A Dbf4 mutant contributes to bypassing the Rad53-mediated block of origins of replication in response to genotoxic stress
Dbf4突变体有助于绕过Rad53介导的复制起始点阻滞，以应对基因毒性应激。
期刊:Journal of Biological Chemistry
影响因子:3.9
doi:10.1074/jbc.M110.190843
Duch, Alba; Palou, Gloria; Jonsson, Zophonias O; Palou, Roger; Calvo, Enrique; Wohlschlegel, James; Quintana, David G
