日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Autophagy modulates glioblastoma cell sensitivity to Selinexor-mediated XPO1 inhibition.

自噬调节胶质母细胞瘤细胞对塞利尼索介导的 XPO1 抑制的敏感性

Tang Yongjian, Sprinzen Lisa, Terada Yukinori, Kiang Karrie M, Li Chuntao, Zeng Yu, Liu Fangkun, Zhou Hongshu, Liang Xisong, Zhang Jianzhong, Pieper Russell O, Chen Bo, Zhang Liyang

Metabolic imaging detects elevated glucose flux through the pentose phosphate pathway associated with TERT expression in low-grade gliomas

代谢成像检测到低级别胶质瘤中与TERT表达相关的磷酸戊糖途径葡萄糖通量升高。

Viswanath, Pavithra; Batsios, Georgios; Ayyappan, Vinay; Taglang, Céline; Gillespie, Anne Marie; Larson, Peder E Z; Luchman, H Artee; Costello, Joseph F; Pieper, Russell O; Ronen, Sabrina M

MRE11 and UBR5 Co-Operate to Suppress RNF168-Mediated Fusion of Dysfunctional Telomeres.

MRE11 和 UBR5 协同抑制 RNF168 介导的功能异常端粒融合

Tang Yongjian, Mukherjee Joydeep, Pieper Russell O

MR-detectable metabolic biomarkers of response to mutant IDH inhibition in low-grade glioma

低级别胶质瘤中突变型IDH抑制剂治疗反应的磁共振可检测代谢生物标志物

Molloy, Abigail R; Najac, Chloé; Viswanath, Pavithra; Lakhani, Aliya; Subramani, Elavarasan; Batsios, Georgios; Radoul, Marina; Gillespie, Anne Marie; Pieper, Russell O; Ronen, Sabrina M

HDAC inhibition in glioblastoma monitored by hyperpolarized (13) C MRSI

利用超极化(13)C MRSI监测胶质母细胞瘤中的HDAC抑制

Radoul, Marina; Najac, Chloé; Viswanath, Pavithra; Mukherjee, Joydeep; Kelly, Mark; Gillespie, Anne Marie; Chaumeil, Myriam M; Eriksson, Pia; Delos Santos, Romelyn; Pieper, Russell O; Ronen, Sabrina M

Mutant IDH1 Cooperates with ATRX Loss to Drive the Alternative Lengthening of Telomere Phenotype in Glioma

突变型IDH1与ATRX缺失协同作用,驱动胶质瘤中端粒的替代性延长表型

Mukherjee, Joydeep; Johannessen, Tor-Christian; Ohba, Shigeo; Chow, Tracy T; Jones, Lindsey; Pandita, Ajay; Pieper, Russell O

Mutant IDH1-driven cellular transformation increases RAD51-mediated homologous recombination and temozolomide resistance.

突变型 IDH1 驱动的细胞转化增强了 RAD51 介导的同源重组和替莫唑胺耐药性

Ohba Shigeo, Mukherjee Joydeep, See Wendy L, Pieper Russell O

MUTANT IDH1-DRIVEN CELLULAR TRANSFORMATION INCREASES RAD51-MEDIATED HOMOLOGOUS RECOMBINATION AND TEMOZOLOMIDE (TMZ) RESISTANCE

突变型IDH1驱动的细胞转化增强了RAD51介导的同源重组和替莫唑胺(TMZ)耐药性

Depaoli, Maria R; Hay, Jesse C; Graier, Wolfgang F; Malli, Roland; Shostrand, Lisa M; Dever, Emily; Baniya, A; Wang, Q; Lee, W j; Artemieva, M S; Suleymanov, R A; Danilin, I E; Gutierrez Alvarez, E; Barajas, A; Ayesa, R; Vaivre-Douret, L; Lopez, C; Kazda, T; Jancalek, R; Belanova, R; Pospisil, P; Burkon, P; Hynkova, L; Slampa, P; Lakomy, R; Mateo, Roselyn Cristelle; Ortiz, Ricardo; Rosen, Harold; Molenaar, RJ; Dimitrakopoulou, D; Stap, J; Bleeker, FE; Van Noorden, CJF; Pieper, Russell O; Ohba, Shigeo; Mukherjee, Joydeep

NPAS3 demonstrates features of a tumor suppressive role in driving the progression of Astrocytomas

NPAS3 在驱动星形细胞瘤进展中表现出抑癌作用的特征。

Moreira, Frederico; Kiehl, Tim-Rasmus; So, Kelvin; Ajeawung, Norbert F; Honculada, Carmelita; Gould, Peter; Pieper, Russell O; Kamnasaran, Deepak

Gamma interferon-mediated superinduction of B7-H1 in PTEN-deficient glioblastoma: a paradoxical mechanism of immune evasion

γ干扰素介导的PTEN缺陷型胶质母细胞瘤中B7-H1的超诱导:一种矛盾的免疫逃逸机制

Han, Seunggu J; Ahn, Brian J; Waldron, James S; Yang, Isaac; Fang, Shanna; Crane, Courtney A; Pieper, Russell O; Parsa, Andrew T