Perides相关文献与解析，生知库 | 链接生命科学的每一步

Pratibha Singh, Lady Leidy Sanchez-Fernandez, David Ramiro-Cortijo, Pedro Ochoa-Allemant, George Perides, Yan Liu, Esli Medina-Morales, William Yakah, Steven D Freedman, Camilia R Martin

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Mouse

发表日期：2020

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Early Enteral Administration of a Complex Lipid Emulsion Supplement Prevents Postnatal Deficits in Docosahexaenoic and Arachidonic Acids and Increases Tissue Accretion of Lipophilic Nutrients in Preterm Piglets

早期肠内注射复合脂质乳剂补充剂可预防早产仔猪出生后二十二碳六烯酸和花生四烯酸的缺乏，并增加亲脂性营养物质的组织沉积

期刊:Journal of Parenteral and Enteral Nutrition

影响因子:

doi:10.1002/jpen.1697

Olajumoke Akinsulire, George Perides, Lorenzo Anez-Bustillos, Joanne Cluette-Brown, Arthur Nedder, Elizabeth Pollack, Pratibha Singh, Yan Liu, Lady Leidy Sanchez-Fernandez, Evelyn Obregon, Ece Bicak, Savanna Kiefer, William Yakah, Hilda V Gutierrez, Duy T Dao, Mustafa Vurma, Stefan Ehling, Douglas G

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发表日期：2020

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Developmental Accretion of Docosahexaenoic Acid Is Independent of Fatty Acid Transporter Expression in Brain and Lung Tissues of C57BL/6 and Fat1 Mice

C57BL/6 和 Fat1 小鼠脑和肺组织中二十二碳六烯酸的发育积累与脂肪酸转运蛋白的表达无关

期刊:Journal of Nutrition

影响因子:3.8

doi:10.1093/jn/nxz074

Yakah, William; Singh, Pratibha; Perides, George; Brown, Joanne; Freedman, Steven D; Martin, Camilia R

Necroptosis Is an Important Severity Determinant and Potential Therapeutic Target in Experimental Severe Pancreatitis.

坏死性凋亡是实验性重症胰腺炎的重要严重程度决定因素和潜在治疗靶点

期刊:Cellular and Molecular Gastroenterology and Hepatology

影响因子:7.4

doi:10.1016/j.jcmgh.2016.04.002

Louhimo Johanna, Steer Michael L, Perides George

Pharmacological inhibition of PAR2 with the pepducin P2pal-18S protects mice against acute experimental biliary pancreatitis

使用肽类药物 P2pal-18S 对 PAR2 进行药理学抑制可保护小鼠免受急性实验性胆汁性胰腺炎的侵害。

期刊:American Journal of Physiology-Gastrointestinal and Liver Physiology

影响因子:3.3

doi:10.1152/ajpgi.00296.2012

Michael, E S; Kuliopulos, A; Covic, L; Steer, M L; Perides, G

Preparation of pancreatic acinar cells for the purpose of calcium imaging, cell injury measurements, and adenoviral infection

制备胰腺腺泡细胞用于钙成像、细胞损伤测量和腺病毒感染研究

期刊:Jove-Journal of Visualized Experiments

影响因子:1

doi:10.3791/50391

Orabi, Abrahim I; Muili, Kamaldeen A; Wang, Dong; Jin, Shunqian; Perides, George; Husain, Sohail Z

Biliary acute pancreatitis in mice is mediated by the G-protein-coupled cell surface bile acid receptor Gpbar1

小鼠的胆源性急性胰腺炎是由G蛋白偶联细胞表面胆汁酸受体Gpbar1介导的。

期刊:Gastroenterology

影响因子:25.1

doi:10.1053/j.gastro.2009.10.052

Perides, George; Laukkarinen, Johanna M; Vassileva, Galya; Steer, Michael L

Matrix metalloproteinase 9 plays a key role in lyme arthritis but not in dissemination of Borrelia burgdorferi

基质金属蛋白酶9在莱姆关节炎中起关键作用，但在伯氏疏螺旋体的传播中并不起关键作用。

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00214-09

Heilpern, Andrew J; Wertheim, Warren; He, Jia; Perides, George; Bronson, Roderick T; Hu, Linden T

关节炎

发表日期：2009

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Protease-activated receptor-2 exerts contrasting model-specific effects on acute experimental pancreatitis

蛋白酶激活受体-2对急性实验性胰腺炎表现出截然不同的模型特异性效应

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M801779200

Laukkarinen, Johanna M; Weiss, Eric R; van Acker, Gijs J D; Steer, Michael L; Perides, George

A mouse model of acute biliary pancreatitis induced by retrograde pancreatic duct infusion of Na-taurocholate

通过逆行胰管灌注牛磺胆酸钠诱导的急性胆源性胰腺炎小鼠模型

期刊:Gut

影响因子:25.8

doi:10.1136/gut.2007.124230

Laukkarinen, Johanna M; Van Acker, Gijs J D; Weiss, Eric R; Steer, Michael L; Perides, George

胰腺炎

发表日期：2007

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Maltodextrin-induced intestinal injury in a neonatal mouse model

麦芽糊精诱发新生小鼠肠道损伤模型

Early Enteral Administration of a Complex Lipid Emulsion Supplement Prevents Postnatal Deficits in Docosahexaenoic and Arachidonic Acids and Increases Tissue Accretion of Lipophilic Nutrients in Preterm Piglets

早期肠内注射复合脂质乳剂补充剂可预防早产仔猪出生后二十二碳六烯酸和花生四烯酸的缺乏，并增加亲脂性营养物质的组织沉积

Developmental Accretion of Docosahexaenoic Acid Is Independent of Fatty Acid Transporter Expression in Brain and Lung Tissues of C57BL/6 and Fat1 Mice

C57BL/6 和 Fat1 小鼠脑和肺组织中二十二碳六烯酸的发育积累与脂肪酸转运蛋白的表达无关

Necroptosis Is an Important Severity Determinant and Potential Therapeutic Target in Experimental Severe Pancreatitis.

坏死性凋亡是实验性重症胰腺炎的重要严重程度决定因素和潜在治疗靶点

Pharmacological inhibition of PAR2 with the pepducin P2pal-18S protects mice against acute experimental biliary pancreatitis

使用肽类药物 P2pal-18S 对 PAR2 进行药理学抑制可保护小鼠免受急性实验性胆汁性胰腺炎的侵害。

Preparation of pancreatic acinar cells for the purpose of calcium imaging, cell injury measurements, and adenoviral infection

制备胰腺腺泡细胞用于钙成像、细胞损伤测量和腺病毒感染研究

Biliary acute pancreatitis in mice is mediated by the G-protein-coupled cell surface bile acid receptor Gpbar1

小鼠的胆源性急性胰腺炎是由G蛋白偶联细胞表面胆汁酸受体Gpbar1介导的。

Matrix metalloproteinase 9 plays a key role in lyme arthritis but not in dissemination of Borrelia burgdorferi

基质金属蛋白酶9在莱姆关节炎中起关键作用，但在伯氏疏螺旋体的传播中并不起关键作用。

Protease-activated receptor-2 exerts contrasting model-specific effects on acute experimental pancreatitis

蛋白酶激活受体-2对急性实验性胰腺炎表现出截然不同的模型特异性效应

A mouse model of acute biliary pancreatitis induced by retrograde pancreatic duct infusion of Na-taurocholate

通过逆行胰管灌注牛磺胆酸钠诱导的急性胆源性胰腺炎小鼠模型