日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Comparison of receptor expression and cholecystokinin signaling between left and right nodose ganglia

比较左右结状神经节受体表达和胆囊收缩素信号传导

Ritchey, Caitlin R; McCune, Katherine X; Peters, James H

In autonomic adaptation the details matter

在自主神经适应过程中,细节至关重要。

Ritchey, Caitlin R; Peters, James H

Functionally distinct evoked and spontaneous neurotransmission operate via a shared pool of synaptic vesicles in viscerosensory afferents.

功能不同的诱发性和自发性神经传递通过内脏感觉传入神经中共享的突触小泡池发挥作用

Arnold Rachel A, Peters James H

Characterization of NTS-to-VTA projection neurons reveals higher-order synaptic organization and distinct responsiveness to cholecystokinin.

对 NTS 到 VTA 投射神经元的特征分析揭示了更高阶的突触组织和对胆囊收缩素的独特反应

Ritchey Caitlin R, Rossi David J, Peters James H

TRPV1 enhances cholecystokinin signaling in primary vagal afferent neurons and mediates the central effects on spontaneous glutamate release in the NTS.

TRPV1 增强初级迷走神经传入神经元中的胆囊收缩素信号传导,并介导对孤束核中自发性谷氨酸释放的中枢效应

Arnold Rachel A, Fowler Daniel K, Peters James H

Principles of synaptic encoding of brainstem circadian rhythms

脑干昼夜节律突触编码原理

Ragozzino, Forrest J; Karatsoreos, Ilia N; Peters, James H

Circadian regulation of glutamate release pathways shapes synaptic throughput in the brainstem nucleus of the solitary tract (NTS)

昼夜节律调节谷氨酸释放通路,从而影响脑干孤束核(NTS)的突触通量。

Ragozzino, Forrest J; Peterson, Bree Anne; Karatsoreos, Ilia N; Peters, James H

Corticosterone inhibits vagal afferent glutamate release in the nucleus of the solitary tract via retrograde endocannabinoid signaling

皮质酮通过逆行内源性大麻素信号传导抑制孤束核中迷走神经传入的谷氨酸释放。

Ragozzino, Forrest J; Arnold, Rachel A; Kowalski, Cody W; Savenkova, Marina I; Karatsoreos, Ilia N; Peters, James H

Redundant Postsynaptic Functions of SynCAMs 1-3 during Synapse Formation

SynCAM 1-3 在突触形成过程中的冗余突触后功能

Fowler, Daniel K; Peters, James H; Williams, Carly; Washbourne, Philip

Energy-dense diet triggers changes in gut microbiota, reorganization of gut‑brain vagal communication and increases body fat accumulation

高能量饮食会引发肠道菌群改变、肠-脑迷走神经通讯重组,并增加体内脂肪堆积。

Vaughn, Alexandra C; Cooper, Erin M; DiLorenzo, Patricia M; O'Loughlin, Levi J; Konkel, Michael E; Peters, James H; Hajnal, Andras; Sen, Tanusree; Lee, Sun Hye; de La Serre, Claire B; Czaja, Krzysztof