SETBP1 overexpression acts in the place of class-defining mutations to drive FLT3-ITD-mutant AML
SETBP1 过表达可替代类别定义突变,从而驱动 FLT3-ITD 突变型急性髓系白血病 (AML) 的发生。
期刊:Blood Advances
影响因子:7.1
doi:10.1182/bloodadvances.2020003443
Pacharne, Suruchi; Dovey, Oliver M; Cooper, Jonathan L; Gu, Muxin; Friedrich, Mathias J; Rajan, Sandeep S; Barenboim, Maxim; Collord, Grace; Vijayabaskar, M S; Ponstingl, Hannes; De Braekeleer, Etienne; Bautista, Ruben; Mazan, Milena; Rad, Roland; Tzelepis, Konstantinos; Wright, Penny; Gozdecka, Malgorzata; Vassiliou, George S