日期:
2020 年 — 2026 年
2020
2021
2022
2023
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2025
2026
影响因子:

Correction: Li et al. The Role of Endothelial L-PGDS in the Pro-Angiogenic and Anti-Inflammatory Effects of Low-Dose Alcohol Consumption. Cells 2024, 13, 2007

更正:Li 等人,《内皮细胞 L-PGDS 在低剂量酒精摄入的促血管生成和抗炎作用中的作用》,Cells 2024, 13, 2007

Li, Jiyu; Li, Chun; Subedi, Utsab; Subedi, Pushpa; Panchatcharam, Manikandan; Sun, Hong

Influence of chronic alcohol consumption on cerebral ischemia/reperfusion injury in female mice.

慢性酒精摄入对雌性小鼠脑缺血/再灌注损伤的影响

Subedi Utsab, Subedi Pushpa, Rogers Asia, Lu Xiao-Hong, Panchatcharam Manikandan, Sun Hong

From Bench to Bedside: The Evolving Landscape of Stem Cell Therapies for Stroke Rehabilitation

从实验室到临床:干细胞疗法在卒中康复中的演变

Waszczuk, Daniel; Manikandan, Shrivats; Manikandan, Varsha; Stone, Erian; Buehre, Shannon; Shore, Steiv; Panchatcharam, Manikandan; Miriyala, Sumitra

The Role of Endothelial L-PGDS in the Pro-Angiogenic and Anti-Inflammatory Effects of Low-Dose Alcohol Consumption.

内皮细胞 L-PGDS 在低剂量酒精摄入的促血管生成和抗炎作用中的作用

Li Jiyu, Li Chun, Subedi Utsab, Subedi Pushpa, Panchatcharam Manikandan, Sun Hong

Electrophilic Aldehyde 4-Hydroxy-2-Nonenal Mediated Signaling and Mitochondrial Dysfunction

亲电醛4-羟基-2-壬烯醛介导的信号传导和线粒体功能障碍

Sharma, Sudha; Sharma, Papori; Bailey, Tara; Bhattarai, Susmita; Subedi, Utsab; Miller, Chloe; Ara, Hosne; Kidambi, Srivatsan; Sun, Hong; Panchatcharam, Manikandan; Miriyala, Sumitra

Disrupted Blood-Brain Barrier and Mitochondrial Impairment by Autotaxin-Lysophosphatidic Acid Axis in Postischemic Stroke

缺血性卒中后,自泌素-溶血磷脂酸轴介导的血脑屏障破坏和线粒体功能障碍

Bhattarai, Susmita; Sharma, Sudha; Ara, Hosne; Subedi, Utsab; Sun, Grace; Li, Chun; Bhuiyan, Md Shenuarin; Kevil, Christopher; Armstrong, William P; Minvielle, Miles T; Miriyala, Sumitra; Panchatcharam, Manikandan

Protective Effect of Low-Dose Alcohol Consumption against Post-Ischemic Neuronal Apoptosis: Role of L-PGDS

低剂量酒精摄入对缺血后神经元凋亡的保护作用:L-PGDS 的作用

Li, Chun; Li, Jiyu; Loreno, Ethyn G; Miriyala, Sumitra; Panchatcharam, Manikandan; Sun, Hong

SOD2 deficiency in cardiomyocytes defines defective mitochondrial bioenergetics as a cause of lethal dilated cardiomyopathy

心肌细胞中SOD2缺乏导致线粒体生物能量学缺陷,而这正是致命性扩张型心肌病的原因之一。

Sharma, Sudha; Bhattarai, Susmita; Ara, Hosne; Sun, Grace; St Clair, Daret K; Bhuiyan, Md Shenuarin; Kevil, Christopher; Watts, Megan N; Dominic, Paari; Shimizu, Takahiko; McCarthy, Kevin J; Sun, Hong; Panchatcharam, Manikandan; Miriyala, Sumitra

Dysfunctional Mitochondrial Dynamic and Oxidative Phosphorylation Precedes Cardiac Dysfunction in R120G-αB-Crystallin-Induced Desmin-Related Cardiomyopathy

R120G-αB-晶状体蛋白诱导的结蛋白相关心肌病中,线粒体动力学和氧化磷酸化功能障碍先于心脏功能障碍发生。

Alam, Shafiul; Abdullah, Chowdhury S; Aishwarya, Richa; Morshed, Mahboob; Nitu, Sadia S; Miriyala, Sumitra; Panchatcharam, Manikandan; Kevil, Christopher G; Orr, A Wayne; Bhuiyan, Md Shenuarin

Cardiac-specific inactivation of LPP3 in mice leads to myocardial dysfunction and heart failure

小鼠心脏特异性敲除LPP3基因会导致心肌功能障碍和心力衰竭。

Chandra, Mini; Escalante-Alcalde, Diana; Bhuiyan, Md Shenuarin; Orr, Anthony Wayne; Kevil, Christopher; Morris, Andrew J; Nam, Hyung; Dominic, Paari; McCarthy, Kevin J; Miriyala, Sumitra; Panchatcharam, Manikandan