日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

IL-17A-mediated neutrophil recruitment limits expansion of segmented filamentous bacteria

IL-17A介导的中性粒细胞募集限制了节段丝状细菌的增殖。

Flannigan, K L; Ngo, V L; Geem, D; Harusato, A; Hirota, S A; Parkos, C A; Lukacs, N W; Nusrat, A; Gaboriau-Routhiau, V; Cerf-Bensussan, N; Gewirtz, A T; Denning, T L

Neutrophil interactions with epithelial-expressed ICAM-1 enhances intestinal mucosal wound healing

中性粒细胞与上皮细胞表达的ICAM-1相互作用可促进肠黏膜伤口愈合。

Sumagin, R; Brazil, J C; Nava, P; Nishio, H; Alam, A; Luissint, A C; Weber, D A; Neish, A S; Nusrat, A; Parkos, C A

Norovirus in symptomatic and asymptomatic individuals: cytokines and viral shedding

诺如病毒在有症状和无症状个体中的细胞因子和病毒脱落

Newman, K L; Moe, C L; Kirby, A E; Flanders, W D; Parkos, C A; Leon, J S

Human norovirus infection and the acute serum cytokine response

人类诺如病毒感染与急性血清细胞因子反应

Newman, K L; Moe, C L; Kirby, A E; Flanders, W D; Parkos, C A; Leon, J S

Neutrophil-derived JAML inhibits repair of intestinal epithelial injury during acute inflammation.

中性粒细胞衍生的 JAML 抑制急性炎症期间肠道上皮损伤的修复

Weber D A, Sumagin R, McCall I C, Leoni G, Neumann P A, Andargachew R, Brazil J C, Medina-Contreras O, Denning T L, Nusrat A, Parkos C A

Protein kinase CK2 is a critical regulator of epithelial homeostasis in chronic intestinal inflammation.

蛋白激酶 CK2 是慢性肠道炎症中上皮稳态的关键调节因子

Koch S, Capaldo C T, Hilgarth R S, Fournier B, Parkos C A, Nusrat A

Guanylate-binding protein-1 is expressed at tight junctions of intestinal epithelial cells in response to interferon-gamma and regulates barrier function through effects on apoptosis

鸟苷酸结合蛋白-1在干扰素-γ刺激下于肠上皮细胞紧密连接处表达,并通过影响细胞凋亡来调节肠道屏障功能。

Schnoor, M; Betanzos, A; Weber, D A; Parkos, C A

Clostridium difficile toxins disrupt epithelial barrier function by altering membrane microdomain localization of tight junction proteins

艰难梭菌毒素通过改变紧密连接蛋白的膜微区定位来破坏上皮屏障功能。

Nusrat, A; von Eichel-Streiber, C; Turner, J R; Verkade, P; Madara, J L; Parkos, C A

Unmasking of intestinal epithelial lateral membrane beta1 integrin consequent to transepithelial neutrophil migration in vitro facilitates inv-mediated invasion by Yersinia pseudotuberculosis

体外跨上皮迁移导致肠上皮侧膜β1整合素暴露,从而促进假结核耶尔森菌通过inv介导的侵袭。

McCormick, B A; Nusrat, A; Parkos, C A; D'Andrea, L; Hofman, P M; Carnes, D; Liang, T W; Madara, J L

Epithelial exposure to hypoxia modulates neutrophil transepithelial migration

上皮细胞暴露于缺氧环境会调节中性粒细胞的跨上皮迁移。

Colgan, S P; Dzus, A L; Parkos, C A