日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

LncRNA-MEG3 Mediated Diabetic Cerebral Ischemia-Reperfusion Injury-Induced Apoptosis via Modulating Interaction Between Annexin A2 and Akt in Mitochondria

LncRNA-MEG3 通过调节线粒体中 Annexin A2 与 Akt 之间的相互作用介导糖尿病脑缺血再灌注损伤诱导的细胞凋亡

Wanqing Zhou, Chongyi Tan, Di Xiong, Cheng Chen, Yanfei Zhao, Yongqiu Xie, Bei Sun, Zhihua Wang, Pingping Xia, Zhi Ye

O-sialoglycoprotein Endopeptidase (OSGEP) Suppresses Hepatic Ischemia-Reperfusion Injury-Induced Ferroptosis Through Modulating the MEK/ERK Signaling Pathway

O-唾液酸糖蛋白内肽酶(OSGEP)通过调节MEK/ERK信号通路抑制肝脏缺血再灌注损伤引起的铁死亡

Yuanyuan Tao #, Wanqing Zhou #, Cheng Chen, Qian Zhang, Zhuoyi Liu, Pingping Xia, Zhi Ye, Chunling Li

Long noncoding RNA Meg3 mediates ferroptosis induced by oxygen and glucose deprivation combined with hyperglycemia in rat brain microvascular endothelial cells, through modulating the p53/GPX4 axis

长链非编码RNA Meg3通过调节p53/GPX4轴介导大鼠脑微血管内皮细胞因氧和糖缺乏结合高血糖引起的铁死亡

Cheng Chen, Yan Huang, Pingping Xia, Fan Zhang, Longyan Li, E Wang, Qulian Guo, Zhi Ye

ALDH 2 conferred neuroprotection on cerebral ischemic injury by alleviating mitochondria-related apoptosis through JNK/caspase-3 signing pathway

ALDH 2 通过 JNK/caspase-3 信号通路减轻线粒体相关细胞凋亡,对脑缺血损伤起到神经保护作用

Pingping Xia, Fan Zhang, Yajing Yuan, Cheng Chen, Yan Huang, Longyan Li, E Wang, Qulian Guo, Zhi Ye

Aberrantly hydroxymethylated differentially expressed genes and the associated protein pathways in osteoarthritis

骨关节炎中异常羟甲基化差异表达基因及其相关蛋白通路

Fang, Yang; Wang, Pingping; Xia, Lin; Bai, Suwen; Shen, Yonggang; Li, Qing; Wang, Yang; Zhu, Jinhang; Du, Juan; Shen, Bing

A Reduced Transcriptome Approach to Assess Environmental Toxicants Using Zebrafish Embryo Test

利用斑马鱼胚胎试验,通过简化转录组分析方法评估环境毒物

Wang, Pingping; Xia, Pu; Yang, Jianghua; Wang, Zhihao; Peng, Ying; Shi, Wei; Villeneuve, Daniel L; Yu, Hongxia; Zhang, Xiaowei

Pioglitazone Confers Neuroprotection Against Ischemia-Induced Pyroptosis due to its Inhibitory Effects on HMGB-1/RAGE and Rac1/ROS Pathway by Activating PPAR-ɤ

吡格列酮通过激活 PPAR-ɤ 对 HMGB-1/RAGE 和 Rac1/ROS 通路产生抑制作用,从而发挥神经保护作用,防止缺血引起的细胞焦亡

Pingping Xia, Yundan Pan, Fan Zhang, Na Wang, E Wang, Qulian Guo, Zhi Ye