Oiso Yutaka相关文献与解析，生知库 | 链接生命科学的每一步

Fujisawa, Haruki; Sugimura, Yoshihisa; Takagi, Hiroshi; Mizoguchi, Hiroyuki; Takeuchi, Hideyuki; Izumida, Hisakazu; Nakashima, Kohtaro; Ochiai, Hiroshi; Takeuchi, Seiji; Kiyota, Atsushi; Fukumoto, Kazuya; Iwama, Shintaro; Takagishi, Yoshiko; Hayashi, Yoshitaka; Arima, Hiroshi; Komatsu, Yukio; Murata, Yoshiharu; Oiso, Yutaka

发表日期：2016

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Secreted factors from dental pulp stem cells improve glucose intolerance in streptozotocin-induced diabetic mice by increasing pancreatic β-cell function

牙髓干细胞分泌因子通过增强胰岛β细胞功能改善链脲佐菌素诱导的糖尿病小鼠的葡萄糖耐受不良。

期刊:Bmj Open Diabetes Research & Care

影响因子:4.1

doi:10.1136/bmjdrc-2015-000128

Izumoto-Akita, Takako; Tsunekawa, Shin; Yamamoto, Akihito; Uenishi, Eita; Ishikawa, Kota; Ogata, Hidetada; Iida, Atsushi; Ikeniwa, Makoto; Hosokawa, Kaori; Niwa, Yasuhiro; Maekawa, Ryuya; Yamauchi, Yuichiro; Seino, Yusuke; Hamada, Yoji; Hibi, Hideharu; Arima, Hiroshi; Ueda, Minoru; Oiso, Yutaka

Angioblast Derived from ES Cells Construct Blood Vessels and Ameliorate Diabetic Polyneuropathy in Mice

源自胚胎干细胞的血管母细胞可构建血管并改善小鼠糖尿病性多发性神经病变

期刊:Journal of Diabetes Research

影响因子:3.4

doi:10.1155/2015/257230

Himeno, Tatsuhito; Kamiya, Hideki; Naruse, Keiko; Cheng, Zhao; Ito, Sachiko; Shibata, Taiga; Kondo, Masaki; Kato, Jiro; Okawa, Tetsuji; Fujiya, Atsushi; Suzuki, Hirohiko; Kito, Tetsutaro; Hamada, Yoji; Oiso, Yutaka; Isobe, Kenichi; Nakamura, Jiro

A novel loss-of-function mutation of GATA3 (p.R299Q) in a Japanese family with Hypoparathyroidism, Deafness, and Renal Dysplasia (HDR) syndrome

日本一个患有甲状旁腺功能减退、耳聋和肾发育不良（HDR）综合征的家族中发现了一种新的GATA3功能缺失突变（p.R299Q）。

期刊:BMC Endocrine Disorders

影响因子:3.3

doi:10.1186/s12902-015-0065-7

Okawa, Tetsuji; Yoshida, Masanori; Usui, Takeshi; Kudou, Takahiro; Iwasaki, Yasumasa; Fukuoka, Kazuki; Takahashi, Norio; Uehara, Yuka; Oiso, Yutaka

Long-term pancreatic beta cell exposure to high levels of glucose but not palmitate induces DNA methylation within the insulin gene promoter and represses transcriptional activity

胰岛β细胞长期暴露于高浓度葡萄糖（而非棕榈酸酯）会诱导胰岛素基因启动子内的DNA甲基化，并抑制其转录活性。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0115350

Ishikawa, Kota; Tsunekawa, Shin; Ikeniwa, Makoto; Izumoto, Takako; Iida, Atsushi; Ogata, Hidetada; Uenishi, Eita; Seino, Yusuke; Ozaki, Nobuaki; Sugimura, Yoshihisa; Hamada, Yoji; Kuroda, Akio; Shinjo, Keiko; Kondo, Yutaka; Oiso, Yutaka

Anti-albuminuric effects of spironolactone in patients with type 2 diabetic nephropathy: a multicenter, randomized clinical trial

螺内酯治疗2型糖尿病肾病患者的抗蛋白尿作用：一项多中心随机临床试验

期刊:Clinical and Experimental Nephrology

影响因子:1.7

doi:10.1007/s10157-015-1106-2

Kato, Sawako; Maruyama, Shoichi; Makino, Hirofumi; Wada, Jun; Ogawa, Daisuke; Uzu, Takashi; Araki, Hisazumi; Koya, Daisuke; Kanasaki, Keizo; Oiso, Yutaka; Goto, Motomitsu; Nishiyama, Akira; Kobori, Hiroyuki; Imai, Enyu; Ando, Masahiko; Matsuo, Seiichi

Therapeutic efficacy of bone marrow-derived mononuclear cells in diabetic polyneuropathy is impaired with aging or diabetes

骨髓单核细胞治疗糖尿病性多发性神经病变的疗效会随着年龄增长或糖尿病的加重而降低。

期刊:

影响因子:

doi:10.1111/jdi.12272

Kondo, Masaki; Kamiya, Hideki; Himeno, Tatsuhito; Naruse, Keiko; Nakashima, Eitaro; Watarai, Atsuko; Shibata, Taiga; Tosaki, Takahiro; Kato, Jiro; Okawa, Tetsuji; Hamada, Yoji; Isobe, Ken-Ichi; Oiso, Yutaka; Nakamura, Jiro

Fructose induces glucose-dependent insulinotropic polypeptide, glucagon-like peptide-1 and insulin secretion: Role of adenosine triphosphate-sensitive K(+) channels

果糖诱导葡萄糖依赖性促胰岛素多肽、胰高血糖素样肽-1和胰岛素分泌：ATP敏感性钾离子通道的作用

期刊:

影响因子:

doi:10.1111/jdi.12356

Seino, Yusuke; Ogata, Hidetada; Maekawa, Ryuya; Izumoto, Takako; Iida, Atsushi; Harada, Norio; Miki, Takashi; Seino, Susumu; Inagaki, Nobuya; Tsunekawa, Shin; Oiso, Yutaka; Hamada, Yoji

免疫/内分泌

发表日期：2015

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Sensory and motor physiological functions are impaired in gastric inhibitory polypeptide receptor-deficient mice

胃抑制多肽受体缺陷小鼠的感觉和运动生理功能受损。

期刊:

影响因子:

doi:10.1111/jdi.12129

Okawa, Tetsuji; Kamiya, Hideki; Himeno, Tatsuhito; Seino, Yusuke; Tsunekawa, Shin; Hayashi, Yoshitaka; Harada, Norio; Yamada, Yuichiro; Inagaki, Nobuya; Seino, Yutaka; Oiso, Yutaka; Nakamura, Jiro

信号转导

发表日期：2014

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Actin dynamics regulated by the balance of neuronal Wiskott-Aldrich syndrome protein (N-WASP) and cofilin activities determines the biphasic response of glucose-induced insulin secretion

神经元Wiskott-Aldrich综合征蛋白（N-WASP）和cofilin活性平衡所调节的肌动蛋白动力学决定了葡萄糖诱导胰岛素分泌的双相反应。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M113.464420

Uenishi, Eita; Shibasaki, Tadao; Takahashi, Harumi; Seki, Chihiro; Hamaguchi, Hitomi; Yasuda, Takao; Tatebe, Masao; Oiso, Yutaka; Takenawa, Tadaomi; Seino, Susumu

Chronic Hyponatremia Causes Neurologic and Psychologic Impairments

慢性低钠血症会导致神经和心理功能障碍

Secreted factors from dental pulp stem cells improve glucose intolerance in streptozotocin-induced diabetic mice by increasing pancreatic β-cell function

牙髓干细胞分泌因子通过增强胰岛β细胞功能改善链脲佐菌素诱导的糖尿病小鼠的葡萄糖耐受不良。

Angioblast Derived from ES Cells Construct Blood Vessels and Ameliorate Diabetic Polyneuropathy in Mice

源自胚胎干细胞的血管母细胞可构建血管并改善小鼠糖尿病性多发性神经病变

A novel loss-of-function mutation of GATA3 (p.R299Q) in a Japanese family with Hypoparathyroidism, Deafness, and Renal Dysplasia (HDR) syndrome

日本一个患有甲状旁腺功能减退、耳聋和肾发育不良（HDR）综合征的家族中发现了一种新的GATA3功能缺失突变（p.R299Q）。

Long-term pancreatic beta cell exposure to high levels of glucose but not palmitate induces DNA methylation within the insulin gene promoter and represses transcriptional activity

胰岛β细胞长期暴露于高浓度葡萄糖（而非棕榈酸酯）会诱导胰岛素基因启动子内的DNA甲基化，并抑制其转录活性。

Anti-albuminuric effects of spironolactone in patients with type 2 diabetic nephropathy: a multicenter, randomized clinical trial

螺内酯治疗2型糖尿病肾病患者的抗蛋白尿作用：一项多中心随机临床试验

Therapeutic efficacy of bone marrow-derived mononuclear cells in diabetic polyneuropathy is impaired with aging or diabetes

骨髓单核细胞治疗糖尿病性多发性神经病变的疗效会随着年龄增长或糖尿病的加重而降低。

Fructose induces glucose-dependent insulinotropic polypeptide, glucagon-like peptide-1 and insulin secretion: Role of adenosine triphosphate-sensitive K(+) channels

果糖诱导葡萄糖依赖性促胰岛素多肽、胰高血糖素样肽-1和胰岛素分泌：ATP敏感性钾离子通道的作用

Sensory and motor physiological functions are impaired in gastric inhibitory polypeptide receptor-deficient mice

胃抑制多肽受体缺陷小鼠的感觉和运动生理功能受损。

Actin dynamics regulated by the balance of neuronal Wiskott-Aldrich syndrome protein (N-WASP) and cofilin activities determines the biphasic response of glucose-induced insulin secretion

神经元Wiskott-Aldrich综合征蛋白（N-WASP）和cofilin活性平衡所调节的肌动蛋白动力学决定了葡萄糖诱导胰岛素分泌的双相反应。