日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Misprogramming of glucose metabolism impairs recovery of hippocampal slices from neuronal GLT-1 knockout mice and contributes to excitotoxic injury through mitochondrial superoxide production.

葡萄糖代谢的错误编程会损害神经元 GLT-1 敲除小鼠海马切片的恢复,并通过线粒体超氧化物的产生导致兴奋性毒性损伤

Li S, Wang J, Andersen J V, Aldana B I, Zhang B, Prochownik E V, Rosenberg P A

Targeted therapeutics in melanoma

黑色素瘤的靶向治疗

Zhuang, D; Mannava, S; Grachtchouk, V; Tang, W-H; Patil, S; Wawrzyniak, J A; Berman, A E; Giordano, T J; Prochownik, E V; Soengas, M S; Nikiforov, M A; Ali, Z; Fisher, R; Larkin, J

Functional and physical communication between oncoproteins and tumor suppressors

癌蛋白和抑癌蛋白之间的功能性和物理性通讯

Prochownik, E V

Differential patterns of DNA binding by myc and max proteins

myc 和 max 蛋白对 DNA 结合的不同模式

Prochownik, E V; VanAntwerp, M E

Attenuation of serum inducibility of immediate early genes by oncoproteins in tyrosine kinase signaling pathways

酪氨酸激酶信号通路中癌蛋白对血清诱导早期基因表达的减弱作用

Yu, C L; Prochownik, E V; Imperiale, M J; Jove, R

Antisense-mediated reduction in thrombospondin reverses the malignant phenotype of a human squamous carcinoma

反义介导的血小板反应蛋白减少可逆转人类鳞状细胞癌的恶性表型

Castle, V; Varani, J; Fligiel, S; Prochownik, E V; Dixit, V

Depletion of c-myc with specific antisense sequences reverses the transformed phenotype in ras oncogene-transformed NIH 3T3 cells

利用特异性反义序列敲低c-myc可逆转ras癌基因转化的NIH 3T3细胞的转化表型。

Sklar, M D; Thompson, E; Welsh, M J; Liebert, M; Harney, J; Grossman, H B; Smith, M; Prochownik, E V

The leucine zipper of c-Myc is required for full inhibition of erythroleukemia differentiation

c-Myc的亮氨酸拉链结构域是完全抑制红白血病分化所必需的。

Smith, M J; Charron-Prochownik, D C; Prochownik, E V

Induction of type C virions from normal rat kidney cells by 2-deoxy-D-glucose

2-脱氧-D-葡萄糖诱导正常大鼠肾细胞产生C型病毒颗粒

Prochownik, E V; Panem, S; Kirsten, W H