Parimal Samir相关文献与解析，生知库 | 链接生命科学的每一步

Ankita Singh #, Awadalkareem Adam #, Aditi, Bi-Hung Peng, Xiaoying Yu, Jing Zou, Vikram V Kulkarni, Peter Kan, Wei Jiang, Pei-Yong Shi, Parimal Samir, Irma Cisneros, Tian Wang

代谢

免疫

心血管

SNIP1 and PRC2 coordinate cell fates of neural progenitors during brain development

SNIP1和PRC2在脑发育过程中协调神经祖细胞的命运

期刊:Nature Communications

影响因子:14.7

doi:10.1038/s41467-023-40487-4

Yurika Matsui ,Mohamed Nadhir Djekidel ,Katherine Lindsay ,Parimal Samir ,Nina Connolly ,Gang Wu ,Xiaoyang Yang ,Yiping Fan ,Beisi Xu ,Jamy C Peng

Brain transcriptomics reveal the activation of neuroinflammation pathways during acute Orientia tsutsugamushi infection in mice

脑转录组学揭示小鼠急性恙虫病东方体感染过程中神经炎症通路的激活

期刊:Frontiers in Immunology

影响因子:5.7

doi:10.3389/fimmu.2023.1194881

Yuejin Liang, Aditi, Florence Onyoni, Hui Wang, Casey Gonzales, Piyanate Sunyakumthorn, Ping Wu, Parimal Samir, Lynn Soong

Immunology/Inflammation

Neuroscience

发表日期：2023

文献求助收藏

Synergism of TNF-α and IFN-γ Triggers Inflammatory Cell Death, Tissue Damage, and Mortality in SARS-CoV-2 Infection and Cytokine Shock Syndromes

TNF-α和IFN-γ的协同作用可引发SARS-CoV-2感染和细胞因子休克综合征中的炎症细胞死亡、组织损伤和死亡

期刊:Cell

影响因子:45.5

doi:10.1016/j.cell.2020.11.025

Rajendra Karki ，Bhesh Raj Sharma ，Shraddha Tuladhar ，Evan Peter Williams ，Lillian Zalduondo ，Parimal Samir ，Min Zheng ，Balamurugan Sundaram ，Balaji Banoth ，R K Subbarao Malireddi ，Patrick Schreiner ，Geoffrey Neale ，Peter Vogel ，Richard Webby ，Colleen Beth Jonsson ，Thirumala-Devi Kanneganti

ADAR1 restricts ZBP1-mediated immune response and PANoptosis to promote tumorigenesis

ADAR1 限制 ZBP1 介导的免疫反应和 PANoptosis 以促进肿瘤发生

期刊:Cell Reports

影响因子:7.5

doi:10.1016/j.celrep.2021.109858

Rajendra Karki, Balamurugan Sundaram, Bhesh Raj Sharma, SangJoon Lee, R K Subbarao Malireddi, Lam Nhat Nguyen, Shelbi Christgen, Min Zheng, Yaqiu Wang, Parimal Samir, Geoffrey Neale, Peter Vogel, Thirumala-Devi Kanneganti

DDX3X coordinates host defense against influenza virus by activating the NLRP3 inflammasome and type I interferon response

DDX3X 通过激活 NLRP3 炎症小体和 I 型干扰素反应来协调宿主对流感病毒的防御

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1016/j.jbc.2021.100579

Sannula Kesavardhana, Parimal Samir, Min Zheng, R K Subbarao Malireddi, Rajendra Karki, Bhesh Raj Sharma, David E Place, Benoit Briard, Peter Vogel, Thirumala-Devi Kanneganti

TLR and IKK Complex-Mediated Innate Immune Signaling Inhibits Stress Granule Assembly

TLR 和 IKK 复合物介导的先天免疫信号抑制应激颗粒组装

期刊:Journal of Immunology

影响因子:3.6

doi:10.4049/jimmunol.2100115

Parimal Samir, David E Place, R K Subbarao Malireddi, Thirumala-Devi Kanneganti

Integrated stress response restricts macrophage necroptosis

综合应激反应限制巨噬细胞坏死性凋亡

期刊:Life Science Alliance

影响因子:3.3

doi:10.26508/lsa.202101260

David E Place, Parimal Samir, Rk Subbarao Malireddi, Thirumala-Devi Kanneganti

Inflammatory Cell Death, PANoptosis, Mediated by Cytokines in Diverse Cancer Lineages Inhibits Tumor Growth

炎症性细胞死亡、PANoptosis，由多种癌症谱系中的细胞因子介导，抑制肿瘤生长

期刊:Immunohorizons

影响因子:

doi:10.4049/immunohorizons.2100059

R K Subbarao Malireddi, Rajendra Karki, Balamurugan Sundaram, Balabhaskararao Kancharana, SangJoon Lee, Parimal Samir, Thirumala-Devi Kanneganti

Galactosaminogalactan activates the inflammasome to provide host protection

半乳糖胺半乳聚糖激活炎症小体，从而提供宿主保护

期刊:Nature

影响因子:50.5

doi:10.1038/s41586-020-2996-z

Benoit Briard,Thierry Fontaine,Parimal Samir,David E Place,Laetitia Muszkieta,R K Subbarao Malireddi,Rajendra Karki,Shelbi Christgen,Perrine Bomme,Peter Vogel,Rémi Beau,Emilia Mellado,Oumaima Ibrahim-Granet,Bernard Henrissat,Ravi C Kalathur,Cam Robinson,Jean-Paul Latgé,Thirumala-Devi Kanneganti

A murine model of post-acute neurological sequelae following SARS-CoV-2 variant infection

SARS-CoV-2 变异感染后急性神经系统后遗症的小鼠模型

SNIP1 and PRC2 coordinate cell fates of neural progenitors during brain development

SNIP1和PRC2在脑发育过程中协调神经祖细胞的命运

Brain transcriptomics reveal the activation of neuroinflammation pathways during acute Orientia tsutsugamushi infection in mice

脑转录组学揭示小鼠急性恙虫病东方体感染过程中神经炎症通路的激活

Synergism of TNF-α and IFN-γ Triggers Inflammatory Cell Death, Tissue Damage, and Mortality in SARS-CoV-2 Infection and Cytokine Shock Syndromes

TNF-α和IFN-γ的协同作用可引发SARS-CoV-2感染和细胞因子休克综合征中的炎症细胞死亡、组织损伤和死亡

ADAR1 restricts ZBP1-mediated immune response and PANoptosis to promote tumorigenesis

ADAR1 限制 ZBP1 介导的免疫反应和 PANoptosis 以促进肿瘤发生

DDX3X coordinates host defense against influenza virus by activating the NLRP3 inflammasome and type I interferon response

DDX3X 通过激活 NLRP3 炎症小体和 I 型干扰素反应来协调宿主对流感病毒的防御

TLR and IKK Complex-Mediated Innate Immune Signaling Inhibits Stress Granule Assembly

TLR 和 IKK 复合物介导的先天免疫信号抑制应激颗粒组装

Integrated stress response restricts macrophage necroptosis

综合应激反应限制巨噬细胞坏死性凋亡

Inflammatory Cell Death, PANoptosis, Mediated by Cytokines in Diverse Cancer Lineages Inhibits Tumor Growth

炎症性细胞死亡、PANoptosis，由多种癌症谱系中的细胞因子介导，抑制肿瘤生长

Galactosaminogalactan activates the inflammasome to provide host protection

半乳糖胺半乳聚糖激活炎症小体，从而提供宿主保护