Park Kwon Moo相关文献与解析，生知库 | 链接生命科学的每一步

Twin Glomeruli: a Newly Discovered Marker of Neonephrogenesis in the Ischemia-Reperfusion Injured Adult Mouse Kidney.

双肾小球：缺血再灌注损伤成年小鼠肾脏中新生肾发生的新发现标志。

期刊:Tissue Engineering and Regenerative Medicine

Hwang Hanguk, Woo Dongju, Park You Ri, Kong Min Jung, Lee Heedong, Park Kwon Moo, Nam Yong Seok, Choi Je-Yong, Nam Sungwook, Nam Eon Jung, Park Sun-Hee, Kim Hongtae, Lee Sang Yeon, Lee Soo Ho, Lim Jeong Ok, Park Mae Ja

Mouse

发表日期：2026

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MON-661 Melanocortin-4 Receptor is Rapidly Ubiquitinated, Exits Primary Cilia and Undergoes Endocytosis After Activation

MON-661 黑皮质素-4 受体激活后迅速泛素化，离开初级纤毛并发生内吞作用

期刊:Redox Biology

影响因子:11.9

doi:10.1016/j.redox.2021.102077

Han, Yong Kwon; Kim, Ji Su; Lee, Gwan Beom; Lim, Jae Hang; Park, Kwon Moo; Diamant, Aristides G; Ojeda-Naharros, Irene; Vaisse, Christian; Nachury, Maxence V

Cilia-deficient renal tubule cells are primed for injury with mitochondrial defects and aberrant tryptophan metabolism.

纤毛缺陷的肾小管细胞由于线粒体缺陷和色氨酸代谢异常而容易受到损伤

期刊:American Journal of Physiology-Renal Physiology

影响因子:3.4

doi:10.1152/ajprenal.00225.2023

Zuo Xiaofeng, Winkler Brennan, Lerner Kasey, Ilatovskaya Daria V, Zamaro Aleksandra S, Dang Yujing, Su Yanhui, Deng Peifeng, Fitzgibbon Wayne, Hartman Jessica, Park Kwon Moo, Lipschutz Joshua H

Macrophages promote Fibrinogenesis during kidney injury

巨噬细胞在肾损伤期间促进纤维蛋白生成

期刊:Frontiers in Medicine

影响因子:3

doi:10.3389/fmed.2023.1206362

Oh, Hanna; Kwon, Ohbin; Kong, Min Jung; Park, Kwon Moo; Baek, Jea-Hyun

Electrophysiological Properties of Ion Channels in Ascaris suum Tissue Incorporated into Planar Lipid Bilayers

猪蛔虫组织中离子通道整合到平面脂双层中的电生理特性

期刊:Korean Journal of Parasitology

影响因子:1.4

doi:10.3347/kjp.2021.59.4.329

Park, Kwon Moo; Kim, Sun-Don; Park, Jin Bong; Hong, Sung-Jong; Ryu, Pan Dong

发表日期：2021

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Fragmentation of kidney epithelial cell primary cilia occurs by cisplatin and these cilia fragments are excreted into the urine

顺铂会使肾脏上皮细胞原发纤毛碎裂，这些纤毛碎片会排泄到尿液中

期刊:Redox Biology

影响因子:10.7

doi:10.1016/j.redox.2018.09.017

Min Jung Kong, Sang Hong Bak, Ki-Hwan Han, Jee In Kim, Jeen-Woo Park, Kwon Moo Park

Mitochondrial NADP+-dependent isocitrate dehydrogenase deficiency increases cisplatin-induced oxidative damage in the kidney tubule cells

线粒体 NADP+ 依赖性异柠檬酸脱氢酶缺乏会增加顺铂诱导的肾小管细胞氧化损伤

期刊:Cell Death & Disease

影响因子:8.1

doi:10.1038/s41419-018-0537-6

Min Jung Kong, Sang Jun Han, Jee In Kim, Jeen-Woo Park, Kwon Moo Park

Can Tissue Cilia Lengths and Urine Cilia Proteins Be Markers of Kidney Diseases?

组织纤毛长度和尿纤毛蛋白能否作为肾脏疾病的标志物？

期刊:

影响因子:

doi:10.4068/cmj.2018.54.2.83

Park, Kwon Moo

发表日期：2018

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Mitochondrial NADP+-Dependent Isocitrate Dehydrogenase Deficiency Exacerbates Mitochondrial and Cell Damage after Kidney Ischemia-Reperfusion Injury

线粒体 NADP+ 依赖性异柠檬酸脱氢酶缺乏加剧肾缺血再灌注损伤后的线粒体和细胞损伤

期刊:Journal of the American Society of Nephrology

影响因子:10.3

doi:10.1681/ASN.2016030349

Sang Jun Han, Hee-Seong Jang, Mi Ra Noh, Jinu Kim, Min Jung Kong, Jee In Kim, Jeen-Woo Park, Kwon Moo Park

Deficiency of exocyst complex component Exoc5 exacerbates the progression of kidney fibrosis.

外泌体复合物成分 Exoc5 的缺乏会加剧肾纤维化的进展。

Twin Glomeruli: a Newly Discovered Marker of Neonephrogenesis in the Ischemia-Reperfusion Injured Adult Mouse Kidney.

双肾小球：缺血再灌注损伤成年小鼠肾脏中新生肾发生的新发现标志。

MON-661 Melanocortin-4 Receptor is Rapidly Ubiquitinated, Exits Primary Cilia and Undergoes Endocytosis After Activation

MON-661 黑皮质素-4 受体激活后迅速泛素化，离开初级纤毛并发生内吞作用

Cilia-deficient renal tubule cells are primed for injury with mitochondrial defects and aberrant tryptophan metabolism.

纤毛缺陷的肾小管细胞由于线粒体缺陷和色氨酸代谢异常而容易受到损伤

Macrophages promote Fibrinogenesis during kidney injury

巨噬细胞在肾损伤期间促进纤维蛋白生成

Electrophysiological Properties of Ion Channels in Ascaris suum Tissue Incorporated into Planar Lipid Bilayers

猪蛔虫组织中离子通道整合到平面脂双层中的电生理特性

Fragmentation of kidney epithelial cell primary cilia occurs by cisplatin and these cilia fragments are excreted into the urine

顺铂会使肾脏上皮细胞原发纤毛碎裂，这些纤毛碎片会排泄到尿液中

Mitochondrial NADP+-dependent isocitrate dehydrogenase deficiency increases cisplatin-induced oxidative damage in the kidney tubule cells

线粒体 NADP+ 依赖性异柠檬酸脱氢酶缺乏会增加顺铂诱导的肾小管细胞氧化损伤

Can Tissue Cilia Lengths and Urine Cilia Proteins Be Markers of Kidney Diseases?

组织纤毛长度和尿纤毛蛋白能否作为肾脏疾病的标志物？

Mitochondrial NADP+-Dependent Isocitrate Dehydrogenase Deficiency Exacerbates Mitochondrial and Cell Damage after Kidney Ischemia-Reperfusion Injury

线粒体 NADP+ 依赖性异柠檬酸脱氢酶缺乏加剧肾缺血再灌注损伤后的线粒体和细胞损伤