Peter Teismann相关文献与解析，生知库 | 链接生命科学的每一步

Herrera-Rivero, Marisol; Garvert, Linda; Horn, Katrin; Löbner, Margrit; Weitzel, Elena Caroline; Stoll, Monika; Lichtner, Peter; Teismann, Henning; Teumer, Alexander; Van der Auwera, Sandra; Völzke, Henry; Völker, Uwe; Andlauer, Till F M; Meinert, Susanne; Heilmann-Heimbach, Stefanie; Forstner, Andreas J; Streit, Fabian; Witt, Stephanie H; Kircher, Tilo; Dannlowski, Udo; Scholz, Markus; Riedel-Heller, Steffi G; Grabe, Hans J; Baune, Bernhard T; Berger, Klaus

发表日期：2025

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Neurochemical, histological, and behavioral profiling of the acute, sub-acute, and chronic MPTP mouse model of Parkinson's disease

帕金森病急性、亚急性和慢性 MPTP 小鼠模型的神经化学、组织学和行为分析

期刊:Journal of Neurochemistry

影响因子:4.2

doi:10.1111/jnc.15699

Matteo Santoro, Paola Fadda, Katie J Klephan, Claire Hull, Peter Teismann, Bettina Platt, Gernot Riedel

Evidence for a role of adaptive immune response in the disease pathogenesis of the MPTP mouse model of Parkinson's disease

有证据表明适应性免疫反应在帕金森病 MPTP 小鼠模型的发病机制中发挥作用

期刊:Glia

影响因子:5.4

doi:10.1002/glia.22935

Heather L Martin, Matteo Santoro, Sarah Mustafa, Gernot Riedel, John V Forrester, Peter Teismann

In-vivo evidence that high mobility group box 1 exerts deleterious effects in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model and Parkinson's disease which can be attenuated by glycyrrhizin

体内证据表明，高迁移率族蛋白 B1 在 1-甲基-4-苯基-1,2,3,6-四氢吡啶模型和帕金森病中发挥有害作用，而甘草甜素可以减轻这种作用

期刊:Neurobiology of Disease

影响因子:5.1

doi:10.1016/j.nbd.2016.02.018

Matteo Santoro, Walter Maetzler, Petros Stathakos, Heather L Martin, Markus A Hobert, Tim W Rattay, Thomas Gasser, John V Forrester, Daniela Berg, Kevin J Tracey, Gernot Riedel, Peter Teismann

Increasing levels of the endocannabinoid 2-AG is neuroprotective in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine mouse model of Parkinson's disease

内源性大麻素 2-AG 水平的提高对患有帕金森病的 1-甲基-4-苯基-1,2,3,6-四氢吡啶小鼠模型具有神经保护作用

期刊:Experimental Neurology

影响因子:4.6

doi:10.1016/j.expneurol.2015.07.024

Ross B Mounsey, Sarah Mustafa, Lianne Robinson, Ruth A Ross, Gernot Riedel, Roger G Pertwee, Peter Teismann

S100B Up-Regulates Macrophage Production of IL1β and CCL22 and Influences Severity of Retinal Inflammation

S100B 上调巨噬细胞产生 IL1β 和 CCL22 并影响视网膜炎症的严重程度

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0132688

Jennifer Niven, Joseph Hoare, Debbie McGowan, Gayathri Devarajan, Shigeyoshi Itohara, Monique Gannagé, Peter Teismann, Isabel Crane

S100B is increased in Parkinson's disease and ablation protects against MPTP-induced toxicity through the RAGE and TNF-α pathway

帕金森病中 S100B 增多，消融可通过 RAGE 和 TNF-α 通路预防 MPTP 诱导的毒性

期刊:Brain

影响因子:10.6

doi:10.1093/brain/aws250

Kinnari Sathe, Walter Maetzler, Johannes D Lang, Ross B Mounsey, Corina Fleckenstein, Heather L Martin, Claudia Schulte, Sarah Mustafa, Matthis Synofzik, Zvonimir Vukovic, Shigeyoshi Itohara, Daniela Berg, Peter Teismann

Automated behavioral phenotyping reveals presymptomatic alterations in a SCA3 genetrap mouse model

自动行为表型分析揭示了 SCA3 基因诱捕小鼠模型中的症状前改变

期刊:Journal of Genetics and Genomics

影响因子:6.6

doi:10.1016/j.jgg.2012.04.009

Jeannette Hübener, Nicolas Casadei, Peter Teismann, Mathias W Seeliger, Maria Björkqvist, Stephan von Hörsten, Olaf Riess, Huu Phuc Nguyen

发表日期：2012

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Pharmacological manipulation of peroxisome proliferator-activated receptor γ (PPARγ) reveals a role for anti-oxidant protection in a model of Parkinson's disease

过氧化物酶体增殖激活受体 γ (PPARγ) 的药理学操作揭示了抗氧化保护在帕金森病模型中的作用

期刊:Experimental Neurology

影响因子:4.6

doi:10.1016/j.expneurol.2012.02.017

Heather L Martin, Ross B Mounsey, Sarah Mustafa, Kinnari Sathe, Peter Teismann

MPP(+)-induced toxicity in the presence of dopamine is mediated by COX-2 through oxidative stress

多巴胺存在下 MPP(+) 诱导的毒性由 COX-2 通过氧化应激介导

期刊:Naunyn-Schmiedebergs Archives of Pharmacology

影响因子:3.1

doi:10.1007/s00210-011-0660-8

Ya-Ching Hsieh, Ross B Mounsey, Peter Teismann

表观遗传

发表日期：2011

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A meta-analysis of genome-wide studies of resilience in the German population

一项针对德国人群韧性的全基因组研究的荟萃分析

Neurochemical, histological, and behavioral profiling of the acute, sub-acute, and chronic MPTP mouse model of Parkinson's disease

帕金森病急性、亚急性和慢性 MPTP 小鼠模型的神经化学、组织学和行为分析

Evidence for a role of adaptive immune response in the disease pathogenesis of the MPTP mouse model of Parkinson's disease

有证据表明适应性免疫反应在帕金森病 MPTP 小鼠模型的发病机制中发挥作用

In-vivo evidence that high mobility group box 1 exerts deleterious effects in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model and Parkinson's disease which can be attenuated by glycyrrhizin

体内证据表明，高迁移率族蛋白 B1 在 1-甲基-4-苯基-1,2,3,6-四氢吡啶模型和帕金森病中发挥有害作用，而甘草甜素可以减轻这种作用

Increasing levels of the endocannabinoid 2-AG is neuroprotective in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine mouse model of Parkinson's disease

内源性大麻素 2-AG 水平的提高对患有帕金森病的 1-甲基-4-苯基-1,2,3,6-四氢吡啶小鼠模型具有神经保护作用

S100B Up-Regulates Macrophage Production of IL1β and CCL22 and Influences Severity of Retinal Inflammation

S100B 上调巨噬细胞产生 IL1β 和 CCL22 并影响视网膜炎症的严重程度

S100B is increased in Parkinson's disease and ablation protects against MPTP-induced toxicity through the RAGE and TNF-α pathway

帕金森病中 S100B 增多，消融可通过 RAGE 和 TNF-α 通路预防 MPTP 诱导的毒性

Automated behavioral phenotyping reveals presymptomatic alterations in a SCA3 genetrap mouse model

自动行为表型分析揭示了 SCA3 基因诱捕小鼠模型中的症状前改变

Pharmacological manipulation of peroxisome proliferator-activated receptor γ (PPARγ) reveals a role for anti-oxidant protection in a model of Parkinson's disease

过氧化物酶体增殖激活受体 γ (PPARγ) 的药理学操作揭示了抗氧化保护在帕金森病模型中的作用

MPP(+)-induced toxicity in the presence of dopamine is mediated by COX-2 through oxidative stress

多巴胺存在下 MPP(+) 诱导的毒性由 COX-2 通过氧化应激介导