Thiol oxidative stress induced by metabolic disorders amplifies macrophage chemotactic responses and accelerates atherogenesis and kidney injury in LDL receptor-deficient mice
代谢紊乱诱导的硫醇氧化应激会增强巨噬细胞趋化反应,并加速低密度脂蛋白受体缺陷小鼠的动脉粥样硬化和肾损伤。
期刊:Arteriosclerosis Thrombosis and Vascular Biology
影响因子:7.4
doi:10.1161/ATVBAHA.109.191759
Qiao, Mu; Zhao, Qingwei; Lee, Chi Fung; Tannock, Lisa R; Smart, Eric J; LeBaron, Richard G; Phelix, Clyde F; Rangel, Yolanda; Asmis, Reto
巨噬细胞
氧化应激
巨噬细胞
信号转导
心血管
代谢
肾损伤
细胞生物学
毒理研究
动脉粥样硬化
免疫/内分泌