloid pathologies and memory deficits.

在阿尔茨海默病 TgCRND8 小鼠模型中逆转自噬功能障碍可改善淀粉样蛋白病理和记忆缺陷

期刊:Brain

影响因子:11.7

doi:10.1093/brain/awq341

Yang Dun-Sheng, Stavrides Philip, Mohan Panaiyur S, Kaushik Susmita, Kumar Asok, Ohno Masuo, Schmidt Stephen D, Wesson Daniel, Bandyopadhyay Urmi, Jiang Ying, Pawlik Monika, Peterhoff Corrinne M, Yang Austin J, Wilson Donald A, St George-Hyslop Peter, Westaway David, Mathews Paul M, Levy Efrat, Cuervo Ana M, Nixon Ralph A

Impairments in remote memory stabilization precede hippocampal synaptic and cognitive failures in 5XFAD Alzheimer mouse model

在5XFAD阿尔茨海默病小鼠模型中，远期记忆稳定性的损伤先于海马突触和认知功能障碍出现。

期刊:Neurobiology of Disease

影响因子:5.6

doi:10.1016/j.nbd.2008.10.006

Kimura, Ryoichi; Ohno, Masuo

Autophosphorylation of alphaCaMKII is differentially involved in new learning and unlearning mechanisms of memory extinction

αCaMKII的自磷酸化在记忆消退的新学习和遗忘机制中发挥着不同的作用。

期刊:Learning & Memory

影响因子:2.2

doi:10.1101/lm.1049608

Kimura, Ryoichi; Silva, Alcino J; Ohno, Masuo

BACE1 gene deletion prevents neuron loss and memory deficits in 5XFAD APP/PS1 transgenic mice.

BACE1 基因缺失可防止 5XFAD APP/PS1 转基因小鼠出现神经元丢失和记忆缺陷

期刊:Neurobiology of Disease

影响因子:5.6

doi:10.1016/j.nbd.2006.12.008

Ohno Masuo, Cole Sarah L, Yasvoina Marina, Zhao Jie, Citron Martin, Berry Robert, Disterhoft John F, Vassar Robert

神经科学

发表日期：2007

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Intraneuronal beta-amyloid aggregates, neurodegeneration, and neuron loss in transgenic mice with five familial Alzheimer's disease mutations: potential factors in amyloid plaque formation.

携带五种家族性阿尔茨海默病突变的转基因小鼠的神经元内β-淀粉样蛋白聚集体、神经退行性变和神经元丢失：淀粉样斑块形成的潜在因素

期刊:Journal of Neuroscience

影响因子:4

doi:10.1523/JNEUROSCI.1202-06.2006

Oakley Holly, Cole Sarah L, Logan Sreemathi, Maus Erika, Shao Pei, Craft Jeffery, Guillozet-Bongaarts Angela, Ohno Masuo, Disterhoft John, Van Eldik Linda, Berry Robert, Vassar Robert

神经科学

发表日期：2006

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BACE1 at the crossroads of a vicious circle between Alzheimer's disease and diabetes mellitus

BACE1 处于阿尔茨海默病和糖尿病恶性循环的十字路口

Accelerated long-term forgetting: A sensitive paradigm for detecting subtle cognitive impairment and evaluating BACE1 inhibitor efficacy in preclinical Alzheimer's disease

加速长期遗忘：一种用于检测轻微认知障碍和评估BACE1抑制剂在阿尔茨海默病临床前期疗效的敏感范式

Glutamatergic transmission aberration: a major cause of behavioral deficits in a murine model of Down's syndrome

谷氨酸能传递异常：唐氏综合征小鼠模型行为缺陷的主要原因

Reversal of autophagy dysfunction in the TgCRND8 mouse model of Alzheimer's disease ameliorates amyloid pathologies and memory deficits.

在阿尔茨海默病 TgCRND8 小鼠模型中逆转自噬功能障碍可改善淀粉样蛋白病理和记忆缺陷

Impairments in remote memory stabilization precede hippocampal synaptic and cognitive failures in 5XFAD Alzheimer mouse model

在5XFAD阿尔茨海默病小鼠模型中，远期记忆稳定性的损伤先于海马突触和认知功能障碍出现。

Autophosphorylation of alphaCaMKII is differentially involved in new learning and unlearning mechanisms of memory extinction

αCaMKII的自磷酸化在记忆消退的新学习和遗忘机制中发挥着不同的作用。

BACE1 gene deletion prevents neuron loss and memory deficits in 5XFAD APP/PS1 transgenic mice.

BACE1 基因缺失可防止 5XFAD APP/PS1 转基因小鼠出现神经元丢失和记忆缺陷

Intraneuronal beta-amyloid aggregates, neurodegeneration, and neuron loss in transgenic mice with five familial Alzheimer's disease mutations: potential factors in amyloid plaque formation.

携带五种家族性阿尔茨海默病突变的转基因小鼠的神经元内β-淀粉样蛋白聚集体、神经退行性变和神经元丢失：淀粉样斑块形成的潜在因素