Popugaeva Elena相关文献与解析，生知库 | 链接生命科学的每一步

Potential Drug Candidates to Treat TRPC6 Channel Deficiencies in the Pathophysiology of Alzheimer's Disease and Brain Ischemia

治疗阿尔茨海默病和脑缺血病理生理学中TRPC6通道缺陷的潜在候选药物

期刊:Cells

影响因子:5.2

doi:10.3390/cells9112351

Prikhodko, Veronika; Chernyuk, Daria; Sysoev, Yurii; Zernov, Nikita; Okovityi, Sergey; Popugaeva, Elena

Reversal of Calcium Dysregulation as Potential Approach for Treating Alzheimer's Disease

逆转钙稳态失衡是治疗阿尔茨海默病的一种潜在方法

期刊:Current Alzheimer Research

影响因子:

doi:10.2174/1567205017666200528162046

Popugaeva, Elena; Chernyuk, Daria; Bezprozvanny, Ilya

阿尔茨海默症

发表日期：2020

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Dysregulation of Intracellular Calcium Signaling in Alzheimer's Disease

阿尔茨海默病中细胞内钙信号传导失调

期刊:Antioxidants & Redox Signaling

影响因子:6.1

doi:10.1089/ars.2018.7506

Popugaeva, Elena; Pchitskaya, Ekaterina; Bezprozvanny, Ilya

Calcium signaling and molecular mechanisms underlying neurodegenerative diseases

钙信号传导和神经退行性疾病的分子机制

期刊:Cell Calcium

影响因子:4

doi:10.1016/j.ceca.2017.06.008

Pchitskaya, Ekaterina; Popugaeva, Elena; Bezprozvanny, Ilya

Disturbed calcium signaling in spinocerebellar ataxias and Alzheimer's disease

脊髓小脑性共济失调和阿尔茨海默病中的钙信号传导紊乱

期刊:Seminars in Cell & Developmental Biology

影响因子:6

doi:10.1016/j.semcdb.2015.03.010

Egorova, Polina; Popugaeva, Elena; Bezprozvanny, Ilya

Calcium signaling, excitability, and synaptic plasticity defects in a mouse model of Alzheimer's disease

阿尔茨海默病小鼠模型中钙信号传导、兴奋性和突触可塑性缺陷

期刊:Journal of Alzheimers Disease

影响因子:3.1

doi:10.3233/JAD-142427

Zhang, Hua; Liu, Jie; Sun, Suya; Pchitskaya, Ekaterina; Popugaeva, Elena; Bezprozvanny, Ilya

Reduced synaptic STIM2 expression and impaired store-operated calcium entry cause destabilization of mature spines in mutant presenilin mice

突变型早老素小鼠突触STIM2表达降低和胞内钙库操纵性钙内流受损导致成熟树突棘不稳定。

期刊:Neuron

影响因子:15

doi:10.1016/j.neuron.2014.02.019

Sun, Suya; Zhang, Hua; Liu, Jie; Popugaeva, Elena; Xu, Nan-Jie; Feske, Stefan; White, Charles L 3rd; Bezprozvanny, Ilya

Commentary on "Synaptic function is modulated by LRRK2 and glutamate release is increased in cortical neurons of G2019S LRRK2 knock-in mice"

对“LRRK2 调节突触功能，G2019S LRRK2 敲入小鼠皮层神经元中谷氨酸释放增加”一文的评论

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2014.00351

Popugaeva, Elena; Bezprozvanny, Ilya

N-N-Substituted Piperazine, Cmp2, Improves Cognitive and Motor Functions in 5xFAD Mice.

NN取代哌嗪Cmp2可改善5xFAD小鼠的认知和运动功能

Role of Neuronal TRPC6 Channels in Synapse Development, Memory Formation and Animal Behavior

神经元TRPC6通道在突触发育、记忆形成和动物行为中的作用

Potential Drug Candidates to Treat TRPC6 Channel Deficiencies in the Pathophysiology of Alzheimer's Disease and Brain Ischemia

治疗阿尔茨海默病和脑缺血病理生理学中TRPC6通道缺陷的潜在候选药物

Reversal of Calcium Dysregulation as Potential Approach for Treating Alzheimer's Disease

逆转钙稳态失衡是治疗阿尔茨海默病的一种潜在方法

Dysregulation of Intracellular Calcium Signaling in Alzheimer's Disease

阿尔茨海默病中细胞内钙信号传导失调

Calcium signaling and molecular mechanisms underlying neurodegenerative diseases

钙信号传导和神经退行性疾病的分子机制

Disturbed calcium signaling in spinocerebellar ataxias and Alzheimer's disease

脊髓小脑性共济失调和阿尔茨海默病中的钙信号传导紊乱

Calcium signaling, excitability, and synaptic plasticity defects in a mouse model of Alzheimer's disease

阿尔茨海默病小鼠模型中钙信号传导、兴奋性和突触可塑性缺陷

Reduced synaptic STIM2 expression and impaired store-operated calcium entry cause destabilization of mature spines in mutant presenilin mice

突变型早老素小鼠突触STIM2表达降低和胞内钙库操纵性钙内流受损导致成熟树突棘不稳定。

Commentary on "Synaptic function is modulated by LRRK2 and glutamate release is increased in cortical neurons of G2019S LRRK2 knock-in mice"

对“LRRK2 调节突触功能，G2019S LRRK2 敲入小鼠皮层神经元中谷氨酸释放增加”一文的评论