Peters-Golden Marc相关文献与解析，生知库

Longitudinal Single-Cell RNA-seq Profiling of Lung Cell Phenotypes, Signaling, and Cross-talk During Fibrosis Resolution

肺纤维化消退过程中肺细胞表型、信号传导和相互作用的纵向单细胞RNA测序分析

doi:10.64898/2026.04.06.716772

Speth, Jennifer; Wong, Vivian T; Guzman, Steve D; Liu, Yang; Walker, Natalie M; Zemans, Rachel L; Blackwell, Timothy S; Aguilar, Carlos A; Peters-Golden, Marc; Fortier, Sean M

Glycolytic reprogramming of resident alveolar macrophages contributes to reduced SOCS3 secretion in non-small cell lung cancer

肺泡驻留巨噬细胞的糖酵解重编程导致非小细胞肺癌中SOCS3分泌减少

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2025.1708467

Speth, Jennifer M; Haggadone, Mikel D; Peters-Golden, Marc

Distinct cAMP Regulation in Scleroderma Lung and Skin Myofibroblasts Governs Their Dedifferentiation via p38Î± Inhibition.

硬皮病肺和皮肤肌成纤维细胞中独特的 cAMP 调节通过 p38α 抑制控制其去分化

期刊:FASEB Journal

影响因子:4.2

doi:10.1096/fj.202500694RR

Baas Jared D, Varga John, Feghali-Bostwick Carol, Peters-Golden Marc, Fortier Sean M

细胞生物学

发表日期：2025

文献求助收藏

Reimagining Fibrosis Research, Outcomes, and Therapeutics Through the Lens of Resolution

从分辨率视角重新构想纤维化研究、结果和治疗方法

期刊:Seminars in Respiratory and Critical Care Medicine

影响因子:2.7

doi:10.1055/a-2666-7479

Fortier, Sean M; Redente, Elizabeth F; Peters-Golden, Marc

发表日期：2025

文献求助收藏

Distinct cAMP regulation in scleroderma lung and skin myofibroblasts governs their dedifferentiation via p38Î± inhibition.

硬皮病肺和皮肤肌成纤维细胞中独特的 cAMP 调节通过 p38α 抑制来控制其去分化

期刊:

影响因子:

doi:10.1101/2025.02.26.640163

Baas Jared D, Varga John, Feghali-Bostwick Carol, Peters-Golden Marc, Fortier Sean M

细胞生物学

发表日期：2025

文献求助收藏

Leukotrienes in Innate Immunity: Still Underappreciated after All These Years?

白三烯在先天免疫中的作用：这么多年过去了，它仍然没有得到应有的重视吗？

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.2200599

Serezani, C Henrique; Divangahi, Maziar; Peters-Golden, Marc

免疫/内分泌

发表日期：2023

文献求助收藏

Antifibrotic factor KLF4 is repressed by the miR-10/TFAP2A/TBX5 axis in dermal fibroblasts: insights from twins discordant for systemic sclerosis

真皮成纤维细胞中抗纤维化因子KLF4受miR-10/TFAP2A/TBX5轴抑制：来自系统性硬化症不一致双胞胎的启示

期刊:Annals of the Rheumatic Diseases

影响因子:20.6

doi:10.1136/annrheumdis-2021-221050

Malaab, Maya; Renaud, Ludivine; Takamura, Naoko; Zimmerman, Kip D; da Silveira, Willian A; Ramos, Paula S; Haddad, Sandra; Peters-Golden, Marc; Penke, Loka R; Wolf, Bethany; Hardiman, Gary; Langefeld, Carl D; Medsger, Thomas A; Feghali-Bostwick, Carol A

From Biomarker to Mechanism? F2-isoprostanes in Pulmonary Fibrosis

从生物标志物到机制？F2-异前列烷在肺纤维化中的作用

期刊:American Journal of Respiratory and Critical Care Medicine

影响因子:19.4

doi:10.1164/rccm.202205-0914ED

Thatcher, Thomas H; Peters-Golden, Marc

发表日期：2022

文献求助收藏

Illuminating the lung regenerative potential of prostanoids

揭示前列腺素的肺再生潜力

期刊:Science Advances

影响因子:12.5

doi:10.1126/sciadv.abp8322

Fortier, Sean M; Penke, Loka R; Peters-Golden, Marc

发表日期：2022

文献求助收藏

Mechanistic basis for the antifibrotic actions of cAMP-based therapies

cAMP疗法抗纤维化作用的机制基础

Longitudinal Single-Cell RNA-seq Profiling of Lung Cell Phenotypes, Signaling, and Cross-talk During Fibrosis Resolution

肺纤维化消退过程中肺细胞表型、信号传导和相互作用的纵向单细胞RNA测序分析

Glycolytic reprogramming of resident alveolar macrophages contributes to reduced SOCS3 secretion in non-small cell lung cancer

肺泡驻留巨噬细胞的糖酵解重编程导致非小细胞肺癌中SOCS3分泌减少

Distinct cAMP Regulation in Scleroderma Lung and Skin Myofibroblasts Governs Their Dedifferentiation via p38Î± Inhibition.

硬皮病肺和皮肤肌成纤维细胞中独特的 cAMP 调节通过 p38α 抑制控制其去分化

Reimagining Fibrosis Research, Outcomes, and Therapeutics Through the Lens of Resolution

从分辨率视角重新构想纤维化研究、结果和治疗方法

Distinct cAMP regulation in scleroderma lung and skin myofibroblasts governs their dedifferentiation via p38Î± inhibition.

硬皮病肺和皮肤肌成纤维细胞中独特的 cAMP 调节通过 p38α 抑制来控制其去分化

Leukotrienes in Innate Immunity: Still Underappreciated after All These Years?

白三烯在先天免疫中的作用：这么多年过去了，它仍然没有得到应有的重视吗？

Antifibrotic factor KLF4 is repressed by the miR-10/TFAP2A/TBX5 axis in dermal fibroblasts: insights from twins discordant for systemic sclerosis

真皮成纤维细胞中抗纤维化因子KLF4受miR-10/TFAP2A/TBX5轴抑制：来自系统性硬化症不一致双胞胎的启示

From Biomarker to Mechanism? F2-isoprostanes in Pulmonary Fibrosis

从生物标志物到机制？F2-异前列烷在肺纤维化中的作用

Illuminating the lung regenerative potential of prostanoids

揭示前列腺素的肺再生潜力