日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

High-resolution structure of a Na+/H+ antiporter dimer obtained by pulsed electron paramagnetic resonance distance measurements

利用脉冲电子顺磁共振距离测量法获得的Na+/H+反向转运蛋白二聚体的高分辨率结构

Hilger, D; Polyhach, Y; Padan, E; Jung, H; Jeschke, G

Transcription of nhaA, the main Na(+)/H(+) antiporter of Escherichia coli, is regulated by Na(+) and growth phase

大肠杆菌主要Na⁺/H⁺逆向转运蛋白nhaA的转录受Na⁺和生长阶段的调控。

Dover, N; Padan, E

Cyanobacterial sulfide-quinone reductase: cloning and heterologous expression.

蓝藻硫化物醌还原酶:克隆和异源表达

Bronstein M, Schütz M, Hauska G, Padan E, Shahak Y

Projection structure of NhaA, a secondary transporter from Escherichia coli, at 4.0 A resolution

大肠杆菌二级转运蛋白NhaA的投影结构,分辨率为4.0 Å。

Williams, K A; Geldmacher-Kaufer, U; Padan, E; Schuldiner, S; Kühlbrandt, W

Regulation of ntcA expression and nitrite uptake in the marine Synechococcus sp. strain WH 7803

海洋聚球藻WH 7803菌株中ntcA表达和亚硝酸盐吸收的调控

Lindell, D; Padan, E; Post, A F

The Na+-specific interaction between the LysR-type regulator, NhaR, and the nhaA gene encoding the Na+/H+ antiporter of Escherichia coli

大肠杆菌LysR型调节因子NhaR与编码Na+/H+反向转运蛋白的nhaA基因之间存在Na+特异性相互作用

Carmel, O; Rahav-Manor, O; Dover, N; Shaanan, B; Padan, E

A single amino acid substitution (Glu134-->Ala) in NhaR1 increases the inducibility by Na+ of the product of nhaA, a Na+/H+ antiporter gene in Escherichia coli

NhaR1 中的单个氨基酸替换(Glu134→Ala)增加了大肠杆菌中 Na+/H+ 反向转运蛋白基因 nhaA 产物的 Na+ 诱导性。

Carmel, O; Dover, N; Rahav-Manor, O; Dibrov, P; Kirsch, D; Karpel, R; Schuldiner, S; Padan, E

Histidine-226 is part of the pH sensor of NhaA, a Na+/H+ antiporter in Escherichia coli

组氨酸-226是NhaA(大肠杆菌中的一种Na+/H+反向转运蛋白)pH传感器的组成部分。

Gerchman, Y; Olami, Y; Rimon, A; Taglicht, D; Schuldiner, S; Padan, E

Characterization of a Na+/H+ antiporter gene of Escherichia coli

大肠杆菌Na+/H+反向转运蛋白基因的特性分析

Goldberg, E B; Arbel, T; Chen, J; Karpel, R; Mackie, G A; Schuldiner, S; Padan, E

An alkaline shift induces the heat shock response in Escherichia coli

碱性环境变化会诱导大肠杆菌产生热休克反应。

Taglicht, D; Padan, E; Oppenheim, A B; Schuldiner, S