日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

RUVBL1 and RUVBL2 are druggable MYC effector regulators in neuroblastoma cells

RUVBL1 和 RUVBL2 是神经母细胞瘤细胞中可作为药物靶点的 MYC 效应调节因子。

Siaw, Joachim Tetteh; Claeys, Arne; Lai, Wei-Yun; Borenäs, Marcus; Hilgert, Elien; Bekaert, Sarah-Lee; Sanders, Ellen; Kaya, Irem; Van Dorpe, Jo; Speleman, Frank; Durinck, Kaat; Hallberg, Bengt; Palmer, Ruth H; Van den Eynden, Jimmy

Construction and Expression Analysis of the bin (3xOllas) Tool Line

料斗(3xOllas)工具线的构造和表达式分析

Yi, Mengyuan; Sonnenberg, Hannah; Anthonydhason, Vimala; Palmer, Ruth H

Spatial transcriptomics exploration of the primary neuroblastoma microenvironment in archived FFPE samples unveils novel paracrine interactions.

对存档的 FFPE 样本中原发性神经母细胞瘤微环境的空间转录组学探索揭示了新的旁分泌相互作用。

Siaw Joachim T, Merseburger Peter, Borenäs Marcus, Jansson Caroline, Karlsson Jenny, Claeys Arne, Jennische Eva, Lind Dan E, Gisselsson Nord David, Palmer Ruth H, Van den Eynden Jimmy

ALK signaling primes the DNA damage response sensitizing ALK-driven neuroblastoma to therapeutic ATR inhibition

ALK信号通路启动DNA损伤反应,使ALK驱动的神经母细胞瘤对治疗性ATR抑制剂更加敏感。

Borenäs, Marcus; Umapathy, Ganesh; Lind, Dan E; Lai, Wei-Yun; Guan, Jikui; Johansson, Joel; Jennische, Eva; Schmidt, Alexander; Kurhe, Yeshwant; Gabre, Jonatan L; Aniszewska, Agata; Strömberg, Anneli; Bemark, Mats; Hall, Michael N; Eynden, Jimmy Van den; Hallberg, Bengt; Palmer, Ruth H

Preclinical exploration of the DNA damage response pathway using the interactive neuroblastoma cell line explorer CLEAN

利用交互式神经母细胞瘤细胞系探索器 CLEAN 对 DNA 损伤反应通路进行临床前探索

Gabre, Jonatan L; Merseburger, Peter; Claeys, Arne; Siaw, Joachim; Bekaert, Sarah-Lee; Speleman, Frank; Hallberg, Bengt; Palmer, Ruth H; Van den Eynden, Jimmy

ALK fusion NSCLC oncogenes promote survival and inhibit NK cell responses via SERPINB4 expression

ALK融合的非小细胞肺癌癌基因通过SERPINB4表达促进细胞存活并抑制NK细胞反应。

Chuang, Tzu-Po; Lai, Wei-Yun; Gabre, Jonatan L; Lind, Dan E; Umapathy, Ganesh; Bokhari, Abdulmalik A; Bergman, Bengt; Kristenson, Linnea; Thorén, Fredrik B; Le, Anh; Doebele, Robert C; Van den Eynden, Jimmy; Palmer, Ruth H; Hallberg, Bengt

ALK F1174S mutation impairs ALK kinase activity in EML4-ALK variant 1 and sensitizes EML4-ALK variant 3 to crizotinib

ALK F1174S 突变会损害 EML4-ALK 变体 1 中的 ALK 激酶活性,并使 EML4-ALK 变体 3 对克唑替尼敏感。

Guan, Jikui; Chuang, Tzu-Po; Vikström, Anders; Palmer, Ruth H; Hallberg, Bengt

Sustained Response to Entrectinib in an Infant With a Germline ALKAL2 Variant and Refractory Metastatic Neuroblastoma With Chromosomal 2p Gain and Anaplastic Lymphoma Kinase and Tropomyosin Receptor Kinase Activation

一名携带生殖系ALKAL2变异、染色体2p扩增、间变性淋巴瘤激酶和原肌球蛋白受体激酶激活的难治性转移性神经母细胞瘤患儿对恩曲替尼治疗产生持续反应

Treis, Diana; Umapathy, Ganesh; Fransson, Susanne; Guan, Jikui; Mendoza-García, Patricia; Siaw, Joachim T; Wessman, Sandra; Gordon Murkes, Lena; Stenman, Jakob J E; Djos, Anna; Elfman, Lotta H M; Johnsen, John Inge; Hallberg, Bengt; Palmer, Ruth H; Martinsson, Tommy; Kogner, Per

Chromosome Imbalances in Neuroblastoma-Recent Molecular Insight into Chromosome 1p-deletion, 2p-gain, and 11q-deletion Identifies New Friends and Foes for the Future

神经母细胞瘤中的染色体失衡——近期对1p染色体缺失、2p染色体获得和11q染色体缺失的分子机制研究揭示了未来可能面临的新挑战

Guan, Jikui; Hallberg, Bengt; Palmer, Ruth H

Targeting anaplastic lymphoma kinase in neuroblastoma

靶向治疗神经母细胞瘤中的间变性淋巴瘤激酶

Umapathy, Ganesh; Mendoza-Garcia, Patricia; Hallberg, Bengt; Palmer, Ruth H