日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Targeting Regnase-1 unleashes CAR T cell antitumor activity for osteosarcoma and creates a proinflammatory tumor microenvironment

靶向Regnase-1可释放CAR T细胞对骨肉瘤的抗肿瘤活性,并创造促炎性肿瘤微环境。

Adeshakin, Adeleye O; Shi, Hao; Perry, S Scott; Sheppard, Heather; Nguyen, Phuong; Sun, Xiang; Zhou, Peipei; Métais, Jean-Yves; Cunningham, Trevor; Anil, K C; Tian, Liqing; Peche, Vivek; Prater, Mollie S; Langfitt, Deanna M; Pruett-Miller, Shondra M; Yustein, Jason T; Krenciute, Giedre; DeRenzo, Christopher; Chi, Hongbo; Gottschalk, Stephen

Apoptosis-Inducing-Factor-Dependent Mitochondrial Function Is Required for T Cell but Not B Cell Function

凋亡诱导因子依赖的线粒体功能是T细胞功能所必需的,但并非B细胞功能所必需的。

Milasta, Sandra; Dillon, Christopher P; Sturm, Oliver E; Verbist, Katherine C; Brewer, Taylor L; Quarato, Giovanni; Brown, Scott A; Frase, Sharon; Janke, Laura J; Perry, S Scott; Thomas, Paul G; Green, Douglas R

Id1 attenuates Notch signaling and impairs T-cell commitment by elevating Deltex1 expression

Id1通过提高Deltex1的表达来减弱Notch信号传导并损害T细胞分化。

Wang, Hong-Cheng; Perry, S Scott; Sun, Xiao-Hong

Regulation of lymphocyte development by cell-type-specific interpretation of Notch signals

通过细胞类型特异性解读Notch信号来调控淋巴细胞发育

Nie, Lei; Perry, S Scott; Zhao, Ying; Huang, Jiaxue; Kincade, Paul W; Farrar, Michael A; Sun, Xiao-Hong

Interferon-producing killer dendritic cells (IKDCs) arise via a unique differentiation pathway from primitive c-kitHiCD62L+ lymphoid progenitors.

产生干扰素的杀伤性树突状细胞(IKDC)是通过原始 c-kitHiCD62L+ 淋巴祖细胞独特的分化途径产生的

Welner Robert S, Pelayo Rosana, Garrett Karla P, Chen Xinrong, Perry S Scott, Sun Xiao-Hong, Kee Barbara L, Kincade Paul W