Peng Wan-Xin相关文献与解析，生知库 | 链接生命科学的每一步

Loss of YTHDF1 suppresses the progression of malignant rhabdoid tumor of the kidney by regulating Glutathione S-transferase Mu 2 (GSTM2).

YTHDF1 的缺失通过调节谷胱甘肽 S-转移酶 Mu 2 (GSTM2) 抑制肾脏恶性横纹肌样瘤的进展

Xiong Qian-Wen, Liu Yuntao, He Min, Shen Xiao-Die, Zhao Manli, Liu Shuang-Ai, Zhang Gensheng, Liu Qian, Wang Jinhu, Peng Wan-Xin

信号转导

发表日期：2025

文献求助收藏

Retraction Note: FoxM1-mediated RFC5 expression promotes temozolomide resistance

撤稿声明：FoxM1介导的RFC5表达促进替莫唑胺耐药性。

期刊:Cell Biology and Toxicology

影响因子:5.9

doi:10.1007/s10565-024-09887-0

Peng, Wan-Xin; Han, Xiu; Zhang, Chun-Li; Ge, Lu; Du, Feng-Yi; Jin, Jie; Gong, Ai-Hua

发表日期：2024

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Perioperative complication incidence and risk factors for retroperitoneal neuroblastoma in children: analysis of 571 patients

儿童腹膜后神经母细胞瘤围手术期并发症发生率及危险因素：571例患者分析

期刊:World Journal of Pediatrics

影响因子:4.5

doi:10.1007/s12519-023-00773-z

He, Min; Cai, Jia-Bin; Wu, Xuan; Tang, Yin-Bing; Wang, Jin-Yan; Mao, Jun-Qin; Chen, Ji-Jun; Zhang, Li-Feng; Guan, Zhong-Hai; Xiong, Jie-Ni; Peng, Wan-Xin; Wang, Jin-Hu; Tao, Ting

细胞生物学

发表日期：2024

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Corrigendum to "N6-methyladenosine modified LINC00901 promotes pancreatic cancer progression through IGF2BP2/MYC axis" [Genes & Diseases 10 (2023) 554-567]

对“N6-甲基腺苷修饰的LINC00901通过IGF2BP2/MYC轴促进胰腺癌进展”的更正[Genes & Diseases 10 (2023) 554-567]

期刊:Genes and Diseases

影响因子:

doi:10.1016/j.gendis.2024.101352

Peng, Wan-Xin; Liu, Fei; Jiang, Jia-Hong; Yuan, Hang; Zhang, Ziqiang; Yang, Liu; Mo, Yin-Yuan

RETRACTED: IncRNA RMST Enhances DNMT3 Expression through Interaction with HuR

撤稿：IncRNA RMST 通过与 HuR 相互作用增强 DNMT3 表达

期刊:Molecular Therapy

影响因子:12

doi:10.1016/j.ymthe.2023.01.020

Peng, Wan-Xin; Koirala, Pratirodh; Zhang, Wei; Ni, Chao; Wang, Zheng; Yang, Liu; Mo, Yin-Yuan

多发性硬化症

发表日期：2023

文献求助收藏

Roles of lncRNAs in childhood cancer: Current landscape and future perspectives

lncRNA在儿童癌症中的作用：现状与展望

期刊:Frontiers in Oncology

影响因子:3.3

doi:10.3389/fonc.2023.1060107

Liu, Fei; Xiong, Qian-Wen; Wang, Jin-Hu; Peng, Wan-Xin

Roxadustat: Do we know all the answers?

罗沙司他：我们是否掌握了所有答案？

期刊:Biomolecules Biomedicine

影响因子:2.2

doi:10.17305/bb.2022.8437

Li, Qiu-Yu; Xiong, Qian-Wen; Yao, Xuefeng; Liu, Fei; Tang, Xiaoxiao; Fu, Haidong; Tong, Tong; Mao, Jianhua; Peng, Wan-Xin

发表日期：2023

文献求助收藏

Connecting N6-methyladenosine modification to ferroptosis resistance in hepatoblastoma

将N6-甲基腺苷修饰与肝母细胞瘤的铁死亡抵抗联系起来

期刊:Clinical and Translational Medicine

影响因子:6.8

doi:10.1002/ctm2.820

Peng, Wan-Xin; Mo, Yin-Yuan

A link between a synonymous SNP and the clinical response to tyrosine kinase inhibitors

同义SNP与酪氨酸激酶抑制剂的临床反应之间的联系

期刊:

影响因子:

doi:10.21037/ncri.2018.01.03

Peng, Wan-Xin; Fan, Le-Tao; Mo, Yin-Yuan

信号转导

发表日期：2018

文献求助收藏

Correction: Lnc-DC promotes estrogen independent growth and tamoxifen resistance in breast cancer

更正：Lnc-DC促进乳腺癌的雌激素非依赖性生长和三苯氧胺耐药性

Loss of YTHDF1 suppresses the progression of malignant rhabdoid tumor of the kidney by regulating Glutathione S-transferase Mu 2 (GSTM2).

YTHDF1 的缺失通过调节谷胱甘肽 S-转移酶 Mu 2 (GSTM2) 抑制肾脏恶性横纹肌样瘤的进展

Retraction Note: FoxM1-mediated RFC5 expression promotes temozolomide resistance

撤稿声明：FoxM1介导的RFC5表达促进替莫唑胺耐药性。

Perioperative complication incidence and risk factors for retroperitoneal neuroblastoma in children: analysis of 571 patients

儿童腹膜后神经母细胞瘤围手术期并发症发生率及危险因素：571例患者分析

Corrigendum to "N6-methyladenosine modified LINC00901 promotes pancreatic cancer progression through IGF2BP2/MYC axis" [Genes & Diseases 10 (2023) 554-567]

对“N6-甲基腺苷修饰的LINC00901通过IGF2BP2/MYC轴促进胰腺癌进展”的更正[Genes & Diseases 10 (2023) 554-567]

RETRACTED: IncRNA RMST Enhances DNMT3 Expression through Interaction with HuR

撤稿：IncRNA RMST 通过与 HuR 相互作用增强 DNMT3 表达

Roles of lncRNAs in childhood cancer: Current landscape and future perspectives

lncRNA在儿童癌症中的作用：现状与展望

Roxadustat: Do we know all the answers?

罗沙司他：我们是否掌握了所有答案？

Connecting N6-methyladenosine modification to ferroptosis resistance in hepatoblastoma

将N6-甲基腺苷修饰与肝母细胞瘤的铁死亡抵抗联系起来

A link between a synonymous SNP and the clinical response to tyrosine kinase inhibitors

同义SNP与酪氨酸激酶抑制剂的临床反应之间的联系