Proestling相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

The rs2046210 Polymorphism Is Associated with Endometriosis Risk and Elevated Estrogen Receptor 1 Expression in the Eutopic Endometrium of Women with the Disease

rs2046210多态性与子宫内膜异位症风险相关，并且与该病患者正常子宫内膜中雌激素受体1表达升高有关。

期刊:Biomedicines

影响因子:3.9

doi:10.3390/biomedicines12081657

Proestling, Katharina; Schreiber, Martin; Miedl, Heidi; Hudson, Quanah J; Husslein, Heinrich; Kuessel, Lorenz; Gstoettner, Manuela; Wenzl, Rene; Yotova, Iveta

免疫/内分泌

发表日期：2024

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Association of the Estrogen Receptor 1 Polymorphisms rs2046210 and rs9383590 with the Risk, Age at Onset and Prognosis of Breast Cancer

雌激素受体1多态性rs2046210和rs9383590与乳腺癌的风险、发病年龄和预后的关系

影响因子:5.2

doi:10.3390/cells12040515

Miedl, Heidi; Oswald, Denise; Haslinger, Isabella; Gstoettner, Manuela; Wenzl, René; Proestling, Katharina; Schneeberger, Christian; Yotova, Iveta; Schreiber, Martin

免疫/内分泌

发表日期：2023

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MLLT11 Regulates Endometrial Stroma Cell Adhesion, Proliferation and Survival in Ectopic Lesions of Women with Advanced Endometriosis

MLLT11 调控晚期子宫内膜异位症患者异位病灶中子宫内膜间质细胞的黏附、增殖和存活

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms25010439

Katharina Proestling ，Heinrich Husslein ，Quanah James Hudson ，Matthias Witzmann-Stern ，Barbara Widmar ，Zsuzsanna Bagó-Horváth ，Lejla Sandrieser ，Alexandra Perricos ，René Wenzl ，Iveta Yotova

细胞生物学

发表日期：2023

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LINC01133 Inhibits Invasion and Promotes Proliferation in an Endometriosis Epithelial Cell Line

LINC01133抑制子宫内膜异位症上皮细胞系的侵袭并促进其增殖

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms22168385

Iveta Yotova ，Quanah J Hudson ，Florian M Pauler ，Katharina Proestling ，Isabella Haslinger ，Lorenz Kuessel ，Alexandra Perricos ，Heinrich Husslein ，René Wenzl

细胞生物学

发表日期：2021

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Increased serum levels of mBDNF in women with minimal and mild endometriosis have no predictive power for the disease

轻度或极轻度子宫内膜异位症女性血清中mBDNF水平升高并不能预测该疾病的发生。

期刊:Experimental Biology and Medicine

影响因子:2.7

doi:10.1177/1535370217742600

Perricos, Alexandra; Ashjaei, Kazem; Husslein, Heinrich; Proestling, Katharina; Kuessel, Lorenz; Obwegeser, Reinhard; Wenzl, Rene; Yotova, Iveta

发表日期：2018

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Enhanced expression of the stemness-related factors OCT4, SOX15 and TWIST1 in ectopic endometrium of endometriosis patients

子宫内膜异位症患者异位子宫内膜中干性相关因子 OCT4、SOX15 和 TWIST1 的表达增强

期刊:Reproductive Biology and Endocrinology

影响因子:4.2

doi:10.1186/s12958-016-0215-4

Katharina Proestling, Peter Birner, Sukirthini Balendran, Nadine Nirtl, Erika Marton, Gülen Yerlikaya, Lorenz Kuessel, Theresa Reischer, Rene Wenzl, Berthold Streubel, Heinrich Husslein

发表日期：2016

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Enhanced epithelial to mesenchymal transition (EMT) and upregulated MYC in ectopic lesions contribute independently to endometriosis

异位病变中增强的上皮间质转化 (EMT) 和上调的 MYC 独立地导致子宫内膜异位症

期刊:Reproductive Biology and Endocrinology

影响因子:4.2

doi:10.1186/s12958-015-0063-7

Katharina Proestling, Peter Birner, Susanne Gamperl, Nadine Nirtl, Erika Marton, Gülen Yerlikaya, Rene Wenzl, Berthold Streubel, Heinrich Husslein

发表日期：2015

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Inhibition of the mevalonate pathway affects epigenetic regulation in cancer cells

甲羟戊酸途径的抑制会影响癌细胞的表观遗传调控。

期刊:Cancer Genetics

影响因子:2.1

doi:10.1016/j.cancergen.2015.03.008

Karlic, Heidrun; Thaler, Roman; Gerner, Christopher; Grunt, Thomas; Proestling, Katharina; Haider, Florian; Varga, Franz

细胞生物学

发表日期：2015

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Wnt-dependent T-cell factor-4 controls human etravillous trophoblast motility

Wnt 依赖性 T 细胞因子 4 控制人类绒毛膜滋养层细胞运动

期刊:Endocrinology

影响因子:3.8

doi:10.1210/en.2013-2042

Gudrun Meinhardt, Sandra Haider, Peter Haslinger, Katharina Proestling, Christian Fiala, Jürgen Pollheimer, Martin Knöfler

发表日期：2014

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Extravillous trophoblast-associated ADAM12 exerts pro-invasive properties, including induction of integrin beta 1-mediated cellular spreading

绒毛外滋养层细胞相关 ADAM12 具有促侵袭特性，包括诱导整合素 β1 介导的细胞扩散

期刊:Biology of Reproduction

影响因子:3.1

doi:10.1095/biolreprod.113.115279

Katarzyna Biadasiewicz, Valerie Fock, Sabine Dekan, Katharina Proestling, Philipp Velicky, Sandra Haider, Martin Knöfler, Camilla Fröhlich, Jürgen Pollheimer

细胞生物学

发表日期：2014

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